Evidence mapPaperPMID 33909503Full record

ReviewAmerican journal of physiology. Cell physiology2021

Burn-induced hypermetabolism and skeletal muscle dysfunction.

Carly M Knuth, Christopher Auger, Marc G Jeschke

2 registry-linked trialsAbstract readReview
In one paragraph

Review in American journal of physiology. Cell physiology, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. It is linked to 2 registered trials, which are not on this map. Cited by 54 papers, 3 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
54citing papers in PubMed, 3 pooled it
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

NCT06899958 nanot yet recruitingnot on this mapstarted 2027, after this paper: background citation

Metabolism Enhancement by Laser Therapy

TypeinterventionalSponsorMassachusetts General HospitalRan2027 to 2029Enrolled15ConditionsMetabolism DisorderArmsFractional Laser
NCT07590557 nacompletednot on this mapstarted 2024, after this paper: background citation

Rebuilding Capacity With Plyometrics: A Novel and Potent Stimulus for Strength and Power, and Functional Performance in Pediatric Post-Burn Population

TypeinterventionalSponsorQassim UniversityRan2024 to 2025Enrolled62ConditionsPediatric Burns, Burn Injury, Post-Burn Rehabilitation, RehabilitationArmsPlyometric Training, Standard Exercise Program
3 · Its place in the literature

Who cites it

54 citing papers in PubMed, 3 syntheses or guidelines pooled it.

  1. Determination of Burn Size in Mouse Models of Burn Injury: A Scoping Review of Studies Leveraging Scald and Contact Burns.Journal of burn care & research : official publication of the American Burn Association · 2025
    Pooled it
  2. Pooled it
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  4. Review
  5. Article
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  7. [Research on the pharmacokinetics of linezolid in burn patients].Zhonghua shao shang yu chuang mian xiu fu za zhi · 2026
    Article
  8. Review
  9. Cannabis Intoxication Does Not Impact Nutritional Status in Patients With Small Burns.Journal of burn care & research : official publication of the American Burn Association · 2026
    Article
  10. Review
  11. Review
  12. Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors.

Carly M KnuthInstitute of Medical Science, University of Toronto, Toronto, Ontario, Canada.
Christopher AugerDepartment of Biological Sciences, Sunnybrook Research Institute, Toronto, Ontario, Canada.
Marc G JeschkeInstitute of Medical Science, University of Toronto, Toronto, Ontario, Canada.ORCID 0000-0003-0870-1664

Funding

CIHR 123336NIGMS NIH HHS R01 GM133961
6 · The paper itself

Abstract

Critical illnesses, including sepsis, cancer cachexia, and burn injury, invoke a milieu of systemic metabolic and inflammatory derangements that ultimately results in increased energy expenditure leading to fat and lean mass catabolism. Burn injuries present a unique clinical challenge given the magnitude and duration of the hypermetabolic response compared with other forms of critical illness, which drastically increase the risk of morbidity and mortality. Skeletal muscle metabolism is particularly altered as a consequence of burn-induced hypermetabolism, as it primarily provides a main source of fuel in support of wound healing. Interestingly, muscle catabolism is sustained long after the wound has healed, indicating that additional mechanisms beyond wound healing are involved. In this review, we discuss the distinctive pathophysiological response to burn injury with a focus on skeletal muscle function and metabolism. We first examine the diverse consequences on skeletal muscle dysfunction between thermal, electrical, and chemical burns. We then provide a comprehensive overview of the known mechanisms underlying skeletal muscle dysfunction that may be attributed to hypermetabolism. Finally, we review the most promising current treatment options to mitigate muscle catabolism, and by extension improve morbidity and mortality, and end with future directions that have the potential to significantly improve patient care.

Indexed as

Protein BiosynthesisBurnsCachexiaEpigenesis, GeneticExerciseHuman Growth HormoneHumansInsulinMetforminMuscle ProteinsMuscle, SkeletalMuscular AtrophyOxandrolonePropranololProteolysisSepsisHuman Growth HormoneInsulinMetforminMuscle ProteinsOxandrolonePropranololburn injuryhypermetabolisminflammationmuscle dysfunction

Identifiers

PMID33909503
PMCPMC8321793

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.