Evidence mapPaperPMID 32962281Full record

ReviewInternational journal of molecular sciences2020

The Role of microRNAs in Metabolic Syndrome-Related Oxidative Stress.

Adam Włodarski, Justyna Strycharz, Adam Wróblewski, Jacek Kasznicki, Józef Drzewoski, Agnieszka Śliwińska

Open access · goldAbstract readReview
In one paragraph

Review in International journal of molecular sciences, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 48 papers.

0numbers the graph read from it
0cells of the map it votes in
48citing papers in PubMed
3.3field-weighted citation impact, top 6% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

48 citing papers in PubMed, 85 citations in OpenAlex.

  1. Review
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  7. Review
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  9. Review
  10. MicroRNA Profile in Menopausal Women with Insomnia: A Pilot Study.Bulletin of experimental biology and medicine · 2025
    Article
  11. Review
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  15. Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 1 institution in 1 country.

Adam WłodarskiDepartment of Internal Diseases, Diabetology and Clinical Pharmacology, Medical University of Lodz, 92-213 Lodz, Poland.ORCID 0000-0002-8691-7551
Justyna StrycharzDepartment of Medical Biochemistry, Medical University of Lodz, 92-215 Lodz, Poland.ORCID 0000-0002-3817-0377
Adam WróblewskiDepartment of Medical Biochemistry, Medical University of Lodz, 92-215 Lodz, Poland.ORCID 0000-0002-6414-7111
Jacek KasznickiDepartment of Internal Diseases, Diabetology and Clinical Pharmacology, Medical University of Lodz, 92-213 Lodz, Poland.
Józef DrzewoskiCentral Teaching Hospital of the Medical University of Lodz, 92-213 Lodz, Poland.
Agnieszka ŚliwińskaDepartment of Nucleic Acid Biochemistry, Medical University of Lodz, 92-213 Lodz, Poland.ORCID 0000-0002-6864-0704
Medical University of Lodz · PL

Funding

Medical University of Lodz 503/1-159-01/503-21-001Polish Society of Metabolic Disorders. none
6 · The paper itself

Abstract

Oxidative stress (OxS) is the cause and the consequence of metabolic syndrome (MetS), the incidence and economic burden of which is increasing each year. OxS triggers the dysregulation of signaling pathways associated with metabolism and epigenetics, including microRNAs, which are biomarkers of metabolic disorders. In this review, we aimed to summarize the current knowledge regarding the interplay between microRNAs and OxS in MetS and its components. We searched PubMed and Google Scholar to summarize the most relevant studies. Collected data suggested that different sources of OxS (e.g., hyperglycemia, insulin resistance (IR), hyperlipidemia, obesity, proinflammatory cytokines) change the expression of numerous microRNAs in organs involved in the regulation of glucose and lipid metabolism and endothelium. Dysregulated microRNAs either directly or indirectly affect the expression and/or activity of molecules of antioxidative signaling pathways (SIRT1, FOXOs, Keap1/Nrf2) along with effector enzymes (e.g., GPx-1, SOD1/2, HO-1), ROS producers (e.g., NOX4/5), as well as genes of numerous signaling pathways connected with inflammation, insulin sensitivity, and lipid metabolism, thus promoting the progression of metabolic imbalance. MicroRNAs appear to be important epigenetic modifiers in managing the delicate redox balance, mediating either pro- or antioxidant biological impacts. Summarizing, microRNAs may be promising therapeutic targets in ameliorating the repercussions of OxS in MetS.

Indexed as

Oxidative StressSignal TransductionAnimalsEpigenesis, GeneticHumansMetabolic SyndromeMicroRNAsOxidoreductasesMicroRNAsOxidoreductasesantioxidative enzymesantioxidative genesdiabetesinsulin resistancemetabolic syndromemicroRNAsobesityoxidative stressprooxidant enzymesreactive oxygen species

Identifiers

PMID32962281
PMCPMC7555602
OpenAlexW3087566221

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.