Evidence mapPaperPMID 30815552Full record

ArticleEndocrinology, diabetes & metabolism2018

Slope of change in HbA

Ralph A DeFronzo, Ele Ferrannini, Guntram Schernthaner, Stefan Hantel, Ulrich Elsasser, Christopher Lee, Thomas Hach, Søren S Lund

Open access · goldAbstract read
In one paragraph

Article in Endocrinology, diabetes & metabolism, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 10 papers.

0numbers the graph read from it
0cells of the map it votes in
10citing papers in PubMed
1.0field-weighted citation impact, top 23% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

10 citing papers in PubMed, 16 citations in OpenAlex.

  1. Trial
  2. Trial
  3. Article
  4. Article
  5. Article
  6. Precision Medicine in Type 2 Diabetes Mellitus: Utility and Limitations.Diabetes, metabolic syndrome and obesity : targets and therapy · 2023
    Review
  7. Article
  8. Review
  9. Article
  10. Slope of change in HbAEndocrinology, diabetes & metabolism · 2018
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 4 institutions in 4 countries.

Ralph A DeFronzoDiabetes Division University of Texas Health Science Center San Antonio TX USA.ORCID 0000-0003-3839-1724
Ele FerranniniCNR Institute of Clinical Physiology Pisa Italy.ORCID 0000-0002-1384-1584
Guntram SchernthanerDepartment of Medicine Rudolfstiftung Hospital Vienna Vienna Austria.
Stefan HantelBoehringer Ingelheim Pharma GmbH & Co. KG Ingelheim Germany.
Ulrich ElsasserBoehringer Ingelheim Pharma GmbH & Co. KG Ingelheim Germany.
Christopher LeeBoehringer Ingelheim Pharma GmbH & Co. KG Ingelheim Germany.
Thomas HachBoehringer Ingelheim Pharma GmbH & Co. KG Ingelheim Germany.
Søren S LundBoehringer Ingelheim Pharma GmbH & Co. KG Ingelheim Germany.
Boehringer Ingelheim (Germany) · DEIstituto di Fisiologia Clinica · ITRudolfinerhaus Hospital · ATThe University of Texas Health Science Center at San Antonio · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

aimsTo analyse the effect of baseline glycated haemoglobin (HbA MATERIALS AND

methodsUsing regression analyses of individual patient data from two Phase III studies, we compared the change in HbA

resultsSteeper slopes of HbA1c decline were observed with empagliflozin 10 or 25 mg vs sitagliptin monotherapy at week 24. Regression analysis showed slopes of -0.59 (95% CI -0.70, -0.47), -0.49 (95% CI -0.62, -0.37) and -0.29 (95% CI -0.42, -0.15) for empagliflozin 10 mg, empagliflozin 25 mg and sitagliptin, respectively (

conclusionsIncremental reductions in HbA

Indexed as

blood glucoseglycosylated haemoglobin Asodium‐glucose transporter 2

Identifiers

PMID30815552
PMCPMC6354821
OpenAlexW2795513595

What Socratic holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.