Evidence map›Paper›PMID 33917744›Full record

ReviewInternational journal of molecular sciences2021

Endothelial Dysfunction, Inflammation and Coronary Artery Disease: Potential Biomarkers and Promising Therapeutical Approaches.

Diana Jhoseline Medina-Leyte, Oscar Zepeda-García, Mayra Domínguez-Pérez, Antonia González-Garrido, Teresa Villarreal-Molina, Leonor Jacobo-Albavera

Registry-linked trialOpen access · goldAbstract readReview
In one paragraph

Review in International journal of molecular sciences, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. It is linked to trial NCT06502535 (The Effects of Inulin and Inorganic Nitrate on Flow-Mediated Dilation and Gut Microbiome in Adults with Pre-Hypertension and Hypertension), which is not on this map. Cited by 360 papers, 12 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
360citing papers in PubMed, 12 pooled it
24.5field-weighted citation impact, top 1% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

NCT06502535 nacompletednot on this mapstarted 2023, after this paper: background citation

The Effects of Inulin and Inorganic Nitrate on Flow-Mediated Dilation and Gut Microbiome in Adults with Pre-Hypertension and Hypertension

TypeinterventionalSponsorUniversity of ExeterRan2023 to 2024Enrolled22ConditionsEndothelial Dysfunction, High Blood Pressure, Gut Microbiota Dysbiosis and NutritionArmsInulin, Potassium nitrate and inulin
3 · Its place in the literature

Who cites it

360 citing papers in PubMed, 12 syntheses or guidelines pooled it, 545 citations in OpenAlex.

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  18. HMGB1 as a danger signal in vascular remodeling.The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology · 2026
    Review
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300 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 1 institution in 1 country.

Diana Jhoseline Medina-LeyteGenomics of Cardiovascular Diseases Laboratory, National Institute of Genomic Medicine (INMEGEN), Mexico City 14610, Mexico.ORCID 0000-0003-1620-9928
Oscar Zepeda-GarcíaGenomics of Cardiovascular Diseases Laboratory, National Institute of Genomic Medicine (INMEGEN), Mexico City 14610, Mexico.ORCID 0000-0002-7436-6887
Mayra Domínguez-PérezGenomics of Cardiovascular Diseases Laboratory, National Institute of Genomic Medicine (INMEGEN), Mexico City 14610, Mexico.ORCID 0000-0002-6049-0430
Antonia González-GarridoGenomics of Cardiovascular Diseases Laboratory, National Institute of Genomic Medicine (INMEGEN), Mexico City 14610, Mexico.ORCID 0000-0003-2578-3155
Teresa Villarreal-MolinaGenomics of Cardiovascular Diseases Laboratory, National Institute of Genomic Medicine (INMEGEN), Mexico City 14610, Mexico.ORCID 0000-0003-4450-7690
Leonor Jacobo-AlbaveraGenomics of Cardiovascular Diseases Laboratory, National Institute of Genomic Medicine (INMEGEN), Mexico City 14610, Mexico.ORCID 0000-0001-5129-8108
National Institute of Genomic Medicine · MX

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Coronary artery disease (CAD) and its complications are the leading cause of death worldwide. Inflammatory activation and dysfunction of the endothelium are key events in the development and pathophysiology of atherosclerosis and are associated with an elevated risk of cardiovascular events. There is great interest to further understand the pathophysiologic mechanisms underlying endothelial dysfunction and atherosclerosis progression, and to identify novel biomarkers and therapeutic strategies to prevent endothelial dysfunction, atherosclerosis and to reduce the risk of developing CAD and its complications. The use of liquid biopsies and new molecular biology techniques have allowed the identification of a growing list of molecular and cellular markers of endothelial dysfunction, which have provided insight on the molecular basis of atherosclerosis and are potential biomarkers and therapeutic targets for the prevention and or treatment of atherosclerosis and CAD. This review describes recent information on normal vascular endothelium function, as well as traditional and novel potential biomarkers of endothelial dysfunction and inflammation, and pharmacological and non-pharmacological therapeutic strategies aimed to protect the endothelium or reverse endothelial damage, as a preventive treatment for CAD and related complications.

Indexed as

BiomarkersAnimalsCapillary PermeabilityCoronary Artery DiseaseDisease ManagementDisease SusceptibilityEndothelium, VascularHemostasisHumansMolecular Targeted TherapyVasculitisBiomarkersatherosclerosiscoronary artery disease (CAD)endothelial cells (EC)endothelial dysfunctionendotheliuminflammationnovel biomarkerstherapeutics

Identifiers

PMID33917744
PMCPMC8068178
OpenAlexW3148162787

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.