Evidence map›Paper›PMID 33501889›Full record

ArticleAmerican journal of physiology. Gastrointestinal and liver physiology2021

A dietary ketone ester mitigates histological outcomes of NAFLD and markers of fibrosis in high-fat diet fed mice.

Mary P Moore, Rory P Cunningham, Rachel A H Davis, Sarah E Deemer, Brandon M Roberts, Eric P Plaisance, R Scott Rector

Open access · greenAbstract readComparative Study
In one paragraph

Article in American journal of physiology. Gastrointestinal and liver physiology, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 20 papers.

0numbers the graph read from it
0cells of the map it votes in
20citing papers in PubMed
2.3field-weighted citation impact, top 12% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

20 citing papers in PubMed, 23 citations in OpenAlex.

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  13. Targeting ketone body metabolism to treat fatty liver disease.Journal of pharmacy & pharmaceutical sciences : a publication of the Canadian Society for Pharmaceutical Sciences, Societe canadienne des sciences pharmaceutiques · 2024
    Review
  14. Metabolic Messengers: ketone bodies.Nature metabolism · 2023
    Review
  15. Review
  16. Article
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  18. Article
  19. Bioengineered · 2022
    Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 2 institutions in 1 country.

Mary P MooreResearch Service, Harry S. Truman Memorial Veterans Medical Center, Columbia, Missouri.
Rory P CunninghamResearch Service, Harry S. Truman Memorial Veterans Medical Center, Columbia, Missouri.
Rachel A H DavisDepartment of Nutrition Sciences, University of Alabama at Birmingham, Birmingham, Alabama.
Sarah E DeemerDepartment of Nutrition Sciences, University of Alabama at Birmingham, Birmingham, Alabama.
Brandon M RobertsDepartment of Nutrition Sciences, University of Alabama at Birmingham, Birmingham, Alabama.
Eric P PlaisanceDepartment of Nutrition Sciences, University of Alabama at Birmingham, Birmingham, Alabama.ORCID 0000-0001-9621-6583
R Scott RectorResearch Service, Harry S. Truman Memorial Veterans Medical Center, Columbia, Missouri.
University of Alabama at Birmingham · USHarry S. Truman Memorial Veterans' Hospital · US

Funding

Why is the prevalence of obesity so high in U.S. Southern States? Regional predictors of BMI and obesity treatment response.P30DK056336 · NIDDK · UNIVERSITY OF ALABAMA AT BIRMINGHAM · PI James O Hill · 2000 to 2026
$31.9M
UAB Pre-Doctoral Training Program in Obesity-Related ResearchT32HL105349 · NHLBI · UNIVERSITY OF ALABAMA AT BIRMINGHAM · PI BARBARA A GOWER · 2010 to 2026
$5.7M
UAB Obesity Training ProgramT32DK062710 · NIDDK · UNIVERSITY OF ALABAMA AT BIRMINGHAM · PI James O Hill, Brooks C Wingo · 2004 to 2026
$5.5M
Hepatic eNOS in the regulation of NASHI01BX003271 · VA · HARRY S. TRUMAN MEMORIAL VA HOSPITAL · PI RANDY SCOTT RECTOR · 2017 to 2026
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BLRD VA I01 BX003271HHS | NIH | National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK) P30DK056336HHS | NIH | National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK) T32DK062710National Heart and Lung Institute (NHLI) T32HL105349NHLBI NIH HHS T32 HL105349NIDDK NIH HHS P30 DK056336NIDDK NIH HHS T32 DK062710U.S. Department of Veterans Affairs (VA) I01BX003271
6 · The paper itself

Abstract

Nutritional ketosis as a therapeutic tool has been extended to the treatment of metabolic diseases, including obesity, type 2 diabetes, and nonalcoholic fatty liver disease (NAFLD). The purpose of this study was to determine whether dietary administration of the ketone ester (KE) R,S-1,3-butanediol diacetoacetate (BD-AcAc

Indexed as

Diet, High-FatAcetoacetatesAnimalsBiomarkersButylene GlycolsCaloric RestrictionGene Expression RegulationHepatic Stellate CellsInflammation MediatorsLiverLiver Cirrhosis, ExperimentalMacrophage ActivationMaleMiceMice, Inbred C57BLNon-alcoholic Fatty Liver Disease1,3-butanediol diacetoacetateAcetoacetatesBiomarkersButylene GlycolsInflammation Mediatorsfibrosisketone esterketosisNAFLDNASH

Identifiers

PMID33501889
PMCPMC8238172
OpenAlexW3124129828

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.