Evidence mapPaperPMID 30779243Full record

ArticleJournal of internal medicine2019

Investigation of human apoB48 metabolism using a new, integrated non-steady-state model of apoB48 and apoB100 kinetics.

E Björnson, C J Packard, M Adiels, L Andersson, N Matikainen, S Söderlund, J Kahri, C Sihlbom, A Thorsell, H Zhou and 2 more

2 registry-linked trialsOpen access · hybridAbstract read
In one paragraph

Article in Journal of internal medicine, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. It reports registered trial NCT02765399. Cited by 22 papers.

0numbers the graph read from it
0cells of the map it votes in
22citing papers in PubMed
4.7field-weighted citation impact, top 4% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

NCT02765399 phase4completed

The Effect of Liraglutide Treatment on Postprandial Chylomicron and VLDL Kinetics, Liver Fat and de Novo Lipogenesis - a Single-center Randomized Controlled Study

Ran2015Enrolled23Registered outcomes21Posted comparisons42ConditionsType 2 DiabetesArmsliraglutide, Placebo
Open the trial in the graph
NCT02948777 phase4completed

Effects of PCSK9 Inhibition by Evolocumab on Postprandial Lipid Metabolism in Type 2 Diabetes

Ran2016Enrolled14Registered outcomes25Posted comparisons24ConditionsType 2 Diabetes MellitusArmsEvolocumab
PMID 33356392PMID 31917184PMID 221537other papers from this trial
Open the trial in the graph
3 · Its place in the literature

Who cites it

22 citing papers in PubMed, 50 citations in OpenAlex.

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  15. Metabolism of Triglyceride-Rich Lipoproteins.Handbook of experimental pharmacology · 2022
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4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

12 authors at 5 institutions in 4 countries.

E BjörnsonDepartment of Molecular and Clinical Medicine, University of Gothenburg and Sahlgrenska University Hospital, Gothenburg, Sweden.
C J PackardInstitute of Cardiovascular and Medical Sciences, University of Glasgow, Glasgow, UK.
M AdielsDepartment of Molecular and Clinical Medicine, University of Gothenburg and Sahlgrenska University Hospital, Gothenburg, Sweden.
L AnderssonDepartment of Molecular and Clinical Medicine, University of Gothenburg and Sahlgrenska University Hospital, Gothenburg, Sweden.
N MatikainenResearch Programs Unit, Diabetes and Obesity, University of Helsinki, Helsinki, Finland.
S SöderlundResearch Programs Unit, Diabetes and Obesity, University of Helsinki, Helsinki, Finland.
J KahriResearch Programs Unit, Diabetes and Obesity, University of Helsinki, Helsinki, Finland.
C SihlbomProteomics Facility, University of Gothenburg, Gothenburg, Sweden.
A ThorsellProteomics Facility, University of Gothenburg, Gothenburg, Sweden.
H ZhouMerck Research Laboratories, Merck & Co. Inc., Kenilworth, NJ, USA.
M-R TaskinenResearch Programs Unit, Diabetes and Obesity, University of Helsinki, Helsinki, Finland.
J BorénDepartment of Molecular and Clinical Medicine, University of Gothenburg and Sahlgrenska University Hospital, Gothenburg, Sweden.ORCID 0000-0003-0786-8091
Sahlgrenska University Hospital · SEUniversity of Helsinki · FIUniversity of Gothenburg · SEMerck & Co., Inc., Rahway, NJ, USA (United States) · USUniversity of Glasgow · GB

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundTriglyceride-rich lipoproteins and their remnants have emerged as major risk factors for cardiovascular disease. New experimental approaches are required that permit simultaneous investigation of the dynamics of chylomicrons (CM) and apoB48 metabolism and of apoB100 in very low-density lipoproteins (VLDL).

methodsMass spectrometric techniques were used to determine the masses and tracer enrichments of apoB48 in the CM, VLDL

resultsThe kinetic model described the metabolism of apoB48 in CM, VLDL DISCUSSION: This novel non-steady-state model integrates the metabolic properties of both apoB100 and apoB48 and the kinetics of triglyceride. The model is physiologically relevant and provides insight not only into apoB48 release in the basal and postabsorptive states but also into the contribution of the intestine to VLDL pool size and kinetics.

Indexed as

Models, BiologicalAdultApolipoprotein B-100Apolipoprotein B-48HumansKineticsLipoproteins, VLDLMaleMiddle AgedApolipoprotein B-100Apolipoprotein B-48Lipoproteins, VLDLapolipoprotein B48kineticsmodelremnantsstable isotope

Identifiers

PMID30779243
PMCPMC6849847
OpenAlexW2916204077

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.