Evidence mapPaperPMID 33282111Full record

ReviewOxidative medicine and cellular longevity2020

Effects of REDOX in Regulating and Treatment of Metabolic and Inflammatory Cardiovascular Diseases.

Kai Wang, Yanhan Dong, Jing Liu, Lili Qian, Tao Wang, Xiangqian Gao, Kun Wang, Luyu Zhou

Open access · hybridAbstract readReview
In one paragraph

Review in Oxidative medicine and cellular longevity, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 18 papers.

0numbers the graph read from it
0cells of the map it votes in
18citing papers in PubMed
1.1field-weighted citation impact, top 22% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

18 citing papers in PubMed, 27 citations in OpenAlex.

  1. Review
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  7. New Types of Post-Translational Modification of Proteins in Cardiovascular Diseases.Journal of cardiovascular translational research · 2025
    Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 1 institution in 1 country.

Kai WangInstitute of translational medicine, The Affiliated Hospital of Qingdao University, Qingdao University, Qingdao 266021, China.
Yanhan DongInstitute of translational medicine, The Affiliated Hospital of Qingdao University, Qingdao University, Qingdao 266021, China.ORCID https://orcid.org/0000-0001-9940-1303
Jing LiuInstitute of translational medicine, The Affiliated Hospital of Qingdao University, Qingdao University, Qingdao 266021, China.
Lili QianInstitute of translational medicine, The Affiliated Hospital of Qingdao University, Qingdao University, Qingdao 266021, China.
Tao WangInstitute of translational medicine, The Affiliated Hospital of Qingdao University, Qingdao University, Qingdao 266021, China.
Xiangqian GaoInstitute of translational medicine, The Affiliated Hospital of Qingdao University, Qingdao University, Qingdao 266021, China.
Kun WangInstitute of translational medicine, The Affiliated Hospital of Qingdao University, Qingdao University, Qingdao 266021, China.
Luyu ZhouInstitute of translational medicine, The Affiliated Hospital of Qingdao University, Qingdao University, Qingdao 266021, China.ORCID https://orcid.org/0000-0002-8356-122X
Qingdao University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Reduction oxidation (REDOX) reaction is crucial in life activities, and its dynamic balance is regulated by ROS. Reactive oxygen species (ROS) is associated with a variety of metabolic diseases involving in multiple cellular signalling in pathologic and physiological signal transduction. ROS are the by-products of numerous enzymatic reactions in various cell compartments, including the cytoplasm, cell membrane, endoplasmic reticulum (ER), mitochondria, and peroxisome. ROS signalling is not only involved in normal physiological processes but also causes metabolic dysfunction and maladaptive responses to inflammatory signals, which depends on the cell type or tissue environment. Excess oxidants are able to alter the normal structure and function of DNA, lipids, and proteins, leading to mutations or oxidative damage. Therefore, excessive oxidative stress is usually regarded as the cause of various pathological conditions, such as cancer, neurodegeneration, cardiovascular diseases (CVDs), diabetes, and kidney diseases. Currently, it has been possible to detect diabetes and other cardiac diseases by detecting derivatives accompanied by oxidative stress in vivo as biomarkers, but there is no effective method to treat these diseases. In consequence, it is essential for us to seek new therapy targeting these diseases through understanding the role of ROS signalling in regulating metabolic activity, inflammatory activation, and cardiac diseases related to metabolic dysfunction. In this review, we summarize the current literature on REDOX and its role in the regulation of cardiac metabolism and inflammation, focusing on ROS, local REDOX signalling pathways, and other mechanisms.

Indexed as

Oxidation-ReductionAnimalsCardiovascular DiseasesHumansInflammationMitochondriaOxidative StressReactive Oxygen SpeciesReactive Oxygen Species

Identifiers

PMID33282111
PMCPMC7685846
OpenAlexW3106405780

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.