Evidence mapPaperPMID 33235276Full record

ArticleScientific reports2020

Cross-sectional analysis of plasma and CSF metabolomic markers in Huntington's disease for participants of varying functional disability: a pilot study.

Andrew McGarry, John Gaughan, Cory Hackmyer, Jacqueline Lovett, Mohammed Khadeer, Hamza Shaikh, Basant Pradhan, Thomas N Ferraro, Irving W Wainer, Ruin Moaddel

Erratum issuedOpen access · goldAbstract read
In one paragraph

Article in Scientific reports, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. An erratum has been issued. Cited by 24 papers.

0numbers the graph read from it
0cells of the map it votes in
24citing papers in PubMed
2.2field-weighted citation impact, top 12% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

24 citing papers in PubMed, 38 citations in OpenAlex.

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  15. Cholesterol impacts the formation of huntingtin/lipid complexes and subsequent aggregation.Protein science : a publication of the Protein Society · 2023
    Article
  16. Review
  17. Vitamin B3 Biotech · 2023
    Article
  18. Article
  19. Potential mechanisms to modify impaired glucose metabolism in neurodegenerative disorders.Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism · 2023
    Review
  20. Review
4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

10 authors at 3 institutions in 1 country.

Andrew McGarryDepartment of Neurology, Cooper University Hospital and Cooper Medical School of Rowan University, Camden, NJ, USA. McGarry-Andrew@CooperHealth.edu.
John GaughanDepartment of Neurology, Cooper University Hospital and Cooper Medical School of Rowan University, Camden, NJ, USA.
Cory HackmyerDepartment of Neurology, Cooper University Hospital and Cooper Medical School of Rowan University, Camden, NJ, USA.
Jacqueline LovettBiomedical Research Center, National Institute On Aging, National Institutes of Health, Baltimore, MD, 21224, USA.
Mohammed KhadeerBiomedical Research Center, National Institute On Aging, National Institutes of Health, Baltimore, MD, 21224, USA.
Hamza ShaikhDepartment of Neurology, Cooper University Hospital and Cooper Medical School of Rowan University, Camden, NJ, USA.
Basant PradhanDepartment of Neurology, Cooper University Hospital and Cooper Medical School of Rowan University, Camden, NJ, USA.
Thomas N FerraroDepartment of Biomedical Sciences, Cooper Medical School of Rowan University, Camden, NJ, USA.
Irving W WainerDepartment of Neurology, Cooper University Hospital and Cooper Medical School of Rowan University, Camden, NJ, USA.
Ruin MoaddelBiomedical Research Center, National Institute On Aging, National Institutes of Health, Baltimore, MD, 21224, USA. moaddelru@grc.nia.nih.gov.
Cooper University Hospital · USNational Institutes of Health · USCooper Medical School of Rowan University · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Huntington's Disease (HD) is a progressive, fatal neurodegenerative condition. While generally considered for its devastating neurological phenotype, disturbances in other organ systems and metabolic pathways outside the brain have attracted attention for possible relevance to HD pathology, potential as therapeutic targets, or use as biomarkers of progression. In addition, it is not established how metabolic changes in the HD brain correlate to progression across the full spectrum of early to late-stage disease. In this pilot study, we sought to explore the metabolic profile across manifest HD from early to advanced clinical staging through metabolomic analysis by mass spectrometry in plasma and cerebrospinal fluid (CSF). With disease progression, we observed nominally significant increases in plasma arginine, citrulline, and glycine, with decreases in total and D-serine, cholesterol esters, diacylglycerides, triacylglycerides, phosphatidylcholines, phosphatidylethanolamines, and sphingomyelins. In CSF, worsening disease was associated with nominally significant increases in NAD

Indexed as

Disability EvaluationMetabolomicsAdultArginineBiomarkersCreatineCross-Sectional StudiesFemaleGlycineHumansHuntington DiseaseMaleMiddle AgedPilot ProjectsTreatment OutcomeYoung AdultArginineBiomarkersCreatineGlycine

Identifiers

PMID33235276
PMCPMC7686309
OpenAlexW3108234709

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.