Evidence mapPaperPMID 32749600Full record

ArticleJournal of neurology2021

Nerve ultrasound in hereditary transthyretin amyloidosis: red flags and possible progression biomarkers.

Alessandro Salvalaggio, Daniele Coraci, Mario Cacciavillani, Laura Obici, Anna Mazzeo, Marco Luigetti, Francesca Pastorelli, Marina Grandis, Tiziana Cavallaro, Giulia Bisogni and 12 more

Open access · hybridAbstract read
In one paragraph

Article in Journal of neurology, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 29 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
29citing papers in PubMed, 1 pooled it
3.2field-weighted citation impact, top 7% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

29 citing papers in PubMed, 1 synthesis or guideline pooled it, 61 citations in OpenAlex.

  1. Pooled it
  2. Article
  3. Article
  4. Article
  5. Article
  6. Article
  7. Article
  8. Quantitative muscle ultrasound as a disease biomarker in hereditary transthyretin amyloidosis with polyneuropathy.Neurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology · 2024
    Article
  9. Article
  10. CMAJ : Canadian Medical Association journal = journal de l'Association medicale canadienne · 2024
    Article
  11. Early detection of nerve involvement in presymptomatic TTR mutation carriers: exploring potential markers of disease onset.Neurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology · 2024
    Article
  12. Hereditary transthyretin amyloidosis presenting with carpal tunnel syndrome.CMAJ : Canadian Medical Association journal = journal de l'Association medicale canadienne · 2024
    Article
  13. Review
  14. Article
  15. Ultrasonographic nerve enlargement in post-transplanted patient with hereditary transthyretin amyloidosis.Neurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology · 2023
    Article
  16. Article
  17. Review
  18. Cutaneous silent period in ATTRv carriers: a possible early marker of nerve damage?Neurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology · 2022
    Article
  19. Article
  20. Novel approaches to diagnosis and management of hereditary transthyretin amyloidosis.Journal of neurology, neurosurgery, and psychiatry · 2022
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

22 authors at 11 institutions in 1 country.

Alessandro SalvalaggioDepartment of Neurosciences, University of Padova, Via Giustiniani 5, 35128, Padova, Italy. salvalaggio.a@gmail.com.ORCID http://orcid.org/0000-0002-1273-7566
Daniele CoraciNeuroriabilitazione Ad Alta Intensità, Fondazione Policlinico Universitario A. Gemelli IRCCS, Rome, Italy.
Mario CacciavillaniCEMES-EMG Lab, Synlab Group, Padova, Italy.
Laura ObiciAmyloidosis Research and Treatment Centre, Fondazione IRCCS Policlinico San Matteo, Pavia, Italy.
Anna MazzeoUnit of Neurology and Neuromuscular Diseases, Department of Clinical and Experimental Medicine, University of Messina, Messina, Italy.
Marco LuigettiNeurology Unit, Fondazione Policlinico Universitario Gemelli IRCCS, Rome, Italy.
Francesca PastorelliIRCSS Istituto Scienze Neurologiche Città Di Bologna, Bologna, Italy.
Marina GrandisDepartment of Neuroscience, Rehabilitation, Ophthalmology, Genetics, Maternal and Child Health (DiNOGMI), University of Genova, Genova, Italy.
Tiziana CavallaroNeurology Unit, Department of Neuroscience, Biomedicine and Movement Sciences, University of Verona, Verona, Italy.
Giulia BisogniCentro Clinico NEMO Adulti, Roma, Italy.
Alessandro LozzaAmyloidosis Research and Treatment Centre, Fondazione IRCCS Policlinico San Matteo, Pavia, Italy.
Chiara GemelliDepartment of Neuroscience, Rehabilitation, Ophthalmology, Genetics, Maternal and Child Health (DiNOGMI), University of Genova, Genova, Italy.
Luca GentileUnit of Neurology and Neuromuscular Diseases, Department of Clinical and Experimental Medicine, University of Messina, Messina, Italy.
Mario ErmaniDepartment of Neurosciences, University of Padova, Via Giustiniani 5, 35128, Padova, Italy.
Gian Maria FabriziNeurology Unit, Department of Neuroscience, Biomedicine and Movement Sciences, University of Verona, Verona, Italy.
Rosaria PlasmatiIRCSS Istituto Scienze Neurologiche Città Di Bologna, Bologna, Italy.
Marta CampagnoloDepartment of Neurosciences, University of Padova, Via Giustiniani 5, 35128, Padova, Italy.
Francesca CastellaniDepartment of Neurosciences, University of Padova, Via Giustiniani 5, 35128, Padova, Italy.
Roberto GasparottiDepartment of Medical and Surgical Specialties, Radiological Sciences, and Public Health, University of Brescia, Brescia, Italy.
Carlo MartinoliOspedale Policlinico San Martino IRCCS, Genova, Italy.
Luca PaduaNeuroriabilitazione Ad Alta Intensità, Fondazione Policlinico Universitario A. Gemelli IRCCS, Rome, Italy.
Chiara BrianiDepartment of Neurosciences, University of Padova, Via Giustiniani 5, 35128, Padova, Italy.
University of Padua · ITAgostino Gemelli University Polyclinic · ITIstituto delle Scienze Neurologiche di Bologna · ITOspedale Policlinico San Martino · ITPoliclinico San Matteo Fondazione · ITUniversity of Messina · ITUniversity of Verona · ITCentro Clinico Nemo · ITUniversità Cattolica del Sacro Cuore · ITUniversity of Brescia · ITUniversity of Genoa · IT

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundDiagnostic delay of hereditary transthyretin amyloidosis (ATTRv, v for variant) prevents timely treatment and, therefore, concurs to the mortality of the disease. The aim of the present study was to explore with nerve ultrasound (US) possible red flags for early diagnosis in ATTRv patients with carpal tunnel syndrome (CTS) and/or polyneuropathy and in pre-symptomatic carriers.

methodsPatients and pre-symptomatic carriers with a TTR gene mutation were enrolled from seven Italian centers. Severity of CTS was assessed with neurophysiology and clinical evaluation. Median nerve cross-section area (CSA) was measured with US in ATTRv carriers with CTS (TTR-CTS). One thousand one hundred ninety-six idiopathic CTS were used as controls. Nerve US was also performed in several nerve trunks (median, ulnar, radial, brachial plexi, tibial, peroneal, sciatic, sural) in ATTRv patients with polyneuropathy and in pre-symptomatic carriers.

resultsSixty-two subjects (34 men, 28 women, mean age 59.8 years ± 12) with TTR gene mutation were recruited. With regard to CTS, while in idiopathic CTS there was a direct correlation between CTS severity and median nerve CSA (r = 0.55, p < 0.01), in the subgroup of TTR-CTS subjects (16 subjects, 5 with bilateral CTS) CSA did not significantly correlate with CTS severity (r = - 0.473). ATTRv patients with polyneuropathy showed larger CSA than pre-symptomatic carriers in several nerve sites, more pronounced at brachial plexi (p < 0.001).

conclusionsThe present study identifies nerve morphological US patterns that may help in the early diagnosis (morpho-functional dissociation of median nerve in CTS) and monitoring of pre-symptomatic TTR carriers (larger nerve CSA at proximal nerve sites, especially at brachial plexi).

Indexed as

Carpal Tunnel SyndromeAmyloid Neuropathies, FamilialBiomarkersDelayed DiagnosisFemaleHumansItalyMaleMedian NerveMiddle AgedBiomarkersAmyloidotic polyneuropathyATTRvCarpal tunnel syndromeNerve ultrasoundTransthyretin amyloidosis

Identifiers

PMID32749600
PMCPMC7815618
OpenAlexW3047354916

What Socratic holds

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LicenceCC BY
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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.