Evidence map›Paper›PMID 31982480›Full record

ArticleMetabolism: clinical and experimental2020

Twelve weeks of exenatide treatment increases [

Laura G M Janssen, Kimberly J Nahon, Katrien F M Bracké, Dennis van den Broek, Renée Smit, Aashley S D Sardjoe Mishre, Lisa L Koorneef, Borja Martinez-Tellez, Jedrzej Burakiewicz, Hermien E Kan and 7 more

2 registry-linked trialsOpen access · hybridAbstract readControlled Clinical Trial
PubMed Publisher
In one paragraph

Article in Metabolism: clinical and experimental, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. It is linked to 2 registered trials, which are not on this map. Cited by 33 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
33citing papers in PubMed, 1 pooled it
2.5field-weighted citation impact, top 10% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

NCT05419726 recruitingstarted 2023, after this paper: background citation

Brown Adipose Tissue Activity in Response to Semaglutide Administered to Obese Subjects.

Ran2023Enrolled20Registered outcomes3Posted comparisons0ConditionsObesityArmsSemaglutide Injectable Product (not provided by the study)
Open the trial in the graph
NCT03002675 phase4unknown statusnot on this map

The Effect of Exenatide on Brown Adipose Tissue Activity and Energy Expenditure in Healthy Young Men

TypeinterventionalSponsorIngrid JazetRan2016Enrolled24ConditionsObesityArmsBydureon
3 · Its place in the literature

Who cites it

33 citing papers in PubMed, 1 synthesis or guideline pooled it, 52 citations in OpenAlex.

  1. New Mediators in the Crosstalk between Different Adipose Tissues.International journal of molecular sciences · 2024
    Pooled it
  2. Trial
  3. Effects of Glucagon-Like Peptide-1 Receptor Agonists (Mono and Combination Therapy) on Energy Expenditure: A Scoping Review.Obesity reviews : an official journal of the International Association for the Study of Obesity · 2026
    Article
  4. Review
  5. Article
  6. Article
  7. Article
  8. Review
  9. Article
  10. Review
  11. Evaluating the potential of metabolic drugs in obstructive sleep apnea and obesity: a narrative review.Journal of clinical sleep medicine : JCSM : official publication of the American Academy of Sleep Medicine · 2025
    Review
  12. Article
  13. Review
  14. Review
  15. Article
  16. Article
  17. Review
  18. Review
  19. Article
  20. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

17 authors at 2 institutions in 2 countries.

Laura G M JanssenDepartment of Medicine, Division of Endocrinology, Leiden University Medical Center, Leiden, the Netherlands; Einthoven Laboratory for Experimental Vascular Medicine, Leiden University Medical Center, Leiden, the Netherlands.
Kimberly J NahonDepartment of Medicine, Division of Endocrinology, Leiden University Medical Center, Leiden, the Netherlands; Einthoven Laboratory for Experimental Vascular Medicine, Leiden University Medical Center, Leiden, the Netherlands.
Katrien F M BrackéDepartment of Medicine, Division of Endocrinology, Leiden University Medical Center, Leiden, the Netherlands; Einthoven Laboratory for Experimental Vascular Medicine, Leiden University Medical Center, Leiden, the Netherlands.
Dennis van den BroekDepartment of Medicine, Division of Endocrinology, Leiden University Medical Center, Leiden, the Netherlands; Einthoven Laboratory for Experimental Vascular Medicine, Leiden University Medical Center, Leiden, the Netherlands.
Renée SmitDepartment of Medicine, Division of Endocrinology, Leiden University Medical Center, Leiden, the Netherlands; Einthoven Laboratory for Experimental Vascular Medicine, Leiden University Medical Center, Leiden, the Netherlands.
Aashley S D Sardjoe MishreDepartment of Radiology, C.J. Gorter Center for High Field MRI, Leiden University Medical Center, Leiden, the Netherlands.
Lisa L KoorneefDepartment of Medicine, Division of Endocrinology, Leiden University Medical Center, Leiden, the Netherlands; Einthoven Laboratory for Experimental Vascular Medicine, Leiden University Medical Center, Leiden, the Netherlands.
Borja Martinez-TellezDepartment of Medicine, Division of Endocrinology, Leiden University Medical Center, Leiden, the Netherlands; Einthoven Laboratory for Experimental Vascular Medicine, Leiden University Medical Center, Leiden, the Netherlands; PROFITH (PROmoting FITness and Health Through Physical Activity) Research Group, Department of Physical Education and Sports, Faculty of Sport Sciences, University of Granada, Granada, Spain.
Jedrzej BurakiewiczDepartment of Radiology, C.J. Gorter Center for High Field MRI, Leiden University Medical Center, Leiden, the Netherlands.
Hermien E KanDepartment of Radiology, C.J. Gorter Center for High Field MRI, Leiden University Medical Center, Leiden, the Netherlands.
Floris H P van VeldenDepartment of Radiology, Division of Nuclear Medicine, Leiden University Medical Center, Leiden, the Netherlands.
Lenka M Pereira Arias-BoudaDepartment of Radiology, Division of Nuclear Medicine, Leiden University Medical Center, Leiden, the Netherlands; Department of Nuclear Medicine, Alrijne Hospital, Leiderdorp, the Netherlands.
Lioe-Fee de Geus-OeiDepartment of Radiology, Division of Nuclear Medicine, Leiden University Medical Center, Leiden, the Netherlands; Biomedical Photonic Imaging Group, University of Twente, Enschede, the Netherlands.
Jimmy F P BerbéeDepartment of Medicine, Division of Endocrinology, Leiden University Medical Center, Leiden, the Netherlands; Einthoven Laboratory for Experimental Vascular Medicine, Leiden University Medical Center, Leiden, the Netherlands.
Ingrid M JazetDepartment of Medicine, Division of Endocrinology, Leiden University Medical Center, Leiden, the Netherlands.
Mariëtte R BoonDepartment of Medicine, Division of Endocrinology, Leiden University Medical Center, Leiden, the Netherlands; Einthoven Laboratory for Experimental Vascular Medicine, Leiden University Medical Center, Leiden, the Netherlands. Electronic address: m.r.boon@lumc.nl.
Patrick C N RensenDepartment of Medicine, Division of Endocrinology, Leiden University Medical Center, Leiden, the Netherlands; Einthoven Laboratory for Experimental Vascular Medicine, Leiden University Medical Center, Leiden, the Netherlands.
Leiden University Medical Center · NLAlrijne Ziekenhuis · NL

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

aims/hypothesisBrown adipose tissue (BAT) improves energy metabolism by combusting glucose and lipids into heat. Agonism of the glucagon-like peptide-1 receptor (GLP-1R) within the central nervous system activates BAT in mice. Moreover, in patients with type 2 diabetes, GLP-1R agonism lowers body weight and improves glucose and lipid levels, possibly involving BAT activation. Interestingly, people from South Asian descent are prone to develop cardiometabolic disease. We studied the effect of GLP-1R agonism on BAT in humans, specifically in South Asians and Europids without obesity or type 2 diabetes.

methodsTwelve Dutch South Asian and 12 age- and BMI-matched Europid nondiabetic men received 12 weeks extended-release exenatide (Bydureon) in this single-arm prospective study. Before and after treatment, BAT was visualized by a cold-induced [

resultsSince the effect of exenatide on metabolic parameters did not evidently differ between ethnicities, data of all participants were pooled. Exenatide decreased body weight (-1.5 ± 0.4 kg, p < 0.01), without affecting REE or substrate oxidation, and transiently decreased appetite ratings during the first weeks. Exenatide also lowered triglycerides (-15%, p < 0.05) and total cholesterol (-5%, p < 0.05), and tended to lower glucose levels. Notably, exenatide increased BAT metabolic volume (+28%, p < 0.05) and mean standardized uptake value (+11%, p < 0.05) ([ CONCLUSIONS/

interpretationWe show for the first time that GLP-1R agonism increases [ TRIAL REGISTRY: Clinicaltrials.gov NCT03002675.

Indexed as

Adipose Tissue, BrownAdultBody CompositionBody WeightEnergy MetabolismExenatideFluorodeoxyglucose F18HumansMaleOxidation-ReductionOxidative PhosphorylationPositron Emission Tomography Computed TomographyRestYoung AdultExenatideFluorodeoxyglucose F18[(18)F]FDG-PET/CTBrown adipose tissueGlucagon-like peptide-1 receptor agonismLipid metabolismMRIWeight loss

Identifiers

PMID31982480
OpenAlexW3002699912

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.