Evidence mapPaperPMID 32161192Full record

ArticleJCI insight2020

Impaired skeletal muscle mitochondrial bioenergetics and physical performance in chronic kidney disease.

Bryan Kestenbaum, Jorge Gamboa, Sophia Liu, Amir S Ali, Eric Shankland, Thomas Jue, Cecilia Giulivi, Lucas R Smith, Jonathan Himmelfarb, Ian H de Boer and 2 more

Open access · goldAbstract read
In one paragraph

Article in JCI insight, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 51 papers.

0numbers the graph read from it
0cells of the map it votes in
51citing papers in PubMed
7.1field-weighted citation impact, top 2% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

51 citing papers in PubMed, 79 citations in OpenAlex.

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  5. High prevalence of sarcopenia, osteoporosis and osteosarcopenia in patients with chronic kidney disease compared with healthy controls.Osteoporosis international : a journal established as result of cooperation between the European Foundation for Osteoporosis and the National Osteoporosis Foundation of the USA · 2026
    Article
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  11. Multiparametric quantitative magnetic resonance imaging of skeletal muscle in CKD.American journal of physiology. Renal physiology · 2025
    Article
  12. Intermuscular Adipose Tissue in CKD: A Window into Muscle Quality and Health?Clinical journal of the American Society of Nephrology : CJASN · 2025
    Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

12 authors at 4 institutions in 1 country.

Bryan KestenbaumDivision of Nephrology, Department of Medicine, and.
Jorge GamboaDepartment of Medicine, Vanderbilt University Medical Center, Nashville, Tennessee, USA.
Sophia LiuDepartment of Radiology, University of Washington, Seattle, Washington, USA.
Amir S AliDepartment of Radiology, University of Washington, Seattle, Washington, USA.
Eric ShanklandDepartment of Radiology, University of Washington, Seattle, Washington, USA.
Thomas JueDepartment of Biochemistry and Molecular Medicine, School of Medicine.
Cecilia GiuliviDepartment of Molecular Biosciences, School of Veterinary Medicine, and.
Lucas R SmithDepartment of Physical Medicine and Rehabilitation, School of Medicine, UCD, Davis, California, USA.
Jonathan HimmelfarbDivision of Nephrology, Department of Medicine, and.
Ian H de BoerDivision of Nephrology, Department of Medicine, and.
Kevin ConleyDepartment of Radiology, University of Washington, Seattle, Washington, USA.
Baback RoshanravanDivision of Nephrology, Department of Medicine, School of Medicine, UCD, Sacramento, California, USA.
University of Washington · USUniversity of California, Davis · USUniversity of California Davis Medical Center · USVanderbilt University Medical Center · US

Funding

NIDDK NIH HHS K23 DK099442NIDDK NIH HHS K23 DK100533NIDDK NIH HHS R01 DK099199NIDDK NIH HHS R01 DK101509NIDDK NIH HHS R03 DK114502
6 · The paper itself

Abstract

The maintenance of functional independence is the top priority of patients with chronic kidney disease (CKD). Defects in mitochondrial energetics may compromise physical performance and independence. We investigated associations of the presence and severity of kidney disease with in vivo muscle energetics and the association of muscle energetics with physical performance. We performed measures of in vivo leg and hand muscle mitochondrial capacity (ATPmax) and resting ATP turnover (ATPflux) using 31phosphorus magnetic resonance spectroscopy and oxygen uptake (O2 uptake) by optical spectroscopy in 77 people (53 participants with CKD and 24 controls). We measured physical performance using the 6-minute walk test. Participants with CKD had a median estimated glomerular filtration rate (eGFR) of 33 ml/min per 1.73 m2. Participants with CKD had a -0.19 mM/s lower leg ATPmax compared with controls but no difference in hand ATPmax. Resting O2 uptake was higher in CKD compared with controls, despite no difference in ATPflux. ATPmax correlated with eGFR and serum bicarbonate among participants with GFR <60. ATPmax of the hand and leg correlated with 6-minute walking distance. The presence and severity of CKD associate with muscle mitochondrial capacity. Dysfunction of muscle mitochondrial energetics may contribute to reduced physical performance in CKD.

Indexed as

Energy MetabolismPhysical Functional PerformanceAdenosine TriphosphateAgedFemaleGlomerular Filtration RateHumansMagnetic Resonance SpectroscopyMaleMiddle AgedMitochondria, MuscleMuscle, SkeletalOxygen ConsumptionRenal Insufficiency, ChronicSeverity of Illness IndexAdenosine TriphosphateAgingDiabetesMitochondriaMuscleNephrology

Identifiers

PMID32161192
PMCPMC7141399
OpenAlexW3011095353

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.