Evidence map›Paper›PMID 33711140›Full record

ArticleThe British journal of surgery2021

Acute kidney injury following endovascular intervention for peripheral artery disease.

E Katsogridakis, T Lea, T Yap, A Batchelder, P Saha, A Diamantopoulos, N Saratzis, R Davies, H Zayed, M J Bown and 1 more

Open access · bronzeAbstract read
In one paragraph

Article in The British journal of surgery, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 10 papers.

0numbers the graph read from it
0cells of the map it votes in
10citing papers in PubMed
2.3field-weighted citation impact, top 12% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

10 citing papers in PubMed, 17 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors at 3 institutions in 2 countries.

E KatsogridakisDepartment of Cardiovascular Sciences and National Institute for Health Research Leicester Biomedical Research Centre, University of Leicester, Leicester, UK.
T LeaDepartment of Vascular Surgery, Guy's and St Thomas' Hospital NHS Foundation Trust, London, UK.
T YapDepartment of Vascular Surgery, Guy's and St Thomas' Hospital NHS Foundation Trust, London, UK.
A BatchelderDepartment of Cardiovascular Sciences and National Institute for Health Research Leicester Biomedical Research Centre, University of Leicester, Leicester, UK.
P SahaDepartment of Vascular Surgery, Guy's and St Thomas' Hospital NHS Foundation Trust, London, UK.
A DiamantopoulosDepartment of Vascular Surgery, Guy's and St Thomas' Hospital NHS Foundation Trust, London, UK.
N SaratzisDepartment of Vascular Surgery, Aristotle University Medical School, Papageorgiou General Hospital, Thessaloniki, Greece.
R DaviesDepartment of Cardiovascular Sciences and National Institute for Health Research Leicester Biomedical Research Centre, University of Leicester, Leicester, UK.
H ZayedDepartment of Vascular Surgery, Guy's and St Thomas' Hospital NHS Foundation Trust, London, UK.
M J BownDepartment of Cardiovascular Sciences and National Institute for Health Research Leicester Biomedical Research Centre, University of Leicester, Leicester, UK.
A SaratzisDepartment of Cardiovascular Sciences and National Institute for Health Research Leicester Biomedical Research Centre, University of Leicester, Leicester, UK.
St Thomas' Hospital · GBUniversity of Leicester · GBPapageorgiou General Hospital · GR

Funding

British Heart Foundation
6 · The paper itself

Abstract

backgroundThe incidence of, and risk factors for, acute kidney injury (AKI) after endovascular intervention for peripheral artery disease (PAD) remain unknown. The aim of this study was to assess the proportion of patients who develop AKI and explore the risk factors.

methodsProspectively collected data on patients undergoing femoropopliteal endovascular intervention for symptomatic PAD across three vascular centres were analysed. The proportion of patients developing AKI (according to the Kidney Disease Improving Global Outcomes definition) within 48 h, and the proportion developing the composite Major Adverse Kidney Events (MAKE) endpoints (death, dialysis, drop in estimated glomerular filtration rate at least 25 per cent) at 30 days (MAKE30) and remains 90 days (MAKE90) were calculated. Multivariable regression analysis was used to assess predictors of AKI, and the association between AKI and death.

resultsSome 2041 patients were included in the analysis. AKI developed in 239 patients (11.7 per cent), with 47 (2.3 per cent) requiring dialysis within 30 days, and 18 (0.9 per cent) requiring ongoing dialysis. The MAKE30 and MAKE90 composite endpoints were reached in 358 (17.5 per cent) and 449 (22.0 per cent) patients respectively. Risk factors for AKI were age, sex, congestive heart failure, chronic limb-threatening ischaemia, emergency procedure, and pre-existing chronic kidney disease. AKI, dementia, congestive heart failure, and major amputation were risk factors for medium-term mortality.

conclusionAKI is a common complication after intervention for PAD and is associated with medium-term mortality.

Indexed as

Acute Kidney InjuryAgedEndovascular ProceduresFemaleHumansMalePeripheral Arterial DiseaseProspective StudiesRisk Factors

Identifiers

PMID33711140
PMCPMC7954277
OpenAlexW3123496556

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.