Evidence map›Paper›PMID 33833437›Full record

ReviewNature reviews. Rheumatology2021

Cardiovascular effects of approved drugs for rheumatoid arthritis.

Fabiola Atzeni, Javier Rodríguez-Carrio, Călin D Popa, Michael T Nurmohamed, Gabriella Szűcs, Zoltán Szekanecz

Erratum issuedAbstract readReview
PubMed Publisher
In one paragraph

Review in Nature reviews. Rheumatology, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. An erratum has been issued. Cited by 59 papers.

0numbers the graph read from it
0cells of the map it votes in
59citing papers in PubMed
13.3field-weighted citation impact, top 1% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

59 citing papers in PubMed, 101 citations in OpenAlex.

  1. Trial
  2. The risk of venous thromboembolism in RA.Rheumatology (Oxford, England) · 2026
    Review
  3. Review
  4. Article
  5. Review
  6. Article
  7. Article
  8. Article
  9. Article
  10. Article
  11. Article
  12. Review
  13. Metabolomics in spondylarthritis.BMC rheumatology · 2025
    Article
  14. Article
  15. Article
  16. Article
  17. Review
  18. Article
  19. Article
  20. Review
4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

6 authors at 5 institutions in 4 countries.

Fabiola AtzeniRheumatology Unit, Department of Experimental and Internal Medicine, University of Messina, Messina, Italy. atzenifabiola@hotmail.com.
Javier Rodríguez-CarrioDepartment of Functional Biology, Immunology Area, Faculty of Medicine, University of Oviedo, Oviedo, Spain.
Călin D PopaDepartment of Rheumatology, Sint Maartenskliniek Nijmegen, Nijmegen, The Netherlands.
Michael T NurmohamedDeptartment of Rheumatology, Amsterdam University Medical Center & Reade, Amsterdam, The Netherlands.
Gabriella SzűcsDivision of Rheumatology, Faculty of Medicine, University of Debrecen, Debrecen, Hungary.
Zoltán SzekaneczDivision of Rheumatology, Faculty of Medicine, University of Debrecen, Debrecen, Hungary.
University of Debrecen · HUReade · NLSint Maartenskliniek · NLUniversidad de Oviedo · ESUniversity of Messina · IT

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The risk of cardiovascular disease is increased in patients with rheumatoid arthritis compared with the general population owing to the influence of traditional and non-traditional risk factors. Inflammation has a pivotal contribution and can accelerate the atherosclerotic process. Although dampening inflammation with DMARDs should theoretically abrogate this process, evidence suggests that these drugs can also promote atherosclerosis directly and indirectly, hence adding to an increased cardiovascular burden. However, the extent and direction of the effects largely differ across drugs. Understanding how these drugs influence endothelial damage and vascular repair mechanisms is key to understanding these outcomes. NSAIDs and glucocorticoids can increase the cardiovascular risk. Conversely, conventional, biologic and targeted DMARDs control inflammation and reduce this risk, although some of these drugs can also aggravate traditional factors or thrombotic events. Given these data, the fundamental objective for clinicians should be disease control, in an individualized approach that considers the most appropriate drug for each patient, taking into account joint and cardiovascular outcomes. This Review provides a comprehensive analysis of the effects of DMARDs and other approved drugs on cardiovascular involvement in rheumatoid arthritis, from a clinical and mechanistic perspective, with a roadmap to inform the research agenda.

Indexed as

Antirheumatic AgentsArthritis, RheumatoidCardiovascular DiseasesCardiovascular SystemDrug ApprovalHumansRisk FactorsAntirheumatic Agents

Identifiers

PMID33833437
OpenAlexW3146544401

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.