Evidence mapPaperPMID 32494148Full record

ReviewVascular health and risk management2020

Vascular Calcification: An Important Understanding in Nephrology.

Sepideh Zununi Vahed, Soroush Mostafavi, Seyed Mahdi Hosseiniyan Khatibi, Mohammadali M Shoja, Mohammadreza Ardalan

Open access · goldAbstract readReview
In one paragraph

Review in Vascular health and risk management, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 29 papers.

0numbers the graph read from it
0cells of the map it votes in
29citing papers in PubMed
6.0field-weighted citation impact, top 3% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

29 citing papers in PubMed, 53 citations in OpenAlex.

  1. Article
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  8. Clinical Potential of Hydrogen Sulfide in Peripheral Arterial Disease.International journal of molecular sciences · 2023
    Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 2 institutions in 2 countries.

Sepideh Zununi VahedKidney Research Center, Tabriz University of Medical Sciences, Tabriz, Iran.ORCID 0000-0003-0179-4562
Soroush MostafaviKidney Research Center, Tabriz University of Medical Sciences, Tabriz, Iran.ORCID 0000-0002-8342-7821
Seyed Mahdi Hosseiniyan KhatibiKidney Research Center, Tabriz University of Medical Sciences, Tabriz, Iran.
Mohammadali M ShojaDepartment of Surgery, University of Texas Medical Branch, Galveston, TX, USA.
Mohammadreza ArdalanKidney Research Center, Tabriz University of Medical Sciences, Tabriz, Iran.
Tabriz University of Medical Sciences · IRThe University of Texas Medical Branch at Galveston · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Vascular calcification (VC) is a life-threatening state in chronic kidney disease (CKD). High cardiovascular mortality and morbidity of CKD cases may root from medial VC promoted by hyperphosphatemia. Vascular calcification is an active, highly regulated, and complex biological process that is mediated by genetics, epigenetics, dysregulated form of matrix mineral metabolism, hormones, and the activation of cellular signaling pathways. Moreover, gut microbiome as a source of uremic toxins (eg, phosphate, advanced glycation end products and indoxyl-sulfate) can be regarded as a potential contributor to VC in CKD. Here, an update on different cellular and molecular processes involved in VC in CKD is discussed to elucidate the probable therapeutic pathways in the future.

Indexed as

Renal EliminationVascular RemodelingAnimalsBlood VesselsHumansKidneyPrognosisRenal Insufficiency, ChronicRisk FactorsVascular Calcificationcalcificationchronic kidney diseaseCKDhyperphosphatemiauremiauremic toxins

Identifiers

PMID32494148
PMCPMC7229867
OpenAlexW3022638866

What Socratic holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.