Evidence map›Paper›PMID 32365683›Full record

ArticleNutrients2020

Association of the

Nuria Perez-Diaz-Del-Campo, Itziar Abete, Irene Cantero, Bertha Araceli Marin-Alejandre, J Ignacio Monreal, Mariana Elorz, José Ignacio Herrero, Alberto Benito-Boillos, Jose I Riezu-Boj, Fermín I Milagro and 3 more

Open access · goldAbstract read
In one paragraph

Article in Nutrients, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
2.0field-weighted citation impact, top 13% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed, 18 citations in OpenAlex.

  1. Review
  2. Article
  3. Article
  4. Review
  5. Review
  6. Article
  7. Article
  8. Mitochondrial Mutations and Genetic Factors Determining NAFLD Risk.International journal of molecular sciences · 2021
    Review
  9. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

13 authors at 3 institutions in 1 country.

Nuria Perez-Diaz-Del-CampoDepartment of Nutrition, Food Science and Physiology, Faculty of Pharmacy and Nutrition, University of Navarra, 31008 Pamplona, Spain.ORCID 0000-0003-0479-8825
Itziar AbeteDepartment of Nutrition, Food Science and Physiology, Faculty of Pharmacy and Nutrition, University of Navarra, 31008 Pamplona, Spain.ORCID 0000-0002-6475-5387
Irene CanteroDepartment of Nutrition, Food Science and Physiology, Faculty of Pharmacy and Nutrition, University of Navarra, 31008 Pamplona, Spain.
Bertha Araceli Marin-AlejandreDepartment of Nutrition, Food Science and Physiology, Faculty of Pharmacy and Nutrition, University of Navarra, 31008 Pamplona, Spain.ORCID 0000-0003-0741-8197
J Ignacio MonrealNavarra Institute for Health Research (IdiSNA), 31008 Pamplona, Spain.
Mariana ElorzNavarra Institute for Health Research (IdiSNA), 31008 Pamplona, Spain.
José Ignacio HerreroNavarra Institute for Health Research (IdiSNA), 31008 Pamplona, Spain.ORCID 0000-0002-9076-6717
Alberto Benito-BoillosNavarra Institute for Health Research (IdiSNA), 31008 Pamplona, Spain.
Jose I Riezu-BojDepartment of Nutrition, Food Science and Physiology, Faculty of Pharmacy and Nutrition, University of Navarra, 31008 Pamplona, Spain.ORCID 0000-0002-1885-8457
Fermín I MilagroDepartment of Nutrition, Food Science and Physiology, Faculty of Pharmacy and Nutrition, University of Navarra, 31008 Pamplona, Spain.ORCID 0000-0002-3228-9916
Josep A TurBiomedical Research Centre Network in Physiopathology of Obesity and Nutrition (CIBERobn), Instituto de Salud Carlos III, 28029 Madrid, Spain.ORCID 0000-0002-6940-0761
J Alfredo MartinezDepartment of Nutrition, Food Science and Physiology, Faculty of Pharmacy and Nutrition, University of Navarra, 31008 Pamplona, Spain.ORCID 0000-0001-5218-6941
M Angeles ZuletDepartment of Nutrition, Food Science and Physiology, Faculty of Pharmacy and Nutrition, University of Navarra, 31008 Pamplona, Spain.ORCID 0000-0002-3926-0892
Instituto de Salud Carlos III · ESNavarre Institute of Health Research · ESUniversidad de Navarra · ES

Funding

Centro de Investigación Biomédica en Red-Fisiopatología de la Obesidad y Nutrición CB12/03/3002Fundació La Marató de TV3 201630.10Health Department of the Government of Navarra 61/2015
6 · The paper itself

Abstract

Non-alcoholic fatty liver disease (NAFLD) is a major cause of liver disease worldwide. Some genetic variants might be involved in the progression of this disease. The study hypothesized that individuals with the rs7359397 T allele have a higher risk of developing severe stages of NAFLD compared with non-carriers where dietary intake according to genotypes could have a key role on the pathogenesis of the disease.

Indexed as

Genetic Association StudiesPolymorphism, GeneticAdaptor Proteins, Signal TransducingAllelesDisease ProgressionFatty LiverFemaleGenetic Predisposition to DiseaseHumansMaleMiddle AgedNon-alcoholic Fatty Liver DiseaseObesityRiskSeverity of Illness IndexAdaptor Proteins, Signal TransducingSH2B1 protein, humanNAFLDobesitypolymorphismsSH2B1steatosis

Identifiers

PMID32365683
PMCPMC7282006
OpenAlexW3021025582

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.