Evidence mapPaperPMID 32039382Full record

ReviewJHEP reports : innovation in hepatology2019

From NASH to diabetes and from diabetes to NASH: Mechanisms and treatment options.

Amalia Gastaldelli, Kenneth Cusi

Registry-linked trialOpen access · goldAbstract readReview
In one paragraph

Review in JHEP reports : innovation in hepatology, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. It is linked to trial NCT04501406 (Effect of Low-Dose Pioglitazone in Patients With Nonalcoholic Steatohepatitis), which is not on this map. Cited by 214 papers, 5 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
214citing papers in PubMed, 5 pooled it
28.6field-weighted citation impact, top 1% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

NCT04501406 phase2recruitingnot on this mapstarted 2020, after this paper: background citation

Effect of Low-Dose Pioglitazone in Patients With Nonalcoholic Steatohepatitis (NASH)

TypeinterventionalSponsorUniversity of FloridaRan2020 to 2027Enrolled166ConditionsType 2 Diabetes Mellitus (T2DM), Nonalcoholic SteatohepatitisArmsPioglitazone, Placebo
3 · Its place in the literature

Who cites it

214 citing papers in PubMed, 5 syntheses or guidelines pooled it, 425 citations in OpenAlex.

  1. Pooled it
  2. Pooled it
  3. Frontiers in nutrition · 2023
    Pooled it
  4. Pooled it
  5. Guideline
  6. Effect of Essential Phospholipids in Metabolic Dysfunction-Associated Steatotic Liver Disease: A Randomised Phase 4 Clinical Trial.Liver international : official journal of the International Association for the Study of the Liver · 2026
    Trial
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  15. Liver fibrosis risk: a silent threat in type 1 diabetes mellitus.Journal of endocrinological investigation · 2026
    Article
  16. Review
  17. Review
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  19. Review
  20. Article

154 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors at 2 institutions in 2 countries.

Amalia GastaldelliCardiometabolic Risk Unit, Institute of Clinical Physiology, National Research Council, Pisa, Italy.
Kenneth CusiDivision of Endocrinology, Diabetes and Metabolism, The University of Florida, and Malcom Randall Veterans Administration Medical Center, Gainesville, Florida.
Istituto di Fisiologia Clinica · ITMalcom Randall VA Medical Center · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The worldwide prevalence of non-alcoholic fatty liver disease (NAFLD) is estimated to have reached 25% or more in adults. NAFLD is prevalent in obese individuals, but may also affect non-obese insulin-resistant individuals. NAFLD is associated with a 2- to 3-fold increased risk of developing type 2 diabetes (T2D), which may be higher in patients with more severe liver disease - fibrosis increases this risk. In NAFLD, not only the close association with obesity, but also the impairment of many metabolic pathways, including decreased hepatic insulin sensitivity and insulin secretion, increase the risk of developing T2D and related comorbidities. Conversely, patients with diabetes have a higher prevalence of steatohepatitis, liver fibrosis and end-stage liver disease. Genetics and mechanisms involving dysfunctional adipose tissue, lipotoxicity and glucotoxicity appear to play a role. In this review, we discuss the altered pathophysiological mechanisms that underlie the development of T2D in NAFLD and vice versa. Although there is no approved therapy for the treatment of NASH, we discuss pharmacological agents currently available to treat T2D that could potentially be useful for the management of NASH.

Identifiers

PMID32039382
PMCPMC7001557
OpenAlexW2963543751

What Socratic holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.