Evidence mapPaperPMID 31446151Full record

ArticleMolecular metabolism2019

Direct and indirect effects of liraglutide on hypothalamic POMC and NPY/AgRP neurons - Implications for energy balance and glucose control.

Zhenyan He, Yong Gao, Linh Lieu, Sadia Afrin, Jianhong Cao, Natalie J Michael, Yanbin Dong, Jia Sun, Hongbo Guo, Kevin W Williams

Open access · goldAbstract read
In one paragraph

Article in Molecular metabolism, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 64 papers.

0numbers the graph read from it
0cells of the map it votes in
64citing papers in PubMed
4.1field-weighted citation impact, top 5% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

64 citing papers in PubMed, 122 citations in OpenAlex.

  1. Article
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  5. GLP-1 physiology and pharmacology along the gut-brain axis.The Journal of clinical investigation · 2026
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  8. The Genetic Blueprint of Obesity: From Pathogenesis to Novel Therapies.Obesity reviews : an official journal of the International Association for the Study of Obesity · 2025
    Review
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  13. Mechanisms of glucagon-like-peptide 1 in the brain beyond metabolic effects.Annals of pediatric endocrinology & metabolism · 2025
    Article
  14. Review
  15. Article
  16. Review
  17. Review
  18. Review
  19. Article
  20. Review

4 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors at 4 institutions in 2 countries.

Zhenyan HeThe National Key Clinical Specialty, The Engineering Technology Research Center of Education Ministry of China, Guangdong Provincial Key Laboratory on Brain Function Repair and Regeneration, Department of Neurosurgery, Zhujiang Hospital, Southern Medical University, Guangzhou 510282, China; Division of Hypothalamic Research, the University of Texas Southwestern Medical Center at Dallas, Dallas, TX, USA.
Yong GaoDivision of Hypothalamic Research, the University of Texas Southwestern Medical Center at Dallas, Dallas, TX, USA; National Laboratory of Medical Molecular Biology, Institute of Basic Medical Sciences, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing 100005, China.
Linh LieuDivision of Hypothalamic Research, the University of Texas Southwestern Medical Center at Dallas, Dallas, TX, USA.
Sadia AfrinDivision of Hypothalamic Research, the University of Texas Southwestern Medical Center at Dallas, Dallas, TX, USA.
Jianhong CaoDivision of Hypothalamic Research, the University of Texas Southwestern Medical Center at Dallas, Dallas, TX, USA; Pi-wei Institute, Guangzhou University of Chinese Medicine, 12 Jichang Road, Guangzhou 510405, China.
Natalie J MichaelDivision of Hypothalamic Research, the University of Texas Southwestern Medical Center at Dallas, Dallas, TX, USA.
Yanbin DongDivision of Hypothalamic Research, the University of Texas Southwestern Medical Center at Dallas, Dallas, TX, USA; Pi-wei Institute, Guangzhou University of Chinese Medicine, 12 Jichang Road, Guangzhou 510405, China.
Jia SunDivision of Hypothalamic Research, the University of Texas Southwestern Medical Center at Dallas, Dallas, TX, USA.
Hongbo GuoThe National Key Clinical Specialty, The Engineering Technology Research Center of Education Ministry of China, Guangdong Provincial Key Laboratory on Brain Function Repair and Regeneration, Department of Neurosurgery, Zhujiang Hospital, Southern Medical University, Guangzhou 510282, China.
Kevin W WilliamsDivision of Hypothalamic Research, the University of Texas Southwestern Medical Center at Dallas, Dallas, TX, USA. Electronic address: Kevin.Williams@UTSouthwestern.edu.
The University of Texas Southwestern Medical Center · USZhujiang Hospital · CNChinese Academy of Medical Sciences & Peking Union Medical College · CNGuangzhou University of Chinese Medicine · CN

Funding

NIDDK NIH HHS P01 DK119130NIDDK NIH HHS R01 DK100699NIDDK NIH HHS R01 DK103185NIDDK NIH HHS R01 DK119169
6 · The paper itself

Abstract

objectiveThe long-acting glucagon-like peptide-1 receptor (GLP-1R) agonist, liraglutide, stimulates insulin secretion and efficiently suppresses food intake to reduce body weight. As such, liraglutide is growing in popularity in the treatment of diabetes and chronic weight management. Within the brain, liraglutide has been shown to alter the activity of hypothalamic proopiomelanocortin (POMC) and Neuropeptide Y/Agouti-related peptide (NPY/AgRP) neurons. Moreover, the acute activities of POMC and NPY neurons have been directly linked to feeding behavior, body weight, and glucose metabolism. Despite the increased usage of liraglutide and other GLP-1 analogues as diabetic and obesity interventions, the cellular mechanisms by which liraglutide alters the activity of metabolically relevant neuronal populations are poorly understood.

methodsIn order to resolve this issue, we utilized neuron-specific transgenic mouse models to identify POMC and NPY neurons for patch-clamp electrophysiology experiments.

resultsWe found that liraglutide directly activated arcuate POMC neurons via TrpC5 channels, sharing a similar mechanistic pathway to the adipose-derived peptide leptin. Liraglutide also indirectly increases excitatory tone to POMC neurons. In contrast, liraglutide inhibited NPY/AgRP neurons through post-synaptic GABA

conclusionsTaken together, our results identify critical molecular mechanisms linking GLP-1 analogues in arcuate POMC and NPY/AgRP neurons with metabolism.

Indexed as

Agouti-Related ProteinAnimalsBlood GlucoseEnergy MetabolismHypoglycemic AgentsHypothalamusLiraglutideMaleMiceMice, TransgenicNeuronsNeuropeptide YPro-OpiomelanocortinAgouti-Related ProteinBlood GlucoseHypoglycemic AgentsLiraglutideNeuropeptide YPro-OpiomelanocortinElectrophysiologyLiraglutideMelanocortinNPY/AgRPPatch-clampPOMC

Identifiers

PMID31446151
PMCPMC6822260
OpenAlexW2965632554

What Socratic holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.