Evidence map›Paper›PMID 31111240›Full record

ReviewCurrent atherosclerosis reports2019

Antisense Oligonucleotides Targeting Lipoprotein(a).

Anne Langsted, Børge G Nordestgaard

Abstract readReview
PubMed Publisher
In one paragraph

Review in Current atherosclerosis reports, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 25 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
25citing papers in PubMed, 1 pooled it
2.5field-weighted citation impact, top 10% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

25 citing papers in PubMed, 1 synthesis or guideline pooled it, 50 citations in OpenAlex.

  1. Pooled it
  2. Review
  3. Review
  4. Review
  5. Novel RNA-Based Therapies in the Management of Dyslipidemias.International journal of molecular sciences · 2025
    Review
  6. Review
  7. Article
  8. Review
  9. Review
  10. Advances in Treatment of Dyslipidemia.International journal of molecular sciences · 2023
    Review
  11. Article
  12. Article
  13. Review
  14. Ten things to know about ten cardiovascular disease risk factors - 2022.American journal of preventive cardiology · 2022
    Review
  15. Review
  16. Review
  17. Emerging Pharmacotherapy to Reduce Elevated Lipoprotein(a) Plasma Levels.American journal of cardiovascular drugs : drugs, devices, and other interventions · 2021
    Review
  18. Ten things to know about ten cardiovascular disease risk factors.American journal of preventive cardiology · 2021
    Review
  19. Article
  20. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors at 2 institutions in 1 country.

Anne LangstedDepartment of Clinical Biochemistry, Herlev Gentofte Hospital, Copenhagen University Hospital, Herlev Ringvej 75, DK-2730, Herlev, Denmark. anne.langsted.01@regionh.dk.
Børge G NordestgaardDepartment of Clinical Biochemistry, Herlev Gentofte Hospital, Copenhagen University Hospital, Herlev Ringvej 75, DK-2730, Herlev, Denmark.
Copenhagen University Hospital · DKUniversity of Copenhagen · DK

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

purpose of reviewHigh lipoprotein(a) levels are observationally and causally, from human genetics, associated with increased risk of cardiovascular disease including myocardial infarction and aortic valve stenosis. The European Atherosclerosis Society recommends screening for elevated lipoprotein(a) levels in high-risk patients. Different therapies have been suggested and some are used to treat elevated lipoprotein(a) levels such as niacin, PCSK9 inhibitors, and CETP inhibitors; however, to date, no randomized controlled trial has demonstrated that lowering of lipoprotein(a) leads to lower risk of cardiovascular disease. RECENT

findingsSynthetic oligonucleotides can be used to inactivate genes involved in disease processes. To lower lipoprotein(a), two antisense oligonucleotides have been developed, one targeting apolipoprotein B and one targeting apolipoprotein(a). Mipomersen is an antisense oligonucleotide targeting apolipoprotein B and thereby reducing levels of all apolipoprotein B containing lipoproteins in the circulation. Mipomersen has been shown to lower lipoprotein(a) by 20-50% in phase 3 studies. AKCEA-APO(a)-L

Indexed as

Antibodies, MonoclonalApolipoproteins BApoprotein(a)AtherosclerosisCholesterol Ester Transfer ProteinsHumansHyperlipoproteinemiasOligodeoxyribonucleotides, AntisenseOligonucleotidesPCSK9 InhibitorsProprotein Convertase 9RNA InterferenceAKCEA-APO(a)-LRxAntibodies, MonoclonalApolipoproteins BApoprotein(a)CETP protein, humanCholesterol Ester Transfer ProteinsmipomersenOligodeoxyribonucleotides, AntisenseOligonucleotidesPCSK9 InhibitorsPCSK9 protein, humanProprotein Convertase 9AKCEA-APO(a)-LRxApolipoprotein(a)Apolipoprotein BMipomersen

Identifiers

PMID31111240
OpenAlexW2945128165

What Socratic holds

Texttitle and abstract
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.