Evidence mapPaperPMID 32716607Full record

ArticleObesity (Silver Spring, Md.)2020

Loss of Uncoupling Protein 3 Attenuates Western Diet-Induced Obesity, Systemic Inflammation, and Insulin Resistance in Rats.

Tyler M Lomax, Sadia Ashraf, Gizem Yilmaz, Romain Harmancey

Open access · greenAbstract read
In one paragraph

Article in Obesity (Silver Spring, Md.), 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.

0numbers the graph read from it
0cells of the map it votes in
8citing papers in PubMed
0.5field-weighted citation impact, top 36% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

8 citing papers in PubMed, 6 citations in OpenAlex.

  1. Deciphering Uncoupling Proteins in Cellular Homeostasis and Metabolic Health.International journal of biological sciences · 2026
    Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 1 institution in 1 country.

Tyler M LomaxDepartment of Physiology and Biophysics, University of Mississippi Medical Center, Jackson, Mississippi, USA.
Sadia AshrafDepartment of Physiology and Biophysics, University of Mississippi Medical Center, Jackson, Mississippi, USA.
Gizem YilmazDepartment of Physiology and Biophysics, University of Mississippi Medical Center, Jackson, Mississippi, USA.
Romain HarmanceyDepartment of Physiology and Biophysics, University of Mississippi Medical Center, Jackson, Mississippi, USA.ORCID 0000-0002-5040-4500
Jackson Memorial Hospital · US

Funding

STRUCTURAL VASCULAR ADAPTATION OF THE MICROCIRCULATIONP01HL051971 · UNIVERSITY OF MISSISSIPPI MEDICAL CENTER · 1993 to 2005
$11.3M
Mississippi INBREP20GM103476 · UNIVERSITY OF SOUTHERN MISSISSIPPI · 2025 to 2025
$4.1M
Project 003 - Lorena AmaralP20GM121334 · UNIVERSITY OF MISSISSIPPI MED CTR · 2025 to 2025
$2.6M
NHLBI NIH HHS P01 HL051971NHLBI NIH HHS R00 HL112952NHLBI NIH HHS R01 HL136438NIGMS NIH HHS P20 GM103476NIGMS NIH HHS P20 GM104357NIGMS NIH HHS P20 GM121334NIGMS NIH HHS P30 GM103328
6 · The paper itself

Abstract

objectiveUncoupling protein 3 (UCP3) is a mitochondrial carrier related to fatty acid metabolism. Although gene variants of UCP3 are associated with human obesity, their contribution to increased adiposity remains unclear. This study investigated the impact that loss of UCP3 has on diet-induced obesity in rats.

methodsMale UCP3 knockout rats (ucp3

resultsLoss of UCP3 decreased fat mass gain, white adipocytes size, and systemic inflammation. The ucp3

conclusionsLoss of UCP3 partially protects rats from diet-induced obesity. This phenotype is related to induction of a compensatory antioxidant mechanism and prevention of iBAT whitening.

Indexed as

Adipose Tissue, BrownAnimalsDiet, WesternInflammationInsulin ResistanceMaleRatsRats, Sprague-DawleyUncoupling Protein 3Ucp3 protein, ratUncoupling Protein 3

Identifiers

PMID32716607
PMCPMC7483834
OpenAlexW3045776823

What Socratic holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.