Evidence map›Paper›PMID 31237726›Full record

ArticleHuman mutation2019

Novel ACTN1 variants in cases of thrombocytopenia.

Anne Vincenot, Paul Saultier, Shinji Kunishima, Marjorie Poggi, Marie-Françoise Hurtaud-Roux, Alain Roussel, Actn Study Coinvestigators, Nicole Schlegel, Marie-Christine Alessi

Registry-linked trialAbstract read
In one paragraph

Article in Human mutation, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. It is linked to trial NCT04272970 (Characterization of New Candidate Genes in Cases of Human Inherited Thrombocytopenia), which is not on this map. Cited by 10 papers.

0numbers the graph read from it
0cells of the map it votes in
10citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

NCT04272970 nacompletednot on this mapstarted 2020, after this paper: background citation

Characterization of New Candidate Genes in Cases of Human Inherited Thrombocytopenia (CATCH). Molecular Etiologies in Cases of Thrombocytopenia

TypeinterventionalSponsorInstitut National de la Santé Et de la Recherche Médicale, FranceRan2020 to 2024Enrolled12ConditionsThrombocytopenia, Inherited Platelet DisorderArmsValidation of a new gene / protein variant implicated in familial CT
3 · Its place in the literature

Who cites it

10 citing papers in PubMed.

  1. Article
  2. Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors.

Anne VincenotCHU Robert Debré, National Reference Center for Inherited Platelet Disorders and Biological Hematology Service, AP-HP, Paris, France.ORCID 0000-0001-7629-5982
Paul SaultierAix-Marseille Univ, INSERM, INRA, C2VN, Marseille, France.ORCID 0000-0002-6327-0898
Shinji KunishimaDepartment of Medical Technology, Gifu University of Medical Science, Seki, Gifu, Japan.ORCID 0000-0001-9212-0082
Marjorie PoggiAix-Marseille Univ, INSERM, INRA, C2VN, Marseille, France.ORCID 0000-0001-6331-9682
Marie-Françoise Hurtaud-RouxCHU Robert Debré, National Reference Center for Inherited Platelet Disorders and Biological Hematology Service, AP-HP, Paris, France.
Alain RousselAix Marseille University, CNRS, AFMB, Marseille, France.ORCID 0000-0002-9831-3261
Actn Study Coinvestigators
Nicole SchlegelCHU Robert Debré, National Reference Center for Inherited Platelet Disorders and Biological Hematology Service, AP-HP, Paris, France.
Marie-Christine AlessiAix-Marseille Univ, INSERM, INRA, C2VN, Marseille, France.ORCID 0000-0003-3927-5792

Funding

Assistance Publique-Hôpitaux de Paris DRCD number P070110Fondation pour la Recherche Médicale P.Saultier/FDM20150633607
6 · The paper itself

Abstract

The ACTN1 gene has been implicated in inherited macrothrombocytopenia. To decipher the spectrum of variants and phenotype of ACTN1-related thrombocytopenia, we sequenced the ACTN1 gene in 272 cases of unexplained chronic or familial thrombocytopenia. We identified 15 rare, monoallelic, nonsynonymous and likely pathogenic ACTN1 variants in 20 index cases from 20 unrelated families. Thirty-one family members exhibited thrombocytopenia. Targeted sequencing was carried out on 12 affected relatives, which confirmed presence of the variant. Twenty-eight of 32 cases with monoallelic ACTN1 variants had mild to no bleeding complications. Eleven cases harbored 11 different unreported ACTN1 variants that were monoallelic and likely pathogenic. Nine variants were located in the α-actinin-1 (ACTN1) rod domain and were predicted to hinder dimer formation. These variants displayed a smaller increase in platelet size compared with variants located outside the rod domain. In vitro expression of the new ACTN1 variants induced actin network disorganization and led to increased thickness of actin fibers. These findings expand the repertoire of ACTN1 variants associated with thrombocytopenia and highlight the high frequency of ACTN1-related thrombocytopenia cases. The rod domain, like other ACTN1 functional domains, may be mutated resulting in actin disorganization in vitro and thrombocytopenia with normal platelet size in most cases.

Indexed as

MutationActininAdolescentAdultAgedChildFemaleGenetic Predisposition to DiseaseHumansMaleMiddle AgedMutagenesis, Site-DirectedPedigreeProtein DomainsSequence Analysis, DNAThrombocytopeniaActininACTN1 protein, humanactinactininACTN1constitutionalplateletrod domainthrombocytopenia

Identifiers

PMID31237726
PMCPMC6900141

What Socratic holds

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Registered trials

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.