Evidence mapPaperPMID 32320071Full record

ReviewImmunological reviews2020

Microbiome, bile acids, and obesity: How microbially modified metabolites shape anti-tumor immunity.

Laura M Sipe, Mehdi Chaib, Ajeeth K Pingili, Joseph F Pierre, Liza Makowski

Open access · greenAbstract readReview
In one paragraph

Review in Immunological reviews, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 52 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
52citing papers in PubMed, 1 pooled it
3.7field-weighted citation impact, top 6% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

52 citing papers in PubMed, 1 synthesis or guideline pooled it, 69 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 1 institution in 1 country.

Laura M SipeDivision of Hematology and Oncology, Department of Medicine, College of Medicine, University of Tennessee Health Science Center, Memphis, TN, USA.
Mehdi ChaibDepartment of Pharmaceutical Sciences, College of Pharmacy, University of Tennessee Health Science Center, Memphis, TN, USA.
Ajeeth K PingiliDivision of Hematology and Oncology, Department of Medicine, College of Medicine, University of Tennessee Health Science Center, Memphis, TN, USA.
Joseph F PierreDepartment of Pediatrics, College of Medicine, University of Tennessee Health Science Center, Memphis, TN, USA.ORCID 0000-0002-4248-1290
Liza MakowskiDivision of Hematology and Oncology, Department of Medicine, College of Medicine, University of Tennessee Health Science Center, Memphis, TN, USA.ORCID 0000-0002-5337-8037
University of Tennessee Health Science Center · US

Funding

Role of microbially-derived metabolites in anti-tumor immunity in breast cancerR01CA253329 · UNIVERSITY OF TENNESSEE HEALTH SCI CTR · 2025 to 2025
$488k
Obesity-driven Metabolic and Molecular Biomarkers of Metformin Response in Endometrial CancerR37CA226969 · NCI · UNIV OF NORTH CAROLINA CHAPEL HILL · PI Victoria Lin Bae-Jump · 2022 to 2022
$356k
NCI NIH HHS F32 CA250192NCI NIH HHS R01 CA253329NCI NIH HHS R37 CA226969
6 · The paper itself

Abstract

Bile acids (BAs) are known facilitators of nutrient absorption but recent paradigm shifts now recognize BAs as signaling molecules regulating both innate and adaptive immunity. Bile acids are synthesized from cholesterol in the liver with subsequent microbial modification and fermentation adding complexity to pool composition. Bile acids act on several receptors such as Farnesoid X Receptor and the G protein-coupled BA receptor 1 (TGR5). Interestingly, BA receptors (BARs) are expressed on immune cells and activation either by BAs or BAR agonists modulates innate and adaptive immune cell populations skewing their polarization toward a more tolerogenic anti-inflammatory phenotype. Intriguingly, recent evidence also suggests that BAs promote anti-tumor immune response through activation and recruitment of tumoricidal immune cells such as natural killer T cells. These exciting findings have redefined BA signaling in health and disease wherein they may suppress inflammation on the one hand, yet promote anti-tumor immunity on the other hand. In this review, we provide our readers with the most recent understanding of the interaction of BAs with the host microbiome, their effect on innate and adaptive immunity in health and disease with a special focus on obesity, bariatric surgery-induced weight loss, and immune checkpoint blockade in cancer.

Indexed as

MicrobiotaAnimalsBariatric SurgeryBile Acids and SaltsBiomarkersDisease SusceptibilityEnergy MetabolismGastrointestinal MicrobiomeHumansImmune Checkpoint InhibitorsImmune Checkpoint ProteinsImmunomodulationNeoplasmsObesityPrognosisSignal TransductionBile Acids and SaltsBiomarkersImmune Checkpoint InhibitorsImmune Checkpoint Proteinsbariatric surgeryimmunometabolismmicrobiomemitochondriaobesitytumor microenvironment

Identifiers

PMID32320071
PMCPMC7841960
OpenAlexW3016858080

What Socratic holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.