Evidence map›Paper›PMID 33182701›Full record

SynthesisInternational journal of molecular sciences2020

Cytokine Pathways and Investigational Target Therapies in Hidradenitis Suppurativa.

Ester Del Duca, Paola Morelli, Luigi Bennardo, Cosimo Di Raimondo, Steven Paul Nisticò

Open access · goldAbstract readSystematic Review
In one paragraph

Synthesis in International journal of molecular sciences, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 43 papers, 4 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
43citing papers in PubMed, 4 pooled it
5.6field-weighted citation impact, top 3% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

43 citing papers in PubMed, 4 syntheses or guidelines pooled it, 72 citations in OpenAlex.

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  19. Identification and Validation of Hub Genes in Hidradenitis Suppurativa.Clinical, cosmetic and investigational dermatology · 2025
    Article
  20. Frontiers in immunology · 2025
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 2 institutions in 1 country.

Ester Del DucaDepartment of Health Science, University of Catanzaro Magna Graecia, 88100 Catanzaro, Italy.ORCID 0000-0001-7948-8536
Paola MorelliDepartment of Health Science, University of Catanzaro Magna Graecia, 88100 Catanzaro, Italy.ORCID 0000-0002-5847-7712
Luigi BennardoDepartment of Health Science, University of Catanzaro Magna Graecia, 88100 Catanzaro, Italy.ORCID 0000-0002-0434-1027
Cosimo Di RaimondoDepartment of Dermatology, University of Rome Tor Vergata, 00133 Rome, Italy.ORCID 0000-0003-4691-9381
Steven Paul NisticòDepartment of Health Science, University of Catanzaro Magna Graecia, 88100 Catanzaro, Italy.
Magna Graecia University · ITUniversity of Rome Tor Vergata · IT

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundHidradenitis suppurativa (HS) is a chronic inflammatory skin disease affecting areas with a high density of apocrine glands and characterized by subcutaneous nodules that may evolve into fistulas with pus secretion.

methodsThe aim of this review is to investigate all current knowledge on cytokine regulation in the pathogenesis of HS. A systematic literature research using the words "cytokine", "interleukin", "pathway", and "hidradenitis suppurativa" was performed in PubMed/Medline and Scopus/Embase databases. A search of the clinicaltrials.gov website for interventional recruiting and completed trials including the term "hidradenitis suppurativa" was also performed up to August 2020. We will discuss the pathogenetic role of various cytokines in HS and potential therapeutic targets for this debilitating disease.

resultsThe pathophysiology underlying this complex condition has not been clearly defined. An upregulation of various cytokines, such as tumor necrosis factor alpha (TNF-α), interleukin (IL)-1, IL-17, IL-23, and other molecules seems to be related to this inflammatory condition. Various cells, such as lymphocytes T Helper 1 and 17 and keratinocytes seem to be involved in the genesis of this condition.

conclusionsSeveral future studies and clinical trials are necessary in order to have new knowledge about HS and to properly treat this complex condition.

Indexed as

Antibodies, Monoclonal, HumanizedBiofilmsClinical Trials as TopicCytokinesHidradenitis SuppurativaHumansImmunologic FactorsInterleukinsMicrobiotaModels, ImmunologicalMolecular Targeted TherapyTumor Necrosis Factor-alphaAntibodies, Monoclonal, HumanizedCytokinesImmunologic FactorsInterleukinsTumor Necrosis Factor-alphacytokinehidradenitis suppurativainterleukin

Identifiers

PMID33182701
PMCPMC7696820
OpenAlexW3098610466

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.