Evidence mapPaperPMID 33077684Full record

ArticleDiabetes2021

Adaptation of Insulin Clearance to Metabolic Demand Is a Key Determinant of Glucose Tolerance.

Amalia Gastaldelli, Muhammad Abdul Ghani, Ralph A DeFronzo

Open access · bronzeAbstract read
In one paragraph

Article in Diabetes, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 47 papers.

0numbers the graph read from it
0cells of the map it votes in
47citing papers in PubMed
3.9field-weighted citation impact, top 5% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

47 citing papers in PubMed, 71 citations in OpenAlex.

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  13. Whole-Genome-Guided Functional Characterization ofFoods (Basel, Switzerland) · 2026
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 1 institution in 2 countries.

Amalia GastaldelliInstitute of Clinical Physiology, Consiglio Nazionale delle Ricerche (CNR), Pisa, Italy defronzo@uthscsa.edu amalia.gastaldelli@ifc.cnr.it.ORCID 0000-0003-2594-1651
Muhammad Abdul GhaniUniversity of Texas Health Science Center, San Antonio, TX.ORCID 0000-0003-4556-1787
Ralph A DeFronzoUniversity of Texas Health Science Center, San Antonio, TX defronzo@uthscsa.edu amalia.gastaldelli@ifc.cnr.it.ORCID 0000-0002-8581-6273
The University of Texas Health Science Center at San Antonio · US

Funding

SGLT2 Inhibitors, Ketogenesis, and KetoacidosisR01DK024092 · NIDDK · YALE UNIVERSITY · 1986 to 2025
$3.6M
Regulation of Hepatic and Peripheral Glucose MetabolismR56DK024092 · UNIVERSITY OF TEXAS HLTH SCI CTR SAN ANT · 2005 to 2005
$73k
NIDDK NIH HHS R01 DK024092NIDDK NIH HHS R56 DK024092
6 · The paper itself

Abstract

With the development of insulin resistance (IR), there is a compensatory increase in the plasma insulin response to offset the defect in insulin action to maintain normal glucose tolerance. The insulin response is the result of two factors: insulin secretion and metabolic clearance rate of insulin (MCR

Indexed as

AdultBlood GlucoseDiabetes Mellitus, Type 2FemaleGlucose Clamp TechniqueGlucose IntoleranceGlucose Tolerance TestHumansInsulinInsulin ResistanceInsulin SecretionMaleMiddle AgedObesityBlood GlucoseInsulin

Identifiers

PMID33077684
PMCPMC7881859
OpenAlexW3093313200

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.