Evidence map›Paper›PMID 32393068›Full record

ReviewAnnals of medicine2020

Role of sodium glucose co-transporter 2 inhibitors in patients with heart failure: an elusive mechanism.

Hafiz Imran, William Nester, Islam Y Elgendy, Marwan Saad

Open access · bronzeAbstract readReview
In one paragraph

Review in Annals of medicine, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
0.1field-weighted citation impact, top 50% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed, 1 citations in OpenAlex.

  1. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 2 institutions in 1 country.

Hafiz ImranCardiovascular Institute, Warren Alpert Medical School at Brown University, Providence, RI, USA.
William NesterCardiovascular Institute, Warren Alpert Medical School at Brown University, Providence, RI, USA.
Islam Y ElgendyDivision of Cardiology, Massachusetts General Hospital, Boston, MA, USA.ORCID 0000-0001-9853-7591
Marwan SaadCardiovascular Institute, Warren Alpert Medical School at Brown University, Providence, RI, USA.
Brown University · USHarvard University · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Heart failure (HF) is a major cause of morbidity and mortality worldwide, and the burden of HF continues to rise. There has been an interest in sodium-glucose co-transporter-2 (SGLT2) inhibitors for their role in reducing HF hospitalizations in pivotal trials. Since these agents were approved by the Food and Drug Administration for the management of diabetes mellitus, multiple small trials and analyses have tried to explain the underlying beneficial mechanisms in HF . In this review, we discuss different mechanisms by which SGLT2 inhibitors play hemodynamic, metabolic, and cellular roles in different HF phenotypes. We also address issues pertaining to the safety of these relatively newer agents.KEY MESSAGESSGLT2 inhibitors are associated with a reduction in HF hospitalizations in both diabetics and non-diabetics.The beneficial role of SGLT2 inhibitors in reducing HF hospitalization is observed among participants with established cardiovascular disease/HF and at-risk population.SGLT2 inhibitors pose an important role in renal protection, another mechanism by which these medications can be helpful in HF patients.

Indexed as

Diabetes Mellitus, Type 2Heart FailureHospitalizationHumansRisk FactorsSodium-Glucose Transporter 2 InhibitorsSodium-Glucose Transporter 2 InhibitorsCardiovascular outcomesdiabetesheart failuresodium glucose co-transporter 2 inhibitors

Identifiers

PMID32393068
PMCPMC7877993
OpenAlexW3025601467

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.