Evidence map›Paper›PMID 30914409›Full record

ArticleEMBO reports2019

SWI/SNF chromatin remodeling controls Notch-responsive enhancer accessibility.

Zoe Pillidge, Sarah J Bray

Open access · bronzeAbstract read
In one paragraph

Article in EMBO reports, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 29 papers.

0numbers the graph read from it
0cells of the map it votes in
29citing papers in PubMed
1.8field-weighted citation impact, top 15% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

29 citing papers in PubMed, 42 citations in OpenAlex.

  1. Review
  2. Set1 promotes Notch-induced transcriptional activation.Cellular and molecular life sciences : CMLS · 2026
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  7. Review
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  12. Review
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  15. Advances in the role of SWI/SNF complexes in tumours.Journal of cellular and molecular medicine · 2023
    Review
  16. Review
  17. Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors at 1 institution in 1 country.

Zoe PillidgeDepartment of Physiology, Development and Neuroscience, University of Cambridge, Cambridge, UK.
Sarah J BrayDepartment of Physiology, Development and Neuroscience, University of Cambridge, Cambridge, UK sjb32@cam.ac.uk.ORCID 0000-0002-1642-599X
University of Cambridge · GB

Funding

Medical Research Council MR/L007177/1
6 · The paper itself

Abstract

Notch signaling plays a key role in many cell fate decisions during development by directing different gene expression programs via the transcription factor CSL, known as Su(H) in

Indexed as

AcetylationAnimalsChromatinChromatin Assembly and DisassemblyChromosomal Proteins, Non-HistoneDrosophilaDrosophila ProteinsGene Expression RegulationHistonesNucleosomesReceptors, NotchRegulatory Sequences, Nucleic AcidTranscription FactorsTranscription, GeneticChromatinChromosomal Proteins, Non-HistoneDrosophila ProteinsHistonesN protein, DrosophilaNucleosomesReceptors, NotchTranscription FactorsDrosophilahistone H3.3Notchnucleosome turnoverSWI/SNF

Identifiers

PMID30914409
PMCPMC6500979
OpenAlexW2888265725

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.