Evidence map›Paper›PMID 32793799›Full record

ArticleAlzheimer's & dementia (Amsterdam, Netherlands)2020

Untargeted metabolomics reveal dysregulations in sugar, methionine, and tyrosine pathways in the prodromal state of AD.

Ihab Hajjar, Chang Liu, Dean P Jones, Karan Uppal

Open access · goldAbstract read
In one paragraph

Article in Alzheimer's & dementia (Amsterdam, Netherlands), 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 17 papers.

0numbers the graph read from it
0cells of the map it votes in
17citing papers in PubMed
2.5field-weighted citation impact, top 10% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

17 citing papers in PubMed, 32 citations in OpenAlex.

  1. Article
  2. Efficacy ofJMIR research protocols · 2026
    Article
  3. Article
  4. Article
  5. Metabolic dysregulation in Alzheimer's disease: A brain metabolomics approach.Alzheimer's & dementia : the journal of the Alzheimer's Association · 2025
    Article
  6. Review
  7. Article
  8. Review
  9. Article
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  11. Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 1 institution in 1 country.

Ihab HajjarMedicine and Neurology Department of Neurology Emory University Atlanta Georgia USA.
Chang LiuDepartment of Medicine Emory University Atlanta Georgia USA.
Dean P JonesDepartment of Medicine Emory University Atlanta Georgia USA.
Karan UppalDepartment of Medicine Emory University Atlanta Georgia USA.
Emory University · US

Funding

The role of the renin-angiotensin-endothelial pathway in ADRF1AG051633 · NIA · EMORY UNIVERSITY · PI HAJJAR, IHAB M, QUYYUMI, ARSHED A · 2015 to 2017
$6.0M
A biomarker-driven trial of angiotensin receptor blockers for prodromal AlzheimerR01AG049752 · NIA · EMORY UNIVERSITY · PI GOLDSTEIN, FELICIA C. · 2016 to 2020
$3.8M
Building a high-resolution multi-omic AD interactome with the AMP-AD and M2OVE-AD ProjectsRF1AG057470 · NIA · EMORY UNIVERSITY · PI HAJJAR, IHAB M, SEYFRIED, NICHOLAS THOMAS · 2017 to 2017
$3.1M
Hypertension, angiotensin receptor blockers, and cognition: effects and mechanismR01AG042127 · NIA · UNIVERSITY OF SOUTHERN CALIFORNIA · PI HAJJAR, IHAB M · 2012 to 2017
$3.0M
NIA NIH HHS R01 AG042127NIA NIH HHS R01 AG049752NIA NIH HHS RF1 AG051633NIA NIH HHS RF1 AG057470
6 · The paper itself

Abstract

introductionAltered metabolism may occur years before clinical manifestations of Alzheimer's disease (AD). We used untargeted metabolomics on the cerebrospinal fluid of patients with mild cognitive impairment (MCI) to uncover metabolomic derangements.

methodsCSF from 92 normal controls and 93 MCI underwent untargeted metabolomics using high-resolution mass spectrometry with liquid chromatography. Partial least squares discriminant analysis was used followed by metabolite annotation and pathway enrichment analysis (PES). Significant features were correlated with disease phenotypes.

resultsWe identified 294 features differentially expressed between the two groups and 94 were annotated. PES showed that sugar regulation (N-glycan,

conclusionThere is a metabolic signature characterized by impairments in sugars, methionine, and tyrosine regulation in MCI. Targeting these pathways may offer new therapeutic approaches to AD.

Indexed as

Alzheimer's diseasecerebrospinal fluidmetabolismmild cognitive impairment

Identifiers

PMID32793799
PMCPMC7418891
OpenAlexW3048860489

What Socratic holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.