Evidence mapPaperPMID 31571412Full record

Trial reportObesity (Silver Spring, Md.)2019

Impact of Acipimox Therapy on Free Fatty Acid Efflux and Endothelial Function in the Metabolic Syndrome: A Randomized Trial.

Aaron W Aday, Allison B Goldfine, Justin M Gregory, Joshua A Beckman

Registry-linked trialOpen access · greenAbstract readRandomized Controlled Trial
In one paragraph

Trial report in Obesity (Silver Spring, Md.), 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. It is linked to trial NCT00759291 (The Impact of Free Fatty Acid Reduction on Vascular Function in the Metabolic Syndrome), which is not on this map. Cited by 7 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
7citing papers in PubMed, 1 pooled it
0.6field-weighted citation impact, top 27% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

NCT00759291 phase2 / phase3completednot on this map

The Impact of Free Fatty Acid Reduction on Vascular Function in the Metabolic Syndrome

TypeinterventionalSponsorBrigham and Women's HospitalRan2006 to 2017Enrolled40ConditionsMetabolic SyndromeArmsacipimox, Placebo
3 · Its place in the literature

Who cites it

7 citing papers in PubMed, 1 synthesis or guideline pooled it, 12 citations in OpenAlex.

  1. Pooled it
  2. Trial
  3. Trial
  4. Article
  5. Article
  6. Article
  7. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 2 institutions in 1 country.

Aaron W AdayVanderbilt Translational and Clinical Cardiovascular Research Center, Division of Cardiovascular Medicine, Department of Medicine, Vanderbilt University Medical Center, Nashville, Tennessee, USA.ORCID 0000-0001-6243-3432
Allison B GoldfineResearch Division, Joslin Diabetes Center, Harvard Medical School, Boston, Massachusetts, USA.
Justin M GregoryIan M. Burr Division of Pediatric Endocrinology and Diabetes, Vanderbilt University Medical Center, Nashville, Tennessee, USA.
Joshua A BeckmanVanderbilt Translational and Clinical Cardiovascular Research Center, Division of Cardiovascular Medicine, Department of Medicine, Vanderbilt University Medical Center, Nashville, Tennessee, USA.
Vanderbilt University Medical Center · USJoslin Diabetes Center · US

Funding

NHLBI NIH HHS K12 HL133117
6 · The paper itself

Abstract

objectiveInsulin resistance is associated with increased lipolysis and elevated concentrations of free fatty acids (FFA), which in turn contribute to impaired vascular function. It was hypothesized that lowering FFA with acipimox, a nicotinic acid derivative that impairs FFA efflux, would improve endothelial function, measured by flow-mediated dilation (FMD), in individuals with metabolic syndrome.

methodsA total of 18 participants with metabolic syndrome and 17 healthy controls were enrolled and treated with acipimox 250 mg orally every 6 hours or placebo for 7 days in a randomized, double-blind, crossover trial.

resultsAcipimox reduced FFA concentrations among individuals with metabolic syndrome to near normal levels (P = 0.01), but there was no change among healthy controls (P = 0.17). Acipimox did not improve endothelial-dependent FMD in either group (metabolic syndrome: P = 0.42; healthy controls: P = 0.16), although endothelial-independent nitroglycerin-mediated dilation among those with metabolic syndrome tended to increase (20.3%, P = 0.06). There were no changes in blood lipids or markers of inflammation following therapy. There was minimal correlation between change in FMD and baseline measures of BMI ( ρ = -0.09) or waist circumference ( ρ = -0.15).

conclusionsIn groups with normal or elevated baseline FFA, short-term reductions do not improve endothelial function assessed by FMD.

Indexed as

AdultAgedBlood GlucoseCross-Over StudiesDouble-Blind MethodEndothelium, VascularFatty Acids, NonesterifiedFemaleHumansHypolipidemic AgentsInsulinInsulin ResistanceLipid MetabolismMaleMetabolic SyndromeMiddle AgedacipimoxBlood GlucoseFatty Acids, NonesterifiedHypolipidemic AgentsInsulinPyrazines

Identifiers

PMID31571412
PMCPMC6832806
OpenAlexW2977727431

What Socratic holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.