Evidence map›Paper›PMID 31511206›Full record

ArticleNan fang yi ke da xue xue bao = Journal of Southern Medical University2019

[Effect of the chemoprotectant tempol on anti-tumor activity of cisplatin].

Shuangyan Ye, Sisi Zeng, Mengqiu Huang, Jianping Chen, Xi Chen, Pengfei Xu, Qianli Wang, Wenwen Gao, Bingsheng Yang, Bingtao Hao and 2 more

Abstract read
In one paragraph

Article in Nan fang yi ke da xue xue bao = Journal of Southern Medical University, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
0.2field-weighted citation impact, top 45% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed, 1 citations in OpenAlex.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

12 authors at 1 institution in 1 country.

Shuangyan YeCancer Research Institute, School of Basic Medical Sciences, Southern Medical University/Guangdong Provincial Key Laboratory of Cancer Immunotherapy/Guangzhou Key Laboratory of Tumor Immunology Research, Guangzhou 510515, China.
Sisi ZengCancer Research Institute, School of Basic Medical Sciences, Southern Medical University/Guangdong Provincial Key Laboratory of Cancer Immunotherapy/Guangzhou Key Laboratory of Tumor Immunology Research, Guangzhou 510515, China.
Mengqiu HuangCancer Research Institute, School of Basic Medical Sciences, Southern Medical University/Guangdong Provincial Key Laboratory of Cancer Immunotherapy/Guangzhou Key Laboratory of Tumor Immunology Research, Guangzhou 510515, China.
Jianping ChenCancer Research Institute, School of Basic Medical Sciences, Southern Medical University/Guangdong Provincial Key Laboratory of Cancer Immunotherapy/Guangzhou Key Laboratory of Tumor Immunology Research, Guangzhou 510515, China.
Xi ChenCancer Research Institute, School of Basic Medical Sciences, Southern Medical University/Guangdong Provincial Key Laboratory of Cancer Immunotherapy/Guangzhou Key Laboratory of Tumor Immunology Research, Guangzhou 510515, China.
Pengfei XuCancer Research Institute, School of Basic Medical Sciences, Southern Medical University/Guangdong Provincial Key Laboratory of Cancer Immunotherapy/Guangzhou Key Laboratory of Tumor Immunology Research, Guangzhou 510515, China.
Qianli WangCancer Research Institute, School of Basic Medical Sciences, Southern Medical University/Guangdong Provincial Key Laboratory of Cancer Immunotherapy/Guangzhou Key Laboratory of Tumor Immunology Research, Guangzhou 510515, China.
Wenwen GaoCancer Research Institute, School of Basic Medical Sciences, Southern Medical University/Guangdong Provincial Key Laboratory of Cancer Immunotherapy/Guangzhou Key Laboratory of Tumor Immunology Research, Guangzhou 510515, China.
Bingsheng YangZhujiang Hospital, Southern Medical University, Guangzhou 510280, China.
Bingtao HaoCancer Research Institute, School of Basic Medical Sciences, Southern Medical University/Guangdong Provincial Key Laboratory of Cancer Immunotherapy/Guangzhou Key Laboratory of Tumor Immunology Research, Guangzhou 510515, China.
Wenhuan HuangNational Key Discipline of Human Anatomy, School of Basic Medical Sciences, Southern Medical University, Guangzhou 510515, China.
Qiuzhen LiuCancer Research Institute, School of Basic Medical Sciences, Southern Medical University/Guangdong Provincial Key Laboratory of Cancer Immunotherapy/Guangzhou Key Laboratory of Tumor Immunology Research, Guangzhou 510515, China.
Southern Medical University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

objectiveTo investigate the effect of the chemoprotectant tempol on the anti-tumor activity of cisplatin (DDP).

methodsThe cellular toxicity of tempol in human colon cancer SW480 cells and mouse colon cancer CT26 cells were evaluated using MTT and cell counting kit-8 assays. CalcuSyn software analysis was used to determine the interaction between tempol and DDP in inhibition of the cell viability. A subcutaneous homograft mouse model of colon cancer was established. The mice were randomly divided into control group, tempol group, cisplatin group and tempol + DDP treatment group with intraperitoneal injections of the indicated agents. The tumor size, body weight and lifespan of the mice were measured, and HE staining was used to analyze the cytotoxic effect of the agents on the kidney and liver. Immunohistochemistry and Western blotting were performed to detect the expression of Bax and Bcl2 in the tumor tissue, and TUNEL staining was used to analyze the tumor cell apoptosis. The level of reactive oxygen species (ROS) in the tumor tissue was determined using flow cytometry.

resultsTempol showed inhibitory effects on the viability of SW480 and CT26 cells. CalcuSyn software analysis showed that tempol had a synergistic anti-tumor effect with DDP (CI < 1). In the homograft mouse model, tempol treatment alone did not produce obvious anti-tumor effect. HE staining showed that the combined use of tempol and DDP alleviated DDP-induced fibrogenesis in the kidneys, but tempol also reduced the anti-tumor activity of DDP. Compared with the mice treated with DDP alone, the mice treated with both tempol and DDP had a significantly larger tumor size (

conclusionsTempol can attenuate the anti-tumor effect of DDP while reducing the side effects of DDP. Caution must be taken and the risks and benefits should be carefully weighed when considering the use of tempol as an anti-oxidant to reduce the toxicities of DDP.

Indexed as

AnimalsAntineoplastic AgentsAntioxidantsApoptosisCell Line, TumorCell ProliferationCisplatinCyclic N-OxidesDrug Resistance, NeoplasmHumansMiceSpin LabelsAntineoplastic AgentsAntioxidantsCisplatinCyclic N-OxidesSpin Labelstempolanti-oxidantschemotherapycisplatindrug toxicityreactive oxygen speciestempol

Identifiers

PMID31511206
PMCPMC6765593
OpenAlexW2988890868

What Socratic holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.