Evidence mapPaperPMID 30968587Full record

ArticlePhysiological reports2019

Sympathoexcitation and impaired arterial baroreflex sensitivity are linked to vascular inflammation in individuals with elevated resting blood pressure.

Ida T Fonkoue, Ngoc-Anh Le, Melanie L Kankam, Dana DaCosta, Toure N Jones, Paul J Marvar, Jeanie Park

Open access · goldAbstract read
In one paragraph

Article in Physiological reports, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 18 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
18citing papers in PubMed, 1 pooled it
2.3field-weighted citation impact, top 11% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

18 citing papers in PubMed, 1 synthesis or guideline pooled it, 27 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 3 institutions in 1 country.

Ida T FonkoueRenal Division, Department of Medicine, Emory University School of Medicine, Atlanta, Georgia.
Ngoc-Anh LeBiomarker Core Laboratory, Atlanta VA Healthcare System, Decatur, Georgia.
Melanie L KankamRenal Division, Department of Medicine, Emory University School of Medicine, Atlanta, Georgia.
Dana DaCostaRenal Division, Department of Medicine, Emory University School of Medicine, Atlanta, Georgia.
Toure N JonesRenal Division, Department of Medicine, Emory University School of Medicine, Atlanta, Georgia.
Paul J MarvarDepartment of Pharmacology and Physiology, Institute for Neuroscience, George Washington University, Washington, District of Columbia.
Jeanie ParkRenal Division, Department of Medicine, Emory University School of Medicine, Atlanta, Georgia.
Emory University · USAtlanta VA Health Care System · USGeorge Washington University · US

Funding

NEPHROLOGY INSTITUTIONAL TRAINING GRANTT32DK007656 · EMORY UNIVERSITY · 1990 to 2004
$741k
Neurovascular Regulation During Exercise in Humans With Chronic Kidney DiseaseR01HL135183 · EMORY UNIVERSITY · 2025 to 2025
$693k
American Heart Association-American Stroke Association 15CSA24340001CSRD VA I01 CX001065CSRD VA I01CX001065NHLBI NIH HHS R01 HL135183NIDDK NIH HHS T32 DK007656NIH HHS T32 DK-00756
6 · The paper itself

Abstract

Elevated Resting Blood Pressure (ERBP) in the prehypertensive range is associated with increased risk of hypertension and cardiovascular disease, the mechanisms of which remain unclear. Prior studies have suggested that ERBP may be associated with overactivation and dysregulation of the sympathetic nervous system (SNS). We hypothesized that compared to normotensives (≤120/80 mmHg), ERBP (120/80-139/89 mmHg) has higher SNS activity, impaired arterial baroreflex sensitivity (BRS), and increased vascular inflammation. Twenty-nine participants were studied: 16 otherwise healthy individuals with ERBP (blood pressure (BP) 130 ± 2/85 ± 2 mmHg) and 13 matched normotensive controls (mean BP 114 ± 2/73 ± 2 mmHg). We measured muscle sympathetic nerve activity (MSNA), beat-to-beat BP, and continuous electrocardiogram at rest and during arterial BRS testing via the modified Oxford technique. Blood was analyzed for the following biomarkers of vascular inflammation: lipoprotein-associated phospholipase A2 (Lp-PLA2), E-selectin, and intercellular adhesion molecule 1 (ICAM-1). Resting MSNA burst frequency (22 ± 2 vs. 16 ± 2 bursts/min, P = 0.036) and burst incidence (36 ± 3 vs. 25 ± 3 bursts/100 heart beats, P = 0.025) were higher in ERBP compared to controls. Cardiovagal BRS was blunted in ERBP compared to controls (13 ± 2 vs. 20 ± 3 msec/mmHg, P = 0.032), while there was no difference in sympathetic BRS between groups. Lp-PLA2 (169 ± 8 vs. 142 ± 9 nmol/min/mL, P = 0.020) and E-selectin (6.89 ± 0.6 vs. 4.45 ± 0.51 ng/mL, P = 0.004) were higher in ERBP versus controls. E-selectin (r = 0.501, P = 0.011) and ICAM-1 (r = 0.481, P = 0.015) were positively correlated with MSNA, while E-selectin was negatively correlated with cardiovagal BRS (r = -0.427, P = 0.030). These findings demonstrate that individuals with ERBP have SNS overactivity and impaired arterial BRS that are linked to biomarkers of vascular inflammation.

Indexed as

AdultBaroreflexBlood PressureBlood Pressure DeterminationFemaleHeart RateHemodynamicsHumansInflammationMalePrehypertensionSympathetic Nervous SystemVascular DiseasesBaroreflexblood pressureinflammationMSNA

Identifiers

PMID30968587
PMCPMC6456445
OpenAlexW2936235775

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.