Evidence map›Paper›PMID 33730416›Full record

ReviewAging cell2021

Proteomics in aging research: A roadmap to clinical, translational research.

Ruin Moaddel, Ceereena Ubaida-Mohien, Toshiko Tanaka, Alexey Lyashkov, Nathan Basisty, Birgit Schilling, Richard D Semba, Claudio Franceschi, Myriam Gorospe, Luigi Ferrucci

Open access · goldAbstract readReview
In one paragraph

Review in Aging cell, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 86 papers, 2 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
86citing papers in PubMed, 2 pooled it
13.3field-weighted citation impact, top 1% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

86 citing papers in PubMed, 2 syntheses or guidelines pooled it, 131 citations in OpenAlex.

  1. Proteomic Markers of Aging and Longevity: A Systematic Review.International journal of molecular sciences · 2024
    Pooled it
  2. Screening for Circulating Inflammatory Proteins Does Not Reveal Plasma Biomarkers of Constant Tinnitus.Journal of the Association for Research in Otolaryngology : JARO · 2023
    Pooled it
  3. Trial
  4. Trial
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  11. Review
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  14. Proteomic Characterization of HIV Infection.Advances in experimental medicine and biology · 2026
    Review
  15. Article
  16. Article
  17. Article
  18. Article
  19. Article
  20. Article

26 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors at 4 institutions in 2 countries.

Ruin MoaddelBiomedical Research Centre, National Institute on Aging, NIH, Baltimore, MD, USA.ORCID 0000-0002-6812-0127
Ceereena Ubaida-MohienBiomedical Research Centre, National Institute on Aging, NIH, Baltimore, MD, USA.ORCID 0000-0002-4301-4758
Toshiko TanakaBiomedical Research Centre, National Institute on Aging, NIH, Baltimore, MD, USA.ORCID 0000-0002-4161-3829
Alexey LyashkovBiomedical Research Centre, National Institute on Aging, NIH, Baltimore, MD, USA.
Nathan BasistyBuck Institute for Research on Aging, Novato, CA, USA.ORCID 0000-0001-6173-1139
Birgit SchillingBuck Institute for Research on Aging, Novato, CA, USA.ORCID 0000-0001-9907-2749
Richard D SembaWilmer Eye Institute, Johns Hopkins University School of Medicine, Baltimore, MD, USA.ORCID 0000-0002-8350-2908
Claudio FranceschiUniversity of Bologna and IRCCS Institute of Neurological Sciences Bologna, Italy.
Myriam GorospeBiomedical Research Centre, National Institute on Aging, NIH, Baltimore, MD, USA.ORCID 0000-0001-5439-3434
Luigi FerrucciBiomedical Research Centre, National Institute on Aging, NIH, Baltimore, MD, USA.ORCID 0000-0002-6273-1613
National Institute on Aging · USBuck Institute for Research on Aging · USInstitute of Neurological Sciences · ITJohns Hopkins University · US

Funding

Post-transcriptional Regulation Of Proliferative and Stress Response GenesZIAAG000511 · NIA · NATIONAL INSTITUTE ON AGING · PI GOROSPE, MYRIAM · 2009 to 2025
$25.4M
Quantitative Proteomics to Develop Robust Senescence-Related Biomarkers for AgingU01AG060906 · NIA · BUCK INSTITUTE FOR RESEARCH ON AGING · PI SCHILLING, BIRGIT · 2018 to 2022
$4.0M
Systemic rejuvenating factors and human aging phenotypes.R01AG057723 · NIA · JOHNS HOPKINS UNIVERSITY · PI SEMBA, RICHARD D · 2018 to 2022
$1.8M
Development of Sensitive and Specific Proteomic Biomarkers of Aging, Health, Frailty, and Morbidity in Human CohortsZIAAG000345 · NIA · NATIONAL INSTITUTE ON AGING · PI BASISTY, NATHAN · 2021 to 2025
$689k
Targeting, Quantifying, and Isolating Heterogeneous Populations of Senescent Cells from Tissues via Cell Surface and Secreted ProteomesZIAAG000346 · NIA · NATIONAL INSTITUTE ON AGING · PI BASISTY, NATHAN · 2021 to 2025
$648k
Quantitating, Targeting, and Phenotyping In Vivo Cellular Senescence in HumansK99AG065484 · NIA · BUCK INSTITUTE FOR RESEARCH ON AGING · PI BASISTY, NATAN BOHDONIVICH · 2020 to 2021
$158k
NIA NIH HHS K99 AG065484NIA NIH HHS R01 AG057723NIA NIH HHS U01 AG060906
6 · The paper itself

Abstract

The identification of plasma proteins that systematically change with age and, independent of chronological age, predict accelerated decline of health is an expanding area of research. Circulating proteins are ideal translational "omics" since they are final effectors of physiological pathways and because physicians are accustomed to use information of plasma proteins as biomarkers for diagnosis, prognosis, and tracking the effectiveness of treatments. Recent technological advancements, including mass spectrometry (MS)-based proteomics, multiplexed proteomic assay using modified aptamers (SOMAscan), and Proximity Extension Assay (PEA, O-Link), have allowed for the assessment of thousands of proteins in plasma or other biological matrices, which are potentially translatable into new clinical biomarkers and provide new clues about the mechanisms by which aging is associated with health deterioration and functional decline. We carried out a detailed literature search for proteomic studies performed in different matrices (plasma, serum, urine, saliva, tissues) and species using multiple platforms. Herein, we identified 232 proteins that were age-associated across studies. Enrichment analysis of the 232 age-associated proteins revealed metabolic pathways previously connected with biological aging both in animal models and in humans, most remarkably insulin-like growth factor (IGF) signaling, mitogen-activated protein kinases (MAPK), hypoxia-inducible factor 1 (HIF1), cytokine signaling, Forkhead Box O (FOXO) metabolic pathways, folate metabolism, advance glycation end products (AGE), and receptor AGE (RAGE) metabolic pathway. Information on these age-relevant proteins, likely expanded and validated in longitudinal studies and examined in mechanistic studies, will be essential for patient stratification and the development of new treatments aimed at improving health expectancy.

Indexed as

AgingAnimalsBiomarkersGene Expression RegulationGeroscienceHumansHypoxia-Inducible Factor 1, alpha SubunitMAP Kinase Signaling SystemMitogen-Activated Protein KinasesPrognosisProteomeProteomicsTranslational Research, BiomedicalBiomarkersHIF1A protein, humanHypoxia-Inducible Factor 1, alpha SubunitMitogen-Activated Protein KinasesProteomeaginggerosciencehumanproteomics

Identifiers

PMID33730416
PMCPMC8045948
OpenAlexW3139191177

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.