Evidence map›Paper›PMID 33828089›Full record

ArticleCell death & disease2021

Kv1.3 voltage-gated potassium channels link cellular respiration to proliferation through a non-conducting mechanism.

Faye L Styles, Moza M Al-Owais, Jason L Scragg, Eulashini Chuntharpursat-Bon, Nishani T Hettiarachchi, Jonathan D Lippiat, Aisling Minard, Robin S Bon, Karen Porter, Piruthivi Sukumar and 2 more

Open access · goldAbstract read
In one paragraph

Article in Cell death & disease, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 15 papers.

0numbers the graph read from it
0cells of the map it votes in
15citing papers in PubMed
1.7field-weighted citation impact, top 15% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

15 citing papers in PubMed, 28 citations in OpenAlex.

  1. Keratinocytes: pleiotropic orchestrators of cutaneous immunity and Inflammation - Emerging therapeutic paradigms in dermatological pathologies.Inflammation research : official journal of the European Histamine Research Society ... [et al.] · 2026
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  4. Metallic nanomedicine in cancer immunotherapy.Acta pharmaceutica Sinica. B · 2025
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  9. Insights into3 Biotech · 2023
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  15. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

12 authors at 1 institution in 1 country.

Faye L StylesSchool of Medicine, University of Leeds, Leeds, LS2 9JT, UK.
Moza M Al-OwaisFaculty of Biological Sciences, University of Leeds, Leeds, LS2 9JT, UK.
Jason L ScraggSchool of Medicine, University of Leeds, Leeds, LS2 9JT, UK.
Eulashini Chuntharpursat-BonSchool of Medicine, University of Leeds, Leeds, LS2 9JT, UK.
Nishani T HettiarachchiSchool of Medicine, University of Leeds, Leeds, LS2 9JT, UK.
Jonathan D LippiatFaculty of Biological Sciences, University of Leeds, Leeds, LS2 9JT, UK.
Aisling MinardSchool of Chemistry, University of Leeds, Leeds, LS2 9JT, UK.
Robin S BonSchool of Medicine, University of Leeds, Leeds, LS2 9JT, UK.ORCID 0000-0003-1733-3680
Karen PorterSchool of Medicine, University of Leeds, Leeds, LS2 9JT, UK.
Piruthivi SukumarSchool of Medicine, University of Leeds, Leeds, LS2 9JT, UK.
Chris PeersSchool of Medicine, University of Leeds, Leeds, LS2 9JT, UK.
Lee D RobertsSchool of Medicine, University of Leeds, Leeds, LS2 9JT, UK. L.D.Roberts@leeds.ac.uk.ORCID 0000-0002-1455-5248
University of Leeds · GB

Funding

Biotechnology and Biological Sciences Research Council BB/L015676/1Biotechnology and Biological Sciences Research Council BB/R013500/1British Heart Foundation FS/14/41/30955Diabetes UK 16/0005382Medical Research Council MR/R014086/1Wellcome Trust 110044/Z/15/Z
6 · The paper itself

Abstract

Cellular energy metabolism is fundamental for all biological functions. Cellular proliferation requires extensive metabolic reprogramming and has a high energy demand. The Kv1.3 voltage-gated potassium channel drives cellular proliferation. Kv1.3 channels localise to mitochondria. Using high-resolution respirometry, we show Kv1.3 channels increase oxidative phosphorylation, independently of redox balance, mitochondrial membrane potential or calcium signalling. Kv1.3-induced respiration increased reactive oxygen species production. Reducing reactive oxygen concentrations inhibited Kv1.3-induced proliferation. Selective Kv1.3 mutation identified that channel-induced respiration required an intact voltage sensor and C-terminal ERK1/2 phosphorylation site, but is channel pore independent. We show Kv1.3 channels regulate respiration through a non-conducting mechanism to generate reactive oxygen species which drive proliferation. This study identifies a Kv1.3-mediated mechanism underlying the metabolic regulation of proliferation, which may provide a therapeutic target for diseases characterised by dysfunctional proliferation and cell growth.

Indexed as

Cell ProliferationCell RespirationHumansKv1.3 Potassium ChannelMembrane PotentialsReactive Oxygen SpeciesTransfectionKCNA3 protein, humanKv1.3 Potassium ChannelReactive Oxygen Species

Identifiers

PMID33828089
PMCPMC8027666
OpenAlexW3150604476

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.