Evidence map›Paper›PMID 32714599›Full record

ArticleNeuronal signaling2020

Cortical cells are altered by factors including bone morphogenetic protein released from a placental barrier model under altered oxygenation.

Veronica H L Leinster, Thomas J Phillips, Nicola Jones, Sharon Sanderson, Katja Simon, Jon Hanley, Charles Patrick Case

Open access · goldAbstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in Neuronal signaling, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact, top 97% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed, 0 citations in OpenAlex.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 3 institutions in 2 countries.

Veronica H L LeinsterSchool of Clinical Sciences, University of Bristol, Southmead Hospital, Bristol, U.K.
Thomas J PhillipsSchool of Clinical Sciences, University of Bristol, Southmead Hospital, Bristol, U.K.ORCID 0000-0002-4709-2550
Nicola JonesSchool of Clinical Sciences, University of Bristol, Southmead Hospital, Bristol, U.K.
Sharon SandersonTranslational Immunology Laboratory, NIHR BRC, John Radcliffe Hospital, Oxford, U.K.
Katja SimonTranslational Immunology Laboratory, NIHR BRC, John Radcliffe Hospital, Oxford, U.K.
Jon HanleySchool of Biochemistry, University of Bristol, Bristol, U.K.
Charles Patrick CaseSchool of Clinical Sciences, University of Bristol, Southmead Hospital, Bristol, U.K.
Southmead Hospital · GBJohn Radcliffe Hospital · GBUniversity of Bristol · GB

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Episodes of hypoxia and hypoxia/reoxygenation during foetal development have been associated with increased risk of neurodevelopmental conditions presenting in later life. The mechanism for this is not understood; however, several authors have suggested that the placenta plays an important role. Previously we found both placentas from a maternal hypoxia model and pre-eclamptic placentas from patients release factors lead to a loss of dendrite complexity in rodent neurons. Here to further explore the nature and origin of these secretions we exposed a simple

Indexed as

AstrocytesBeWoHypoxiaNeurodevelopmentNeuronesRat

Identifiers

PMID32714599
PMCPMC7363303
OpenAlexW3013756396

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.