Evidence mapPaperPMID 33610053Full record

ArticleEnvironment international2021

Exposure to perfluoroalkyl substances and blood pressure in pregnancy among 1436 women from the Odense Child Cohort.

Anna Birukov, Louise Bjørkholt Andersen, Marianne Skovsager Andersen, Julie H Nielsen, Flemming Nielsen, Henriette Boye Kyhl, Jan Stener Jørgensen, Philippe Grandjean, Ralf Dechend, Tina Kold Jensen

Open access · goldAbstract read
In one paragraph

Article in Environment international, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 24 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
24citing papers in PubMed, 1 pooled it
4.2field-weighted citation impact, top 5% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

24 citing papers in PubMed, 1 synthesis or guideline pooled it, 50 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors at 4 institutions in 3 countries.

Anna BirukovDepartment of Molecular Epidemiology, German Institute of Human Nutrition Potsdam-Rehbrücke, Nuthetal, Germany; German Center for Diabetes Research München-Neuherberg, Germany; DZHK (German Centre for Cardiovascular Research), Partner Site Berlin, Berlin, Germany.
Louise Bjørkholt AndersenInstitute of Clinical Research, Faculty of Health Sciences, University of Southern Denmark, Odense, Denmark; Department of Obstetrics and Gynecology, Odense University Hospital, Odense, Denmark.
Marianne Skovsager AndersenInstitute of Clinical Research, Faculty of Health Sciences, University of Southern Denmark, Odense, Denmark; Department of Endocrinology and Metabolism, Odense University Hospital, Odense, Denmark.
Julie H NielsenDepartment of Obstetrics and Gynecology, Odense University Hospital, Odense, Denmark; Department of Endocrinology and Metabolism, Odense University Hospital, Odense, Denmark.
Flemming NielsenDepartment of Clinical Pharmacology, Pharmacy and Environmental Medicine, Institute of Public Health, University of Southern Denmark, Odense, Denmark.
Henriette Boye KyhlOdense Child Cohort, Hans Christian Andersen Children's Hospital, Odense University Hospital, Odense, Denmark; OPEN Patient Data Explorative Network, Odense University Hospital, Odense, Denmark.
Jan Stener JørgensenInstitute of Clinical Research, Faculty of Health Sciences, University of Southern Denmark, Odense, Denmark; Department of Obstetrics and Gynecology, Odense University Hospital, Odense, Denmark.
Philippe GrandjeanDepartment of Clinical Pharmacology, Pharmacy and Environmental Medicine, Institute of Public Health, University of Southern Denmark, Odense, Denmark; Harvard T.H.Chan School of Public Health, Boston, USA.
Ralf DechendDZHK (German Centre for Cardiovascular Research), Partner Site Berlin, Berlin, Germany; Department of Obstetrics and Gynecology, Odense University Hospital, Odense, Denmark; HELIOS-Klinikum, Berlin, Department of Cardiology and Nephrology, Germany; Experimental and Clinical Research Center, Joint Cooperation Between Max-Delbrück Center for Molecular Medicine and Charité - Universitätsmedizin Berlin, Germany.
Tina Kold JensenDepartment of Clinical Pharmacology, Pharmacy and Environmental Medicine, Institute of Public Health, University of Southern Denmark, Odense, Denmark; Odense Child Cohort, Hans Christian Andersen Children's Hospital, Odense University Hospital, Odense, Denmark; OPEN Patient Data Explorative Network, Odense University Hospital, Odense, Denmark. Electronic address: tkjensen@health.sdu.dk.
Odense University Hospital · DKUniversity of Southern Denmark · DKGerman Centre for Cardiovascular Research · DEMax Delbrück Center · DE

Funding

Sources, Transport, Exposure & Effects of PFAS (STEEP) Center - RENEWALP42ES027706 · UNIVERSITY OF RHODE ISLAND · 2025 to 2025
$1.5M
NIEHS NIH HHS P42 ES027706
6 · The paper itself

Abstract

backgroundPrevious studies of association between exposure to poly- and perfluoroalkyl substances (PFAS) and gestational hypertension (GH) and preeclampsia (PE) have shown conflicting results, but most dichotomized outcome and did not study continuous blood pressure (BP) changes.

objectivesTo study the association between PFAS exposure in early pregnancy and maternal BP trajectories in pregnancy, gestational hypertension and preeclampsia.

methods1436 women were enrolled in the Odense Child Cohort in early pregnancy and had a serum sample drawn, from which perfluorohexane sulfonic acid (PFHxS), perfluorooctane sulfonic acid (PFOS), perfluorooctanoic acid (PFOA), perfluorononanoic acid (PFNA) and perfluorodecanoic acid (PFDA) were measured using LC-MS/MS. Repeated BP measurements through pregnancy and information on PE were obtained from hospital files. Adjusted linear mixed models were used to investigate association between PFAS exposure and BP trajectory. Associations between PFAS and PE and GH were assessed by Cox proportional hazards model.

resultsAll women had measurable concentrations of PFAS. In all of many comparisons higher PFAS exposure (apart from PFHxS) was associated with higher systolic (SBP) and diastolic (DBP) blood pressures, although not all were significant, which is unlikely to be due to chance. After adjustment, each doubling in PFOS or PFOA exposure was associated with 0.47 mmHg (95% CI: -0.13; 1.08) and 0.36 mmHg (-0.19; 0.92) higher SBP; and 0.58 mmHg (0.13; 1.04) and 0.37 mmHg (-0.05; 0.79) higher DBP. No clear associations between PFAS exposure and PE or GH were found. DISCUSSION: The magnitude of the association between PFAS exposure and BP might appear small, statistically non-significant and the possible clinical importance low. However, at a population level this may slightly shift the distribution of BP towards an increased incidence of GH. If BP increases in pregnancy, it may have long-term impact on health not only of the pregnant woman but also of her offspring.

Indexed as

Alkanesulfonic AcidsEnvironmental PollutantsFluorocarbonsBlood PressureChromatography, LiquidFemaleHumansPregnancyTandem Mass SpectrometryAlkanesulfonic AcidsEnvironmental PollutantsFluorocarbonsBlood pressureEnvironmental chemicalsPerfluoroalkyl substances (PFAS)PreeclampsiaPregnancy

Identifiers

PMID33610053
PMCPMC11149831
OpenAlexW3132655570

What Socratic holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.