Evidence mapPaperPMID 33748787Full record

ArticleJVS-vascular science2020

Distinct subsets of T cells and macrophages impact venous remodeling during arteriovenous fistula maturation.

Yutaka Matsubara, Gathe Kiwan, Arash Fereydooni, John Langford, Alan Dardik

Open access · goldAbstract read
In one paragraph

Article in JVS-vascular science, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 26 papers.

0numbers the graph read from it
0cells of the map it votes in
26citing papers in PubMed
3.3field-weighted citation impact, top 7% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

26 citing papers in PubMed, 32 citations in OpenAlex.

  1. Trial
  2. Article
  3. Review
  4. Article
  5. Sustained tenascin-C expression drives neointimal hyperplasia and promotes aortocaval fistula failure.American journal of physiology. Heart and circulatory physiology · 2025
    Article
  6. Article
  7. Article
  8. Article
  9. Review
  10. Review
  11. Article
  12. Article
  13. Article
  14. Review
  15. Article
  16. Sex hormones impact early maturation and immune response in the arteriovenous fistula mouse model.American journal of physiology. Heart and circulatory physiology · 2023
    Article
  17. Review
  18. Article
  19. Increased expression of the P2YRenal failure · 2023
    Article
  20. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 2 institutions in 2 countries.

Yutaka MatsubaraVascular Biology and Therapeutics Program, Yale School of Medicine, New Haven, CT.
Gathe KiwanVascular Biology and Therapeutics Program, Yale School of Medicine, New Haven, CT.
Arash FereydooniVascular Biology and Therapeutics Program, Yale School of Medicine, New Haven, CT.
John LangfordVascular Biology and Therapeutics Program, Yale School of Medicine, New Haven, CT.
Alan DardikVascular Biology and Therapeutics Program, Yale School of Medicine, New Haven, CT.ORCID 0000-0001-5022-7367
Yale University · USKyushu University · JP

Funding

Manipulating the matrix to improve arteriovenous fistula patencyR01HL144476 · YALE UNIVERSITY · 2025 to 2025
$758k
Research Training in AnesthesiaT32GM086287 · NIGMS · YALE UNIVERSITY · 2024 to 2025
$526k
NHLBI NIH HHS R01 HL144476NIGMS NIH HHS T32 GM086287
6 · The paper itself

Abstract

Patients with end-stage renal failure depend on hemodialysis indefinitely without renal transplantation, requiring a long-term patent vascular access. While the arteriovenous fistula (AVF) remains the preferred vascular access for hemodialysis because of its longer patency and fewer complications compared with other vascular accesses, the primary patency of AVF is only 50-60%, presenting a clinical need for improvement. AVF mature by developing a thickened vascular wall and increased diameter to adapt to arterial blood pressure and flow volume. Inflammation plays a critical role during vascular remodeling and fistula maturation; increased shear stress triggers infiltration of T-cells and macrophages that initiate inflammation, with involvement of several different subsets of T-cells and macrophages. We review the literature describing distinct roles of the various subsets of T-cells and macrophages during vascular remodeling. Immunosuppression with sirolimus or prednisolone reduces neointimal hyperplasia during AVF maturation, suggesting novel approaches to enhance vascular remodeling. However, M2 macrophages and CD4+ T-cells play essential roles during AVF maturation, suggesting that total immunosuppression may suppress adaptive vascular remodeling. Therefore it is likely that regulation of inflammation during fistula maturation will require a balanced approach to coordinate the various inflammatory cell subsets. Advances in immunosuppressive drug development and delivery systems may allow for more targeted regulation of inflammation to improve vascular remodeling and enhance AVF maturation.

Indexed as

Arteriovenous fistulaInflammationMacrophagesT-cellsVascular remodeling

Identifiers

PMID33748787
PMCPMC7971420
OpenAlexW3081765708

What Socratic holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.