Evidence mapPaperPMID 32830549Full record

ArticleAmerican journal of physiology. Endocrinology and metabolism2020

Caloric restriction recovers impaired β-cell-β-cell gap junction coupling, calcium oscillation coordination, and insulin secretion in prediabetic mice.

Maria Esméria Corezola do Amaral, Vira Kravets, JaeAnn M Dwulet, Nikki L Farnsworth, Robert Piscopio, Wolfgang E Schleicher, Jose Guadalupe Miranda, Richard K P Benninger

Open access · greenAbstract read
In one paragraph

Article in American journal of physiology. Endocrinology and metabolism, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 29 papers.

0numbers the graph read from it
0cells of the map it votes in
29citing papers in PubMed
3.6field-weighted citation impact, top 6% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

29 citing papers in PubMed, 46 citations in OpenAlex.

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  20. Liraglutide increases islet CaDiabetes, obesity & metabolism · 2022
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 2 institutions in 2 countries.

Maria Esméria Corezola do AmaralGraduate Program in Biomedical Sciences, Centro Universitário da Fundação Hermínio Ometto, Araras, São Paulo, Brazil.
Vira KravetsBarbara Davis Center for Childhood Diabetes, University of Colorado Anschutz Medical Campus, Aurora, Denver, Colorado.
JaeAnn M DwuletDepartment of Bioengineering, University of Colorado Anschutz Medical Campus, Aurora, Denver, Colorado.
Nikki L FarnsworthBarbara Davis Center for Childhood Diabetes, University of Colorado Anschutz Medical Campus, Aurora, Denver, Colorado.
Robert PiscopioDepartment of Bioengineering, University of Colorado Anschutz Medical Campus, Aurora, Denver, Colorado.
Wolfgang E SchleicherDepartment of Bioengineering, University of Colorado Anschutz Medical Campus, Aurora, Denver, Colorado.
Jose Guadalupe MirandaDepartment of Bioengineering, University of Colorado Anschutz Medical Campus, Aurora, Denver, Colorado.
Richard K P BenningerBarbara Davis Center for Childhood Diabetes, University of Colorado Anschutz Medical Campus, Aurora, Denver, Colorado.
University of Colorado Anschutz Medical Campus · USCentro Universitário Herminio Ometto de Araras · BR

Funding

ZEBRAFISH TRANSGENIC CORE- NEUROLOGICAL DISORDER CENTERP30NS048154 · UNIVERSITY OF COLORADO DENVER · 2004 to 2005
$1.4M
University of Colorado Anschutz Medical Campus DRCP30DK116073 · UNIVERSITY OF COLORADO DENVER · 2025 to 2025
$1.3M
NCATS NIH HHS UL1 TR001082NIDDK NIH HHS P30 DK116073NIDDK NIH HHS R01 DK102950NIDDK NIH HHS R01 DK106412NINDS NIH HHS P30 NS048154
6 · The paper itself

Abstract

Caloric restriction can decrease the incidence of metabolic diseases, such as obesity and Type 2 diabetes mellitus. The mechanisms underlying the benefits of caloric restriction involved in insulin secretion and glucose homeostasis are not fully understood. Intercellular communication within the islets of Langerhans, mediated by Connexin36 (Cx36) gap junctions, regulates insulin secretion dynamics and glucose homeostasis. The goal of this study was to determine whether caloric restriction can protect against decreases in Cx36 gap junction coupling and altered islet function induced in models of obesity and prediabetes. C57BL6 mice were fed with a high-fat diet (HFD), showing indications of prediabetes after 2 mo, including weight gain, insulin resistance, and elevated fasting glucose and insulin levels. Subsequently, mice were submitted to 1 mo of 40% caloric restriction (2 g/day of HFD). Mice under 40% caloric restriction showed reversal in weight gain and recovered insulin sensitivity, fasting glucose, and insulin levels. In islets of mice fed the HFD, caloric restriction protected against obesity-induced decreases in gap junction coupling and preserved glucose-stimulated calcium signaling, including Ca

Indexed as

Calcium SignalingCaloric RestrictionInsulin SecretionAnimalsCell CommunicationConnexinsDiet, High-FatGap Junction delta-2 ProteinGap JunctionsInsulin ResistanceInsulin-Secreting CellsMaleMiceMice, Inbred C57BLPrediabetic StateConnexinsGap Junction delta-2 Proteincalcium coordinationConnexin36diabetesdiet-induced obesityislets of Langerhans

Identifiers

PMID32830549
PMCPMC7750515
OpenAlexW3081067659

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.