Evidence mapPaperPMID 31336263Full record

ReviewCytokine2019

Stromal cell-derived factor-1 (CXCL12) and its role in bone and muscle biology.

William Gilbert, Robert Bragg, Ahmed M Elmansi, Meghan E McGee-Lawrence, Carlos M Isales, Mark W Hamrick, William D Hill, Sadanand Fulzele

Open access · greenAbstract readReview
In one paragraph

Review in Cytokine, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 38 papers.

0numbers the graph read from it
0cells of the map it votes in
38citing papers in PubMed
2.2field-weighted citation impact, top 10% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

38 citing papers in PubMed, 65 citations in OpenAlex.

  1. Article
  2. Impact of aging on CD146Frontiers in bioengineering and biotechnology · 2026
    Article
  3. Article
  4. Review
  5. Review
  6. Article
  7. Article
  8. Article
  9. Article
  10. Selective PET imaging of CXCR4 using the AlEuropean journal of nuclear medicine and molecular imaging · 2025
    Article
  11. Article
  12. Review
  13. Article
  14. Article
  15. Article
  16. Review
  17. Review
  18. Review
  19. Directing the migration of serum-free,Frontiers in immunology · 2024
    Article
  20. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 3 institutions in 1 country.

William GilbertDepartment of Orthopaedic Surgery, Augusta University, Augusta, GA 30912, United States.
Robert BraggDepartment of Orthopaedic Surgery, Augusta University, Augusta, GA 30912, United States.
Ahmed M ElmansiDepartment of Pathology and Laboratory Medicine, Medical University of South Carolina, Charleston, SC 29403, United States.
Meghan E McGee-LawrenceDepartment of Orthopaedic Surgery, Augusta University, Augusta, GA 30912, United States; Cell Biology and Anatomy, Augusta University, Augusta, GA 30912, United States.
Carlos M IsalesDepartment of Orthopaedic Surgery, Augusta University, Augusta, GA 30912, United States; Department of Medicine, Augusta University, Augusta, GA 30912, United States.
Mark W HamrickDepartment of Orthopaedic Surgery, Augusta University, Augusta, GA 30912, United States; Cell Biology and Anatomy, Augusta University, Augusta, GA 30912, United States.
William D HillDepartment of Pathology and Laboratory Medicine, Medical University of South Carolina, Charleston, SC 29403, United States; Ralph H Johnson Veterans Affairs Medical Center, Charleston, SC 29403, United States.
Sadanand FulzeleDepartment of Orthopaedic Surgery, Augusta University, Augusta, GA 30912, United States; Cell Biology and Anatomy, Augusta University, Augusta, GA 30912, United States. Electronic address: sfulzele@augusta.edu.
Augusta University · USMedical University of South Carolina · USRalph H. Johnson VA Medical Center · US

Funding

MULTIDISCIPLINARY RESEARCH TRAINING IN AGINGT32AG000114 · UNIVERSITY OF MICHIGAN AT ANN ARBOR · 1985 to 2025
$3.7M
PROJECT 4 - Kyn-AhR crosstalk with nuclear receptor-mediated signaling in skeletal agingP01AG036675 · AUGUSTA UNIVERSITY · 2025 to 2025
$2.2M
CSRD VA I01 CX000930NIA NIH HHS P01 AG036675NIA NIH HHS R01 AG067510NIA NIH HHS T32 AG000114
6 · The paper itself

Abstract

Musculoskeletal disorders are the leading cause of disability worldwide; two of the most prevalent of which are osteoporosis and sarcopenia. Each affect millions in the aging population across the world and the associated morbidity and mortality contributes to billions of dollars in annual healthcare cost. Thus, it is important to better understand the underlying pathologic mechanisms of the disease process. Regulatory chemokine, CXCL12, and its receptor, CXCR4, are recognized to be essential in the recruitment, localization, maintenance, development and differentiation of progenitor stem cells of the musculoskeletal system. CXCL12 signaling results in the development and functional ability of osteoblasts, osteoclasts, satellite cells and myoblasts critical to maintaining musculoskeletal homeostasis. Interestingly, one suggested pathologic mechanism of osteoporosis and sarcopenia is a decline in the regenerative capacity of musculoskeletal progenitor stem cells. Thus, because CXCL12 is critical to progenitor function, a disruption in the CXCL12 signaling axis might play a distinct role in these pathological processes. Therefore, in this article, we perform a review of CXCL12, its physiologic and pathologic function in bone and muscle, and potential targets for therapeutic development.

Indexed as

Cell DifferentiationSignal TransductionBone and BonesChemokine CXCL12HumansMusclesOsteoporosisReceptors, CXCR4SarcopeniaStem CellsChemokine CXCL12CXCL12 protein, humanCXCR4 protein, humanReceptors, CXCR4BoneMuscleStromal cell-derived D factor-1 (CXCL12)

Identifiers

PMID31336263
PMCPMC6948927
OpenAlexW2963667019

What Socratic holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.