Evidence mapPaperPMID 33569010Full record

ArticleFrontiers in pharmacology2020

Inhibition of Lipid Accumulation in Skeletal Muscle and Liver Cells: A Protective Mechanism of Bilirubin Against Diabetes Mellitus Type 2.

Claudia A Hana, Eva-Maria Klebermass, Theresa Balber, Markus Mitterhauser, Ruth Quint, Yvonne Hirtl, Antonia Klimpke, Sophie Somloi, Juliana Hutz, Elisabeth Sperr and 6 more

Open access · goldAbstract read
In one paragraph

Article in Frontiers in pharmacology, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.

0numbers the graph read from it
0cells of the map it votes in
6citing papers in PubMed
1.5field-weighted citation impact, top 18% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

6 citing papers in PubMed, 12 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

16 authors at 4 institutions in 2 countries.

Claudia A HanaDepartment of Nutritional Sciences, Faculty of Life Sciences, University of Vienna, Vienna, Austria.
Eva-Maria KlebermassDepartment of Biomedical Imaging and Image-guided Therapy, Division of Nuclear Medicine, Medical University of Vienna, Vienna, Austria.
Theresa BalberDepartment of Biomedical Imaging and Image-guided Therapy, Division of Nuclear Medicine, Medical University of Vienna, Vienna, Austria.
Markus MitterhauserDepartment of Biomedical Imaging and Image-guided Therapy, Division of Nuclear Medicine, Medical University of Vienna, Vienna, Austria.
Ruth QuintDepartment of Nutritional Sciences, Faculty of Life Sciences, University of Vienna, Vienna, Austria.
Yvonne HirtlDepartment of Nutritional Sciences, Faculty of Life Sciences, University of Vienna, Vienna, Austria.
Antonia KlimpkeDepartment of Nutritional Sciences, Faculty of Life Sciences, University of Vienna, Vienna, Austria.
Sophie SomloiDepartment of Nutritional Sciences, Faculty of Life Sciences, University of Vienna, Vienna, Austria.
Juliana HutzDepartment of Nutritional Sciences, Faculty of Life Sciences, University of Vienna, Vienna, Austria.
Elisabeth SperrDepartment of Nutritional Sciences, Faculty of Life Sciences, University of Vienna, Vienna, Austria.
Paulina EderDepartment of Nutritional Sciences, Faculty of Life Sciences, University of Vienna, Vienna, Austria.
Jana JašprováInstitute of Medical Biochemistry and Laboratory Diagnostics, University General Hospital and 1 Faculty of Medicine, Charles University, Prague, Czechia.
Petra ValáškováInstitute of Medical Biochemistry and Laboratory Diagnostics, University General Hospital and 1 Faculty of Medicine, Charles University, Prague, Czechia.
Libor VítekInstitute of Medical Biochemistry and Laboratory Diagnostics, University General Hospital and 1 Faculty of Medicine, Charles University, Prague, Czechia.
Elke HeissDepartment of Pharmacognosy, University of Vienna, Vienna, Austria.
Karl-Heinz WagnerDepartment of Nutritional Sciences, Faculty of Life Sciences, University of Vienna, Vienna, Austria.
University of Vienna · ATCharles University · CZLudwig Boltzmann Institute Applied Diagnostics · ATMedical University of Vienna · AT

Funding

Austrian Science Fund FWF P 29608
6 · The paper itself

Abstract

Ectopic lipid accumulation in skeletal muscle and liver drives the pathogenesis of diabetes mellitus type 2 (DMT2). Mild hyperbilirubinaemia has been repeatedly suggested to play a role in the prevention of DMT2 and is known for its capacity to shape an improved lipid phenotype in humans and in animals. To date, the effect of bilirubin on lipid accumulation in tissues that are prone to ectopic lipid deposition is unclear. Therefore, we analyzed the effect of bilirubin on lipid accumulation in skeletal muscle and liver cell lines. C2C12 skeletal mouse muscle and HepG2 human liver cells were treated with physiological concentrations of free fatty acids (FFA) (0.5 mM and 1 mM) and unconjugated bilirubin (UCB) (17.1 and 55 µM). The intracellular presence of UCB upon exogenous UCB administration was confirmed by HPLC and the lipid accumulation was assessed by using Nile red. Exposure of both cell lines to UCB significantly reduced lipid accumulation by up to 23% (

Indexed as

[18F]FDG uptakebilirubinC2C12 skeletal muscle cellsectopic lipid accumulationHepG2 cellsinsulin resistancelipid accumulationmild hyperbilirubinaemia

Identifiers

PMID33569010
PMCPMC7868327
OpenAlexW3121336663

What Socratic holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.