Evidence mapPaperPMID 32961860Full record

ReviewInternational journal of molecular sciences2020

Pharmacogenetics of Type 2 Diabetes-Progress and Prospects.

Yulia A Nasykhova, Ziravard N Tonyan, Anastasiia A Mikhailova, Maria M Danilova, Andrey S Glotov

Open access · goldFull text readReview
In one paragraph

Review in International journal of molecular sciences, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 28 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
28citing papers in PubMed, 1 pooled it
4.3field-weighted citation impact, top 5% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

28 citing papers in PubMed, 1 synthesis or guideline pooled it, 59 citations in OpenAlex.

  1. Pooled it
  2. Trial
  3. Article
  4. Review
  5. Article
  6. Review
  7. Review
  8. Article
  9. Therapeutic Potential ofJournal of diabetes research · 2025
    Review
  10. Review
  11. Article
  12. Article
  13. Advances in the Insulin-Heart Axis: Current Therapies and Future Directions.International journal of molecular sciences · 2024
    Review
  14. Article
  15. Article
  16. Biomedicines · 2024
    Article
  17. Review
  18. Article
  19. Article
  20. Effects of theJournal of diabetes and metabolic disorders · 2022
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 2 institutions in 1 country.

Yulia A NasykhovaDepartment of Genomic Medicine, D.O. Ott's Institute of Obstetrics, Gynecology and Reproductology, 199034 Saint-Petersburg, Russia.ORCID 0000-0002-3543-4963
Ziravard N TonyanDepartment of Genomic Medicine, D.O. Ott's Institute of Obstetrics, Gynecology and Reproductology, 199034 Saint-Petersburg, Russia.ORCID 0000-0001-9050-5886
Anastasiia A MikhailovaDepartment of Genomic Medicine, D.O. Ott's Institute of Obstetrics, Gynecology and Reproductology, 199034 Saint-Petersburg, Russia.
Maria M DanilovaDepartment of Genomic Medicine, D.O. Ott's Institute of Obstetrics, Gynecology and Reproductology, 199034 Saint-Petersburg, Russia.
Andrey S GlotovDepartment of Genomic Medicine, D.O. Ott's Institute of Obstetrics, Gynecology and Reproductology, 199034 Saint-Petersburg, Russia.ORCID 0000-0002-7465-4504
St Petersburg University · RUResearch Institute of Obstetrics and Gynecology named after D.O. Ott · RU

Funding

Estonia - Russia Cross Border Cooperation Programme 2014-2020 ER24Ministry of Science and Higher Education of the Russian Federation АААА-А20-120041390028-0
6 · The paper itself

Abstract

Type 2 diabetes mellitus (T2D) is a chronic metabolic disease resulting from insulin resistance and progressively reduced insulin secretion, which leads to impaired glucose utilization, dyslipidemia and hyperinsulinemia and progressive pancreatic beta cell dysfunction. The incidence of type 2 diabetes mellitus is increasing worldwide and nowadays T2D already became a global epidemic. The well-known interindividual variability of T2D drug actions such as biguanides, sulfonylureas/meglitinides, DPP-4 inhibitors/GLP1R agonists and SGLT-2 inhibitors may be caused, among other things, by genetic factors. Pharmacogenetic findings may aid in identifying new drug targets and obtaining in-depth knowledge of the causes of disease and its physiological processes, thereby, providing an opportunity to elaborate an algorithm for tailor or precision treatment. The aim of this article is to summarize recent progress and discoveries for T2D pharmacogenetics and to discuss the factors which limit the furthering accumulation of genetic variability knowledge in patient response to therapy that will allow improvement the personalized treatment of T2D.

Indexed as

PharmacogeneticsBenzamidesDiabetes Mellitus, Type 2Dipeptidyl-Peptidase IV InhibitorsGlucagon-Like Peptide-1 ReceptorGlucagon-Like Peptide-1 Receptor AgonistsHumansHypoglycemic AgentsMetforminSodium-Glucose Transporter 2 InhibitorsSulfonylurea CompoundsBenzamidesDipeptidyl-Peptidase IV InhibitorsGLP1R protein, humanGlucagon-Like Peptide-1 ReceptorGlucagon-Like Peptide-1 Receptor AgonistsHypoglycemic AgentsmeglitinideMetforminSodium-Glucose Transporter 2 InhibitorsSulfonylurea CompoundsDPP-4 inhibitorsGLP1R agonistsmeglitinidesmetforminpharmacogeneticspolymorphismSGLT-2 inhibitorssulfonylureastype 2 diabetes

Identifiers

PMID32961860
PMCPMC7555942
OpenAlexW3087082719

What Socratic holds

Textfull text, public
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.