Evidence map›Paper›PMID 33211595›Full record

ArticleJournal of applied physiology (Bethesda, Md. : 1985)2021

The effects of diet composition and chronic obesity on muscle growth and function.

Luís G O de Sousa, Andrea G Marshall, Jennifer E Norman, Jordan D Fuqua, Vitor A Lira, John C Rutledge, Sue C Bodine

Open access · greenAbstract read
In one paragraph

Article in Journal of applied physiology (Bethesda, Md. : 1985), 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 20 papers.

0numbers the graph read from it
0cells of the map it votes in
20citing papers in PubMed
1.7field-weighted citation impact, top 17% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

20 citing papers in PubMed, 29 citations in OpenAlex.

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  11. A fat- and sucrose-enriched diet causes metabolic alterations in mdx mice.American journal of physiology. Regulatory, integrative and comparative physiology · 2023
    Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 2 institutions in 1 country.

Luís G O de SousaDivision of Endocrinology and Metabolism, Department of Internal Medicine, Carver College of Medicine, University of Iowa, Iowa City, Iowa.
Andrea G MarshallDivision of Endocrinology and Metabolism, Department of Internal Medicine, Carver College of Medicine, University of Iowa, Iowa City, Iowa.
Jennifer E NormanDivision of Cardiovascular Medicine, Department of Internal Medicine, University of California, Davis, California.
Jordan D FuquaDepartment of Health and Human Physiology, Obesity Research and Education Initiative, Fraternal Order of Eagles (F.O.E.) Diabetes Research Center, Abboud Cardiovascular Research Center, Pappajohn Biomedical Institute, The University of Iowa, Iowa City, Iowa.
Vitor A LiraDepartment of Health and Human Physiology, Obesity Research and Education Initiative, Fraternal Order of Eagles (F.O.E.) Diabetes Research Center, Abboud Cardiovascular Research Center, Pappajohn Biomedical Institute, The University of Iowa, Iowa City, Iowa.
John C RutledgeDivision of Cardiovascular Medicine, Department of Internal Medicine, University of California, Davis, California.
Sue C BodineDivision of Endocrinology and Metabolism, Department of Internal Medicine, Carver College of Medicine, University of Iowa, Iowa City, Iowa.
University of Iowa · USUniversity of California, Davis · US

Funding

The Role of Intramuscular Lipids in Muscle Anabolic ResistanceR01AR070031 · NIAMS · UNIVERSITY OF IOWA · PI BODINE, SUE C, RUTLEDGE, JOHN CALVERT · 2016 to 2020
$2.3M
NIAMS NIH HHS R01 AR-070031NIAMS NIH HHS R01 AR070031
6 · The paper itself

Abstract

Diet-induced obesity (DIO) is associated with glucose intolerance, insulin resistance (IR), and an increase in intramyocellular lipids (IMCL), which may lead to disturbances in glucose and protein metabolism. To this matter, it has been speculated that chronic obesity and elevated IMCL may contribute to skeletal muscle loss and deficits in muscle function and growth capacity. Thus, we hypothesized that diets with elevated fat content would induce obesity and insulin resistance, leading to a decrease in muscle mass and an attenuated growth response to increased external loading in adult male mice. Male C57BL/6 mice (8 wk of age) were subjected to five different diets, namely, chow, low-dat-diet (LFD), high-fat-diet (HFD), sucrose, or Western diet, for 28 wk. At 25 wk, HFD and Western diets induced a 60.4% and 35.9% increase in body weight, respectively. Interestingly, HFD, but not Western or sucrose, induced glucose intolerance and insulin resistance. Measurement of isometric torque (ankle plantar flexor and ankle dorsiflexor muscles) revealed no effect of DIO on muscle function. At 28 wk of intervention, muscle area and protein synthesis were similar across all diet groups, despite insulin resistance and increased IMCL being observed in HFD and Western diet groups. In response to 30 days of functional overload, an attenuated growth response was observed in only the HFD group. Nevertheless, our results show that DIO alone is not sufficient to induce muscle atrophy and contractile dysfunction in adult male C57BL/6 mice. However, diet composition does have an impact on muscle growth in response to increased external loading.

Indexed as

Glucose IntoleranceInsulin ResistanceAnimalsDiet, High-FatMaleMiceMice, Inbred C57BLMuscle, SkeletalObesityatrophyhypertrophyinsulin resistanceintramuscular lipidsmuscle functionobesityskeletal muscle atrophy

Identifiers

PMID33211595
PMCPMC7944928
OpenAlexW3103654889

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.