Evidence mapPaperPMID 33430853Full record

ArticleBMC medical genomics2021

Novel susceptibility loci identified in a genome-wide association study of type 2 diabetes complications in population of Latvia.

Monta Ustinova, Raitis Peculis, Raimonds Rescenko, Vita Rovite, Linda Zaharenko, Ilze Elbere, Laila Silamikele, Ilze Konrade, Jelizaveta Sokolovska, Valdis Pirags and 1 more

Abstract read
In one paragraph

Article in BMC medical genomics, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 15 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
15citing papers in PubMed, 1 pooled it
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

15 citing papers in PubMed, 1 synthesis or guideline pooled it.

  1. Pooled it
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  14. PDE4DIP in health and diseases.Cellular signalling · 2022
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors.

Monta UstinovaLatvian Biomedical Research and Study Centre, Ratsupites iela 1, Riga, 1067, Latvia.
Raitis PeculisLatvian Biomedical Research and Study Centre, Ratsupites iela 1, Riga, 1067, Latvia.
Raimonds RescenkoLatvian Biomedical Research and Study Centre, Ratsupites iela 1, Riga, 1067, Latvia.
Vita RoviteLatvian Biomedical Research and Study Centre, Ratsupites iela 1, Riga, 1067, Latvia.
Linda ZaharenkoLatvian Biomedical Research and Study Centre, Ratsupites iela 1, Riga, 1067, Latvia.
Ilze ElbereLatvian Biomedical Research and Study Centre, Ratsupites iela 1, Riga, 1067, Latvia.
Laila SilamikeleLatvian Biomedical Research and Study Centre, Ratsupites iela 1, Riga, 1067, Latvia.
Ilze KonradeLatvian Biomedical Research and Study Centre, Ratsupites iela 1, Riga, 1067, Latvia.
Jelizaveta SokolovskaFaculty of Medicine, University of Latvia, Jelgavas iela 3, Riga, 1004, Latvia.
Valdis PiragsLatvian Biomedical Research and Study Centre, Ratsupites iela 1, Riga, 1067, Latvia.
Janis KlovinsLatvian Biomedical Research and Study Centre, Ratsupites iela 1, Riga, 1067, Latvia. klovins@biomed.lu.lv.

Funding

European Regional Development Fund 1.1.1.2/VIAA/2/18/287
6 · The paper itself

Abstract

backgroundType 2 diabetes complications cause a serious emotional and economical burden to patients and healthcare systems globally. Management of both acute and chronic complications of diabetes, which dramatically impair the quality of patients' life, is still an unsolved issue in diabetes care, suggesting a need for early identification of individuals with high risk for developing diabetes complications.

methodsWe performed a genome-wide association study in 601 type 2 diabetes patients after stratifying them according to the presence or absence of four types of diabetes complications: diabetic neuropathy, diabetic nephropathy, macrovascular complications, and ophthalmic complications.

resultsThe analysis revealed ten novel associations showing genome-wide significance, including rs1132787 (GYPA, OR = 2.71; 95% CI = 2.02-3.64) and diabetic neuropathy, rs2477088 (PDE4DIP, OR = 2.50; 95% CI = 1.87-3.34), rs4852954 (NAT8, OR = 2.27; 95% CI = 2.71-3.01), rs6032 (F5, OR = 2.12; 95% CI = 1.63-2.77), rs6935464 (RPS6KA2, OR = 2.25; 95% CI = 6.69-3.01) and macrovascular complications, rs3095447 (CCDC146, OR = 2.18; 95% CI = 1.66-2.87) and ophthalmic complications. By applying the targeted approach of previously reported susceptibility loci we managed to replicate three associations: MAPK14 (rs3761980, rs80028505) and diabetic neuropathy, APOL1 (rs136161) and diabetic nephropathy.

conclusionsTogether these results provide further evidence for the implication of genetic factors in the development of type 2 diabetes complications and highlight several potential key loci, able to modify the risk of developing these conditions. Moreover, the candidate variant approach proves a strong and consistent effect for multiple variants across different populations.

Indexed as

Diabetes Mellitus, Type 2Genetic LociGenetic Predisposition to DiseaseGenome-Wide Association StudyPolymorphism, Single NucleotideAgedDiabetes ComplicationsFemaleHumansMaleMiddle AgedDiabetic complicationsGenome-wide genotypingType 2 diabetes mellitus

Identifiers

PMID33430853
PMCPMC7802349

What Socratic holds

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LicenceCC BY
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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.