Evidence mapPaperPMID 33455994Full record

ArticleJournal of atherosclerosis and thrombosis2022

Nanoparticle-Mediated Delivery of Pitavastatin to Monocytes/Macrophages Inhibits Angiotensin II-Induced Abdominal Aortic Aneurysm Formation in Apoe

Shunsuke Katsuki, Jun-Ichiro Koga, Tetsuya Matoba, Ryuta Umezu, Soichi Nakashiro, Kaku Nakano, Hiroyuki Tsutsui, Kensuke Egashira

Open access · diamondAbstract read
In one paragraph

Article in Journal of atherosclerosis and thrombosis, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 18 papers.

0numbers the graph read from it
0cells of the map it votes in
18citing papers in PubMed
2.2field-weighted citation impact, top 12% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

18 citing papers in PubMed, 23 citations in OpenAlex.

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  12. Multiscale physics-basedFrontiers in drug delivery · 2024
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  16. Single-cell transcriptomeFrontiers in cardiovascular medicine · 2022
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 1 institution in 1 country.

Shunsuke KatsukiThe Department of Cardiovascular Medicine, Graduate School of Medical Sciences, Kyushu University.
Jun-Ichiro KogaThe Department of Cardiovascular Medicine, Graduate School of Medical Sciences, Kyushu University.
Tetsuya MatobaThe Department of Cardiovascular Medicine, Graduate School of Medical Sciences, Kyushu University.
Ryuta UmezuThe Department of Cardiovascular Medicine, Graduate School of Medical Sciences, Kyushu University.
Soichi NakashiroThe Department of Cardiovascular Medicine, Graduate School of Medical Sciences, Kyushu University.
Kaku NakanoThe Department of Cardiovascular Research, Development, and Translational Medicine, Center for Disruptive Cardiovascular Innovation, Kyushu University.
Hiroyuki TsutsuiThe Department of Cardiovascular Medicine, Graduate School of Medical Sciences, Kyushu University.
Kensuke EgashiraThe Department of Cardiovascular Research, Development, and Translational Medicine, Center for Disruptive Cardiovascular Innovation, Kyushu University.
Kyushu University · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

aimAbdominal aortic aneurysm (AAA) is a lethal and multifactorial disease. To prevent a rupture and dissection of enlarged AAA, prophylactic surgery and stenting are currently available. There are, however, no medical therapies preventing these complications of AAA. Statin is one of the candidates, but its efficacy on AAA formation/progression remains controversial. We have previously demonstrated that nanoparticles (NPs) incorporating pitavastatin (Pitava-NPs)-clinical trials using these nanoparticles have been already conducted-suppressed progression of atherosclerosis in apolipoprotein E-deficient ( Apoe

methodsAngiotensin II was intraperitoneally injected by osmotic mini-pumps to induce AAA formation in Apoe

resultsIntravenously administered Pitava-NPs (containing 0.012 mg/kg/week pitavastatin) inhibited AAA formation accompanied with reduction of macrophage accumulation and monocyte chemoattractant protein-1 (MCP-1) expression. Ex vivo molecular imaging revealed that Pitava-NPs not only reduced macrophage accumulation but also attenuated matrix metalloproteinase activity in the abdominal aorta, which was underpinned by attenuated elastin degradation.

conclusionThese results suggest that Pitava-NPs inhibit AAA formation associated with reduced macrophage accumulation and MCP-1 expression. This clinically feasible nanomedicine could be an innovative therapeutic strategy that prevents devastating complications of AAA.

Indexed as

NanoparticlesAngiotensin IIAnimalsAortic Aneurysm, AbdominalApolipoproteins EChemokine CCL2Disease Models, AnimalHydroxymethylglutaryl-CoA Reductase InhibitorsMacrophagesMaleMatrix MetalloproteinasesMiceMice, Inbred C57BLMonocytesPolylactic Acid-Polyglycolic Acid CopolymerQuinolinesAngiotensin IIApolipoproteins EChemokine CCL2Hydroxymethylglutaryl-CoA Reductase InhibitorsMatrix MetalloproteinasespitavastatinPolylactic Acid-Polyglycolic Acid CopolymerQuinolinesAneurysmInflammationMonocyteNanoparticleStatins

Identifiers

PMID33455994
PMCPMC8737070
OpenAlexW3125836040

What Socratic holds

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LicenceCC BY-NC-SA
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.