Evidence mapPaperPMID 31664147Full record

ArticleScientific reports2019

Leucine-rich diet induces a shift in tumour metabolism from glycolytic towards oxidative phosphorylation, reducing glucose consumption and metastasis in Walker-256 tumour-bearing rats.

Laís Rosa Viana, Natália Tobar, Estela Natacha Brandt Busanello, Ana Carolina Marques, Andre Gustavo de Oliveira, Tanes I Lima, Gabrielly Machado, Bianca Gazieri Castelucci, Celso Dario Ramos, Sérgio Q Brunetto and 4 more

Open access · goldAbstract read
In one paragraph

Article in Scientific reports, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 25 papers.

0numbers the graph read from it
0cells of the map it votes in
25citing papers in PubMed
1.6field-weighted citation impact, top 18% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

25 citing papers in PubMed, 42 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

14 authors at 1 institution in 1 country.

Laís Rosa VianaLaboratory of Nutrition and Cancer, Department of Structural and Functional Biology, Biology Institute, University of Campinas, Campinas, SP, Brazil.
Natália TobarDivision of Nuclear Medicine, Department of Radiology, School of Medical Sciences, University of Campinas, Campinas, SP, Brazil.
Estela Natacha Brandt BusanelloSchool of Medical Sciences, Department of Clinical Pathology, State University of Campinas, Campinas, SP, Brazil.
Ana Carolina MarquesSchool of Medical Sciences, Department of Clinical Pathology, State University of Campinas, Campinas, SP, Brazil.
Andre Gustavo de OliveiraLaboratory of Nutrition and Cancer, Department of Structural and Functional Biology, Biology Institute, University of Campinas, Campinas, SP, Brazil.
Tanes I LimaObesity and Comorbidities Research Centre, Biology Institute, University of Campinas, Campinas, SP, Brazil.
Gabrielly MachadoLaboratory of Nutrition and Cancer, Department of Structural and Functional Biology, Biology Institute, University of Campinas, Campinas, SP, Brazil.
Bianca Gazieri CastelucciLaboratory of Cytochemistry and Immunocytochemistry, Department of Biochemistry and Tissue Biology. Biology Institute, University of Campinas, Campinas, SP, Brazil.
Celso Dario RamosDivision of Nuclear Medicine, Department of Radiology, School of Medical Sciences, University of Campinas, Campinas, SP, Brazil.
Sérgio Q BrunettoDivision of Nuclear Medicine, Department of Radiology, School of Medical Sciences, University of Campinas, Campinas, SP, Brazil.
Leonardo Reis SilveiraObesity and Comorbidities Research Centre, Biology Institute, University of Campinas, Campinas, SP, Brazil.
Anibal Eugenio VercesiSchool of Medical Sciences, Department of Clinical Pathology, State University of Campinas, Campinas, SP, Brazil.
Sílvio Roberto ConsonniLaboratory of Cytochemistry and Immunocytochemistry, Department of Biochemistry and Tissue Biology. Biology Institute, University of Campinas, Campinas, SP, Brazil.
Maria Cristina Cintra Gomes-MarcondesLaboratory of Nutrition and Cancer, Department of Structural and Functional Biology, Biology Institute, University of Campinas, Campinas, SP, Brazil. cintgoma@unicamp.br.ORCID http://orcid.org/0000-0002-1713-756X
Universidade Estadual de Campinas (UNICAMP) · BR

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Leucine can stimulate protein synthesis in skeletal muscle, and recent studies have shown an increase in leucine-related mitochondrial biogenesis and oxidative phosphorylation capacity in muscle cells. However, leucine-related effects in tumour tissues are still poorly understood. Thus, we described the effects of leucine in both in vivo and in vitro models of a Walker-256 tumour. Tumour-bearing Wistar rats were randomly distributed into a control group (W; normoprotein diet) and leucine group (LW; leucine-rich diet [normoprotein + 3% leucine]). After 20 days of tumour evolution, the animals underwent

Indexed as

AnimalsCarcinoma 256, WalkerFemaleFood, FormulatedGlucoseGlycolysisLeucineNeoplasm MetastasisOxidative PhosphorylationRatsRats, WistarGlucoseLeucine

Identifiers

PMID31664147
PMCPMC6820796
OpenAlexW2982425498

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.