Evidence map›Paper›PMID 33534053›Full record

ReviewParasitology research2021

The host mTOR pathway and parasitic diseases pathogenesis.

Sajad Rashidi, Reza Mansouri, Mohammad Ali-Hassanzadeh, Zahra Mojtahedi, Reza Shafiei, Amir Savardashtaki, Nasrin Hamidizadeh, Mohammadreza Karimazar, Paul Nguewa, Raúl Manzano-Román

Open access · bronzeAbstract readReview
In one paragraph

Review in Parasitology research, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 16 papers.

0numbers the graph read from it
0cells of the map it votes in
16citing papers in PubMed
3.6field-weighted citation impact, top 6% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

16 citing papers in PubMed, 28 citations in OpenAlex.

  1. Article
  2. Review
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  8. Cognitive Impairment in Multiple Sclerosis.Bioengineering (Basel, Switzerland) · 2023
    Review
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  10. Article
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  12. Article
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  16. The role of autophagy in hepatic fibrosis.American journal of translational research · 2021
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors at 7 institutions in 3 countries.

Sajad RashidiDepartment of Parasitology and Mycology, School of Medicine, Shiraz University of Medical Sciences, Shiraz, Iran.
Reza MansouriDepartment of Immunology, Faculty of Medicine, Shahid Sadoughi University of Medical Sciences and Health Services, Yazd, Iran.
Mohammad Ali-HassanzadehDepartment of Immunology, School of Medicine, Jiroft University of Medical Sciences, Jiroft, Iran.
Zahra MojtahediDepartment of Health Care Administration and Policy, School of Public Health, University of Nevada, Las Vegas, Las Vegas, NV, USA.
Reza ShafieiVector-borne Diseases Research Center, North Khorasan University of Medical Sciences, Bojnurd, Iran.
Amir SavardashtakiDepartment of Medical Biotechnology, School of Advanced Medical Sciences and Technologies, Shiraz University of Medical Sciences, Shiraz, Iran.
Nasrin HamidizadehMolecular Dermatology Research Center, Shiraz University of Medical Sciences, Shiraz, Iran.
Mohammadreza KarimazarDepartment of Parasitology and Mycology, School of Medicine, Shiraz University of Medical Sciences, Shiraz, Iran.
Paul NguewaISTUN Instituto de Salud Tropical, Department of Microbiology and Parasitology, IdiSNA (Navarra Institute for Health Research), University of Navarra, c/ Irunlarrea 1, 31008, Pamplona, Spain. panguewa@unav.es.
Raúl Manzano-RománProteomics Unit, Cancer Research Centre (IBMCC/CSIC/USAL/IBSAL), 37007, Salamanca, Spain. rmanzano@usal.es.
Shiraz University of Medical Sciences · IRInstituto de Investigación Biomédica de Salamanca · ESNorth Khorasan University of Medical Sciences · IRShahid Sadoughi University of Medical Sciences and Health Services · IRUniversidad de Navarra · ESUniversity of Jiroft · IRUniversity of Nevada, Las Vegas · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The mechanistic (or mammalian) target of rapamycin (mTOR) is considered as a critical regulatory enzyme involved in essential signaling pathways affecting cell growth, cell proliferation, protein translation, regulation of cellular metabolism, and cytoskeletal structure. Also, mTOR signaling has crucial roles in cell homeostasis via processes such as autophagy. Autophagy prevents many pathogen infections and is involved on immunosurveillance and pathogenesis. Immune responses and autophagy are therefore key host responses and both are linked by complex mTOR regulatory mechanisms. In recent years, the mTOR pathway has been highlighted in different diseases such as diabetes, cancer, and infectious and parasitic diseases including leishmaniasis, toxoplasmosis, and malaria. The current review underlines the implications of mTOR signals and intricate networks on pathogen infections and the modulation of this master regulator by parasites. Parasitic infections are able to induce dynamic metabolic reprogramming leading to mTOR alterations in spite of many other ways impacting this regulatory network. Accordingly, the identification of parasite effects and interactions over such a complex modulation might reveal novel information regarding the biology of the abovementioned parasites and might allow the development of therapeutic strategies against parasitic diseases. In this sense, the effects of inhibiting the mTOR pathways are also considered in this context in the light of their potential for the prevention and treatment of parasitic diseases.

Indexed as

AnimalsAutophagyCell CycleCell ProliferationHumansImmunityLeishmaniasisMalariaParasitesParasitic DiseasesPhosphorylationProtein BiosynthesisSignal TransductionSirolimusTOR Serine-Threonine KinasesToxoplasmosisSirolimusTOR Serine-Threonine KinasesModulation,mTORParasitesRapamycinSignalingTherapies

Identifiers

PMID33534053
PMCPMC7856335
OpenAlexW3126565314

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.