Evidence map›Paper›PMID 31208522›Full record

ReviewInternational review of neurobiology2019

Disorders of mitochondrial dynamics in peripheral neuropathy: Clues from hereditary neuropathy and diabetes.

Amy E Rumora, Masha G Savelieff, Stacey A Sakowski, Eva L Feldman

Open access · greenAbstract readReview
In one paragraph

Review in International review of neurobiology, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 31 papers.

0numbers the graph read from it
0cells of the map it votes in
31citing papers in PubMed
8.4field-weighted citation impact, top 2% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

31 citing papers in PubMed, 48 citations in OpenAlex.

  1. Trial
  2. Article
  3. Article
  4. Review
  5. Article
  6. Article
  7. Review
  8. Article
  9. Article
  10. Painful Diabetic Neuropathy: The Need for New Approaches.Journal of diabetes science and technology · 2024
    Review
  11. Article
  12. Review
  13. Review
  14. Article
  15. Article
  16. The conundrum of diabetic neuropathies-Past, present, and future.Journal of diabetes and its complications · 2022
    Review
  17. Review
  18. Serum lipidomic determinants of human diabetic neuropathy in type 2 diabetes.Annals of clinical and translational neurology · 2022
    Article
  19. Review
  20. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 1 institution in 1 country.

Amy E RumoraDepartment of Neurology, University of Michigan, Ann Arbor, MI, United States.
Masha G SavelieffDepartment of Neurology, University of Michigan, Ann Arbor, MI, United States.
Stacey A SakowskiDepartment of Neurology, University of Michigan, Ann Arbor, MI, United States.
Eva L FeldmanDepartment of Neurology, University of Michigan, Ann Arbor, MI, United States. Electronic address: efeldman@med.umich.edu.
University of Michigan–Ann Arbor · US

Funding

Coordinating and Bioinformatics Unit for the MMPC/DiaCompU24DK076169 · NIDDK · AUGUSTA UNIVERSITY · PI MCINDOE, RICHARD A. · 2006 to 2018
$42.4M
Integrated Systems Biology Approach to Diabetic Microvascular ComplicationsR24DK082841 · NIDDK · UNIVERSITY OF MICHIGAN AT ANN ARBOR · PI ABCOUWER, STEVEN F, BROSIUS, FRANK C · 2008 to 2020
$13.1M
Multidisciplinary Training Program in Basic Diabetes ResearchT32DK101357 · NIDDK · UNIVERSITY OF MICHIGAN AT ANN ARBOR · PI PETER ARVAN, Ormond A MacDougald · 2014 to 2026
$4.1M
Targeting Inflammation with Salsalate as a Novel Therapy for Diabetic NeuropathyR01DK107956 · NIDDK · UNIVERSITY OF MICHIGAN AT ANN ARBOR · PI BUSUI (POP-BUSUI), RODICA, FELDMAN, EVA LUCILLE · 2016 to 2020
$3.0M
Identifying Alterations in Mitochondrial Dynamics Associated with Diabetic NeuropathyF32DK112642 · NIDDK · UNIVERSITY OF MICHIGAN AT ANN ARBOR · PI RUMORA, AMY E. · 2017 to 2017
$57k
NIDDK NIH HHS F32 DK112642NIDDK NIH HHS R01 DK107956NIDDK NIH HHS R24 DK082841NIDDK NIH HHS T32 DK101357NIDDK NIH HHS U24 DK076169
6 · The paper itself

Abstract

Peripheral neuropathy is a common and debilitating complication of diabetes and prediabetes. Recent clinical studies have identified an association between the development of neuropathy and dyslipidemia in prediabetic and diabetic patients. Despite the prevalence of this complication, studies identifying molecular mechanisms that underlie neuropathy progression in prediabetes or diabetes are limited. However, dysfunctional mitochondrial pathways in hereditary neuropathy provide feasible molecular targets for assessing mitochondrial dysfunction in neuropathy associated with prediabetes or diabetes. Recent studies suggest that elevated levels of dietary saturated fatty acids (SFAs) associated with dyslipidemia impair mitochondrial dynamics in sensory neurons by inducing mitochondrial depolarization, compromising mitochondrial bioenergetics, and impairing axonal mitochondrial transport. This causes lower neuronal ATP and apoptosis. Conversely, monounsaturated fatty acids (MUFAs) restore nerve and sensory mitochondrial function. Understanding the mitochondrial pathways that contribute to neuropathy progression in prediabetes and diabetes may provide therapeutic targets for the treatment of this debilitating complication.

Indexed as

AnimalsDiabetes ComplicationsDiabetes MellitusDyslipidemiasGanglia, SpinalHeredodegenerative Disorders, Nervous SystemHumansHyperglycemiaMitochondrial DynamicsPeripheral Nervous System DiseasesBioenergeticsCharcot-Marie-Tooth diseaseDiabetesFissionFusionHereditary neuropathyMitochondriaMitochondrial associated membranesMitochondrial traffickingPrediabetes

Identifiers

PMID31208522
PMCPMC11533248
OpenAlexW2951472937

What Socratic holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.