Evidence mapPaperPMID 33730349Full record

Trial reportJournal of endocrinological investigation2021

Estimated glucose disposal rate as a candidate biomarker for thrombotic biomarkers in T1D: a pooled analysis.

L L O'Mahoney, N Kietsiriroje, S Pearson, D J West, M Holmes, R A Ajjan, M D Campbell

Open access · hybridAbstract readRandomized Controlled Trial
In one paragraph

Trial report in Journal of endocrinological investigation, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 12 papers.

0numbers the graph read from it
0cells of the map it votes in
12citing papers in PubMed
1.9field-weighted citation impact, top 13% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

12 citing papers in PubMed, 16 citations in OpenAlex.

  1. Article
  2. Article
  3. Article
  4. Article
  5. Article
  6. Article
  7. Article
  8. Article
  9. Article
  10. Article
  11. Article
  12. Observational
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 4 institutions in 3 countries.

L L O'MahoneyDiabetes Research Centre, Leicester General Hospital, University of Leicester, Leicester, UK. llom1@leicester.ac.uk.ORCID http://orcid.org/0000-0002-2786-3290
N KietsirirojeEndocrinology and Metabolism Unit, Faculty of Medicine, Prince of Songkla University, Songkhla, Thailand.ORCID http://orcid.org/0000-0002-5076-4450
S PearsonUniversity of Leeds, Leeds Institute for Cardiovascular and Metabolic Medicine, Leeds, UK.ORCID http://orcid.org/0000-0002-4943-6759
D J WestHuman Nutrition Research Centre, Newcastle University, Newcastle, UK.ORCID http://orcid.org/0000-0003-2246-4925
M HolmesSchool of Food Science and Nutrition, University of Leeds, Leeds, UK.
R A AjjanUniversity of Leeds, Leeds Institute for Cardiovascular and Metabolic Medicine, Leeds, UK.ORCID http://orcid.org/0000-0002-1636-3725
M D CampbellUniversity of Leeds, Leeds Institute for Cardiovascular and Metabolic Medicine, Leeds, UK.ORCID http://orcid.org/0000-0001-5883-5041
University of Leeds · GBNewcastle University · GBPrince of Songkla University · THUniversity of Leicester · GB

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

purposeTo determine the utility of estimated glucose disposal rate (eGDR) as a candidate biomarker for thrombotic biomarkers in patients with type 1 diabetes (T1D).

methodsWe reanalysed baseline pretreatment data in a subset of patients with T1D from two previous RCTs, consisting of a panel of thrombotic markers, including fibrinogen, tissue factor (TF) activity, and plasminogen-activator inhibitor (PAI)-1, and TNFα, and clinical factors (age, T1D duration, HbA1c, insulin requirements, BMI, blood pressure, and eGDR). We employed univariate linear regression models to investigate associations between clinical parameters and eGDR with thrombotic biomarkers.

resultsThirty-two patients were included [mean ± SD age 31 ± 7 years, HbA1c of 58 ± 9 mmol/mol (7.5 ± 0.8%), eGDR 7.73 ± 2.61]. eGDR negatively associated with fibrinogen (P < 0.001), PAI-1 concentrations (P = 0.005), and TF activity (P = 0.020), but not TNFα levels (P = 0.881). We identified 2 clusters of patients displaying significantly different characteristics; 56% (n = 18) were categorised as 'higher-risk', eliciting significantly higher fibrinogen (+ 1514 ± 594 μg/mL; P < 0.001), TF activity (+ 59.23 ± 9.42 pmol/mL; P < 0.001), and PAI-1 (+ 8.48 ± 1.58 pmol/dL; P < 0.001), HbA1c concentrations (+ 14.20 ± 1.04 mmol/mol; P < 0.001), age (+ 7 ± 3 years; P < 0.001), duration of diabetes (15 ± 2 years; P < 0.001), BMI (+ 7.66 ± 2.61 kg/m

conclusionsCompared to BMI and insulin requirements, classical surrogates of insulin resistance, eGDR is a suitable and superior thrombotic risk indicator in T1D.

trial registrationISRCTN4081115; registered 27 June 2017.

Indexed as

Blood GlucoseDiabetes Mellitus, Type 1Glycated HemoglobinThrombosisAdultBiomarkersBlood CoagulationBody Mass IndexCluster AnalysisFemaleFibrinogenHumansInsulinInsulin ResistanceMalePlasminogen Activator Inhibitor 1BiomarkersBlood GlucoseFibrinogenGlycated HemoglobinInsulinPlasminogen Activator Inhibitor 1ThromboplastinCluster analysiseGDRThrombosisType 1 diabetes

Identifiers

PMID33730349
PMCPMC8502148
OpenAlexW3138515746

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.