Evidence map›Paper›PMID 30860936›Full record

ArticleAdipocyte2019

KD025 (SLx-2119) suppresses adipogenesis at intermediate stage in human adipose-derived stem cells.

Duy Trong Vien Diep, Khue Ha Minh Duong, Hojung Choi, Hee-Sook Jun, Kwang-Hoon Chun

Open access · goldAbstract read
In one paragraph

Article in Adipocyte, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed, 1 pooled it
0.9field-weighted citation impact, top 26% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed, 1 synthesis or guideline pooled it, 15 citations in OpenAlex.

  1. Pooled it
  2. Article
  3. Formulary Drug Review: Belumosudil.Hospital pharmacy · 2022
    Review
  4. Review
  5. Article
  6. Article
  7. Article
  8. KD025 Shifts Pulmonary Endothelial Cell Bioenergetics and Decreases Baseline Lung Permeability.American journal of respiratory cell and molecular biology · 2020
    Article
  9. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 1 institution in 1 country.

Duy Trong Vien Diepa Gachon Institute of Pharmaceutical Sciences, College of Pharmacy , Gachon University , Incheon , Republic of Korea.
Khue Ha Minh Duonga Gachon Institute of Pharmaceutical Sciences, College of Pharmacy , Gachon University , Incheon , Republic of Korea.
Hojung Choia Gachon Institute of Pharmaceutical Sciences, College of Pharmacy , Gachon University , Incheon , Republic of Korea.ORCID 0000-0001-9235-5500
Hee-Sook Juna Gachon Institute of Pharmaceutical Sciences, College of Pharmacy , Gachon University , Incheon , Republic of Korea.
Kwang-Hoon Chuna Gachon Institute of Pharmaceutical Sciences, College of Pharmacy , Gachon University , Incheon , Republic of Korea.ORCID 0000-0001-8242-0778
Gachon University · KR

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Rho-associated kinases (ROCKs) have been reported to antagonize adipocyte differentiation, and inhibition of ROCKs by small molecules promotes adipogenesis. Surprisingly, our recent study revealed that the ROCK2-specific inhibitor KD025 (SLx-2119), suppresses differentiation at the intermediate stage in 3T3-L1 preadipocytes. To address whether the anti-adipogenic activity of KD025 is a generalizable property, we examined the effect of KD025 in human adipose-derived stem cells (hADSCs). KD025 significantly suppressed the adipocyte differentiation of hADSCs with downregulation of the protein and mRNA expression of various adipogenic and lipogenic markers, including PPARγ, C/EBPα, SREBP-1c, Glut4 and FABP4. Notably, we observed that adipocyte differentiation is effectively suppressed by exposure to KD025 during the mid-to-late period of adipogenesis but not at the earlier stages, showing stage-specificity. Contrary to expectations, KD025 upregulated the insulin signaling, as confirmed by the increased phosphorylation levels of Akt and GSK-3α/β, and the differentiation-promoting activity of insulin signaling was observed to be overwhelmed by the inhibitory activity. In addition, we observed that other ROCK inhibitors (Y-27632, fasudil, and H-1152P) did not suppress but promoted adipocyte differentiation. These results indicate that KD025 suppresses adipocyte differentiation by modulation of key factors activated at the intermediate stage of differentiation, and not by inhibition of ROCK2.

Indexed as

3T3-L1 CellsAdipocytesAdipogenesisAnimalsCCAAT-Enhancer-Binding Protein-alphaCell DifferentiationFatty Acid-Binding ProteinsGlucose Transporter Type 4Heterocyclic Compounds, 4 or More RingsHumansMesenchymal Stem CellsMicePhosphorylationPPAR gammarho-Associated KinasesSignal TransductionCCAAT-Enhancer-Binding Protein-alphaFABP4 protein, humanFatty Acid-Binding ProteinsGlucose Transporter Type 4Heterocyclic Compounds, 4 or More RingsKD025PPAR gammarho-Associated KinasesROCK2 protein, humanSLC2A4 protein, humanSterol Regulatory Element Binding Protein 1adipogenesisdifferentiationKD025Rho-associated kinaseROCKSLx-2119

Identifiers

PMID30860936
PMCPMC6768280
OpenAlexW2921851754

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.