Evidence mapPaperPMID 32893859Full record

Observational studyBioscience reports2020

Inflammatory and endothelial dysfunction indices among Egyptian females with obesity classes I-III.

Amal Ahmed Mohamed, Wafaa Gh Shousha, Moushira Erfan Zaki, Hala T El-Bassyouni, Hadeel El-Hanafi, Sara M Abdo

Open access · goldAbstract readObservational Study
In one paragraph

Observational study in Bioscience reports, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
0.6field-weighted citation impact, top 30% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed, 8 citations in OpenAlex.

  1. Article
  2. Article
  3. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 4 institutions in 1 country.

Amal Ahmed MohamedBiochemistry Department, National Hepatology and Tropical Medicine Research Institute, Cairo, Egypt.
Wafaa Gh ShoushaChemistry Department, Division of Biochemistry, Faculty of Science, Helwan University, Cairo, Egypt.
Moushira Erfan ZakiBiological Anthropology Department, National Research Centre, Cairo, Egypt.
Hala T El-BassyouniClinical Genetics Department, National Research Centre, Cairo, Egypt.
Hadeel El-HanafiClinical and Chemical Pathology Department, Faculty of Medicine, Cairo University, Egypt.
Sara M AbdoChemistry Department, Division of Biochemistry, Faculty of Science, Helwan University, Cairo, Egypt.
Helwan University · EGNational Research Centre · EGCairo University · EGNational Water Research Center · EG

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundObesity is an alarming threat to health in Egypt. More than one in three Egyptians is obese, the highest rate in the world. We aimed to delineate the variability of inflammation and endothelial dysfunction markers among Egyptian females with different obesity classes.

methodsOut of 130 females, 70 were categorized into three obesity groups: Class I, body mass index (BMI) 30-34.9 kg/m2; Class II, BMI 35-39.9 kg/m2 and Class III BMI ≥ 40 kg/m2, besides 60 control subjects. Anthropometric measurements were recorded and serum levels of tumor necrosis factor-α (TNF-α), C-reactive protein (CRP), interleukin (IL) 6 (IL-6), IL-12, soluble intercellular adhesion molecule 1 (sICAM-1) and soluble vascular adhesion molecule 1 (sVCAM-1) were assessed among participants.

resultsIn all three classes of obesity, significant increase (P <0.05) in BMI, waist-hip ratio, fat mass and body fat mass % were noted. CRP and sVCAM-1 levels were increased among the three obesity groups. TNF-α levels were increased in class II and III obesity groups. IL-6 and IL-12 levels were elevated in class I and class III groups. While, ICAM-1 levels were increased in class III obesity group.

conclusionBased on individuals' BMI, serum levels of TNF-α, CRP, IL-6, IL-12, sVCAM-1 and sICAM-1 are differentially altered with the progression of obesity. We strongly support the hypothesis that, as the obesity rate is still mounting, a subclinical inflammatory reaction has a role in pathogenesis of obesity and emphasize the elevation of endothelial dysfunction in individuals with obesity.

Indexed as

AdultBiomarkersBody Mass IndexC-Reactive ProteinDisease ProgressionEgyptEndothelium, VascularFemaleHumansInflammationInterleukin-12Interleukin-6ObesitySeverity of Illness IndexTumor Necrosis Factor-alphaVascular Cell Adhesion Molecule-1BiomarkersC-Reactive ProteinIL6 protein, humanInterleukin-12Interleukin-6TNF protein, humanTumor Necrosis Factor-alphaVascular Cell Adhesion Molecule-1body mass indexendothelial dysfunctionIL-12inflammationObesityTNF-α

Identifiers

PMID32893859
PMCPMC7507597
OpenAlexW3083880268

What Socratic holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.