Evidence mapPaperPMID 33030764Full record

ReviewCell proliferation2020

S-adenosylmethionine: A metabolite critical to the regulation of autophagy.

Yang Ouyang, Qi Wu, Juanjuan Li, Si Sun, Shengrong Sun

Open access · goldAbstract readReview
In one paragraph

Review in Cell proliferation, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 94 papers.

0numbers the graph read from it
0cells of the map it votes in
94citing papers in PubMed
5.8field-weighted citation impact, top 3% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

94 citing papers in PubMed, 158 citations in OpenAlex.

  1. Trial
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  7. Epigenetic Modulation of the ACE System Underlies the Slow Aerobic Muscle Phenotype and Metabolic Exercise Response.FASEB journal : official publication of the Federation of American Societies for Experimental Biology · 2026
    Article
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  9. Review
  10. Observational
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  20. Review

34 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 1 institution in 1 country.

Yang OuyangDepartment of Breast and Thyroid Surgery, Renmin Hospital of Wuhan University, Wuhan, China.ORCID https://orcid.org/0000-0002-0309-0254
Qi WuDepartment of Breast and Thyroid Surgery, Renmin Hospital of Wuhan University, Wuhan, China.
Juanjuan LiDepartment of Breast and Thyroid Surgery, Renmin Hospital of Wuhan University, Wuhan, China.
Si SunDepartment of Clinical Laboratory, Renmin Hospital of Wuhan University, Wuhan, China.
Shengrong SunDepartment of Breast and Thyroid Surgery, Renmin Hospital of Wuhan University, Wuhan, China.
Wuhan University · CN

Funding

Health Commission of Hubei Province Scientific Research Project WJ2019Q053National Major Scientific Instruments and Equipment Development Projects 2012YQ160203National Natural Science Foundation of China 81302314National Natural Science Foundation of China 81471781National Natural Science Foundation of China 81903166
6 · The paper itself

Abstract

Autophagy is a mechanism that enables cells to maintain cellular homeostasis by removing damaged materials and mobilizing energy reserves in conditions of starvation. Although nutrient availability strongly impacts the process of autophagy, the specific metabolites that regulate autophagic responses have not yet been determined. Recent results indicate that S-adenosylmethionine (SAM) represents a critical inhibitor of methionine starvation-induced autophagy. SAM is primarily involved in four key metabolic pathways: transmethylation, transsulphuration, polyamine synthesis and 5'-deoxyadenosyl 5'-radical-mediated biochemical transformations. SAM is the sole methyl group donor involved in the methylation of DNA, RNA and histones, modulating the autophagic process by mediating epigenetic effects. Moreover, the metabolites of SAM, such as homocysteine, glutathione, decarboxylated SAM and spermidine, also exert important influences on the regulation of autophagy. From our perspective, nuclear-cytosolic SAM is a conserved metabolic inhibitor that connects cellular metabolic status and the regulation of autophagy. In the future, SAM might be a new target of autophagy regulators and be widely used in the treatment of various diseases.

Indexed as

AutophagyAnimalsDNA MethylationEpigenesis, GeneticHumansMetabolic Networks and PathwaysS-AdenosylmethionineS-Adenosylmethionine

Identifiers

PMID33030764
PMCPMC7653241
OpenAlexW3092271214

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.