Evidence mapPaperPMID 33915202Full record

ReviewProgress in lipid research2021

Lipid homeostasis and mevalonate pathway in COVID-19: Basic concepts and potential therapeutic targets.

Maria Chiara Proto, Donatella Fiore, Chiara Piscopo, Cristina Pagano, Mario Galgani, Sara Bruzzaniti, Chiara Laezza, Patrizia Gazzerro, Maurizio Bifulco

Open access · greenAbstract readReview
In one paragraph

Review in Progress in lipid research, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 20 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
20citing papers in PubMed, 1 pooled it
5.9field-weighted citation impact, top 3% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

20 citing papers in PubMed, 1 synthesis or guideline pooled it, 31 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors at 3 institutions in 1 country.

Maria Chiara ProtoDepartment of Pharmacy, University of Salerno, 84084 Fisciano (SA), Italy.
Donatella FioreDepartment of Pharmacy, University of Salerno, 84084 Fisciano (SA), Italy.
Chiara PiscopoDepartment of Pharmacy, University of Salerno, 84084 Fisciano (SA), Italy.
Cristina PaganoDepartment of Molecular Medicine and Medical Biotechnologies, University of Naples "Federico II", 80131 Naples, Italy.
Mario GalganiDepartment of Molecular Medicine and Medical Biotechnologies, University of Naples "Federico II", 80131 Naples, Italy; Institute of Endocrinology and Experimental Oncology, IEOS CNR, 80131 Naples, Italy.
Sara BruzzanitiInstitute of Endocrinology and Experimental Oncology, IEOS CNR, 80131 Naples, Italy; Department of Biology, University of Naples "Federico II", 80126 Naples, Italy.
Chiara LaezzaInstitute of Endocrinology and Experimental Oncology, IEOS CNR, 80131 Naples, Italy.
Patrizia GazzerroDepartment of Pharmacy, University of Salerno, 84084 Fisciano (SA), Italy. Electronic address: pgazzerro@unisa.it.
Maurizio BifulcoDepartment of Molecular Medicine and Medical Biotechnologies, University of Naples "Federico II", 80131 Naples, Italy. Electronic address: maubiful@unina.it.
University of Salerno · ITUniversity of Naples Federico II · ITInstitute for Experimental Endocrinology and Oncology · IT

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Despite encouraging progresses achieved in the management of viral diseases, efficient strategies to counteract infections are still required. The current global challenge highlighted the need to develop a rapid and cost-effective strategy to counteract the SARS-CoV-2 pandemic. Lipid metabolism plays a crucial role in viral infections. Viruses can use the host lipid machinery to support their life cycle and to impair the host immune response. The altered expression of mevalonate pathway-related genes, induced by several viruses, assures survival and spread in host tissue. In some infections, statins, HMG-CoA-reductase inhibitors, reduce cholesterol in the plasma membrane of permissive cells resulting in lower viral titers and failure to internalize the virus. Statins can also counteract viral infections through their immunomodulatory, anti-inflammatory and anti-thrombotic effects. Beyond statins, interfering with the mevalonate pathway could have an adjuvant effect in therapies aimed at mitigating endothelial dysfunction and deregulated inflammation in viral infection. In this review we depicted the historical and current evidence highlighting how lipid homeostasis and mevalonate pathway targeting represents a valid approach to rapidly neutralize viruses, focusing our attention to their potential use as effective targets to hinder SARS-CoV-2 morbidity and mortality. Pros and cons of statins and Mevalonate-pathway inhibitors have been also dissected.

Indexed as

HomeostasisLipid MetabolismCOVID-19COVID-19 Drug TreatmentHumansHydroxymethylglutaryl-CoA Reductase InhibitorsHypolipidemic AgentsMevalonic AcidSARS-CoV-2Hydroxymethylglutaryl-CoA Reductase InhibitorsHypolipidemic AgentsMevalonic AcidCholesterolLipid homeostasisMevalonate pathway inhibitorsSARS-CoV-2StatinsViral infections

Identifiers

PMID33915202
PMCPMC8074527
OpenAlexW3157331149

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.