Evidence mapPaperPMID 32067063Full record

ReviewJournal of molecular medicine (Berlin, Germany)2020

Paracrine signaling in islet function and survival.

Sean M Hartig, Aaron R Cox

Erratum issuedOpen access · greenAbstract readReview
In one paragraph

Review in Journal of molecular medicine (Berlin, Germany), 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. An erratum has been issued. Cited by 40 papers.

0numbers the graph read from it
0cells of the map it votes in
40citing papers in PubMed
4.5field-weighted citation impact, top 5% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

40 citing papers in PubMed, 50 citations in OpenAlex.

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4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

2 authors at 1 institution in 1 country.

Sean M HartigDivision of Endocrinology, Diabetes, and Metabolism, Department of Medicine, Baylor College of Medicine, Houston, TX, 77030, USA.ORCID 0000-0002-2695-2072
Aaron R CoxDivision of Endocrinology, Diabetes, and Metabolism, Department of Medicine, Baylor College of Medicine, Houston, TX, 77030, USA. racox@bcm.edu.ORCID 0000-0002-3330-5746
Baylor College of Medicine · US

Funding

Metabolic Impacts of Type II Interferon Signals in ObesityR01DK114356 · BAYLOR COLLEGE OF MEDICINE · 2025 to 2025
$498k
NIDDK NIH HHS R01 DK114356
6 · The paper itself

Abstract

The pancreatic islet is a dense cellular network comprised of several cell types with endocrine function vital in the control of glucose homeostasis, metabolism, and feeding behavior. Within the islet, endocrine hormones also form an intricate paracrine network with supportive cells (endothelial, neuronal, immune) and secondary signaling molecules regulating cellular function and survival. Modulation of these signals has potential consequences for diabetes development, progression, and therapeutic intervention. Beta cell loss, reduced endogenous insulin secretion, and dysregulated glucagon secretion are hallmark features of both type 1 and 2 diabetes that not only impact systemic regulation of glucose, but also contribute to the function and survival of cells within the islet. Advancing research and technology have revealed new islet biology (cellular identity and transcriptomes) and identified previously unrecognized paracrine signals and mechanisms (somatostatin and ghrelin paracrine actions), while shifting prior views of intraislet communication. This review will summarize the paracrine signals regulating islet endocrine function and survival, the disruption and dysfunction that occur in diabetes, and potential therapeutic targets to preserve beta cell mass and function.

Indexed as

Paracrine CommunicationSignal TransductionAnimalsCell SurvivalGhrelinGlucagon-Secreting CellsGlucoseGlycogenHumansInsulinInsulin-Secreting CellsIslets of LangerhansPancreatic PolypeptideSomatostatinSomatostatin-Secreting CellsGhrelinGlucoseGlycogenInsulinPancreatic PolypeptideSomatostatinDiabetesIslet biologyIslet functionParacrine signalingSurvival

Identifiers

PMID32067063
PMCPMC7899133
OpenAlexW3007084158

What Socratic holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.